Different roles for L3T4+ and Lyt 2+ T cell subsets in the control of an acute herpes simplex virus infection of the skin and nervous system.
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Bovine Herpes Virus 1 (BHV-1) and Herpes Simplex Virus Type 1 (HSV-1) Promote Survival of Latently Infected Sensory Neurons, in Part by Inhibiting ApoptosisSkin CD4(+) memory T cells exhibit combined cluster-mediated retention and equilibration with the circulation.Distinct APC subtypes drive spatially segregated CD4+ and CD8+ T-cell effector activity during skin infection with HSV-1Antigen presentation and the regulation of CD4 memory generation to influenza.Protection against establishment of retroviral persistence by vaccination with a live attenuated virus.Immune response-mediated protection of adult but not neonatal mice from neuron-restricted measles virus infection and central nervous system disease.An important role for major histocompatibility complex class I-restricted T cells, and a limited role for gamma interferon, in protection of mice against lethal herpes simplex virus infection.Towards an understanding of the herpes simplex virus type 1 latency-reactivation cycleDistinctive roles for 2',5'-oligoadenylate synthetases and double-stranded RNA-dependent protein kinase R in the in vivo antiviral effect of an adenoviral vector expressing murine IFN-beta.Critical role for CD4(+) T cells in controlling retrovirus replication and spread in persistently infected mice.CD40 ligand-mediated interactions are involved in the generation of memory CD8(+) cytotoxic T lymphocytes (CTL) but are not required for the maintenance of CTL memory following virus infection.Kinetics of the development of protective immunity in mice vaccinated with a live attenuated retrovirus.Localization of a passively transferred human recombinant monoclonal antibody to herpes simplex virus glycoprotein D to infected nerve fibers and sensory neurons in vivo.Disruption of virion host shutoff activity improves the immunogenicity and protective capacity of a replication-incompetent herpes simplex virus type 1 vaccine strain.Protective T-cell-based immunity induced in neonatal mice by a single replicative cycle of herpes simplex virusProtective mucosal immunity to ocular herpes simplex virus type 1 infection in mice by using Escherichia coli heat-labile enterotoxin B subunit as an adjuvant.Virus-induced delayed-type hypersensitivity reaction is sequentially mediated by CD8+ and CD4+ T lymphocytes.Activated B cells can deliver help for the in vitro generation of antiviral cytotoxic T cells.Herpes simplex virus infection of human fibroblasts and keratinocytes inhibits recognition by cloned CD8+ cytotoxic T lymphocytes.Role of CD8+ T cells and lymphoid dendritic cells in protection from ocular herpes simplex virus 1 challenge in immunized mice.Induction of CD8 T-cell-specific systemic and mucosal immunity against herpes simplex virus with CpG-peptide complexes.Cell surface major histocompatibility complex class II proteins are regulated by the products of the gamma(1)34.5 and U(L)41 genes of herpes simplex virus 1.Overexpression of interleukin-2 by a recombinant herpes simplex virus type 1 attenuates pathogenicity and enhances antiviral immunityHerpes simplex virus type 1 and bovine herpesvirus 1 latency.Experimental investigation of herpes simplex virus latencyHelper T cells, dendritic cells and CTL Immunity.Host response to Sendai virus in mice lacking class II major histocompatibility complex glycoproteins.Vaccination with recombinant vaccinia viruses expressing ICP27 induces protective immunity against herpes simplex virus through CD4+ Th1+ T cells.Gamma interferon expression during acute and latent nervous system infection by herpes simplex virus type 1.Inflammatory infiltration of the trigeminal ganglion after herpes simplex virus type 1 corneal infectionDiffering T-cell requirements for recombinant retrovirus vaccines.Different roles for CD4+ and CD8+ T lymphocytes and macrophage subsets in the control of a generalized virus infection.Rates of reactivation of latent herpes simplex virus from mouse trigeminal ganglia ex vivo correlate directly with viral load and inversely with number of infiltrating CD8+ T cells.Interference with major histocompatibility complex class II-restricted antigen presentation in the brain by herpes simplex virus type 1: a possible mechanism of evasion of the immune response.Anti-CD8 impairs clearance of herpes simplex virus from the nervous system: implications for the fate of virally infected neuronsMechanisms of mouse spleen dendritic cell function in the generation of influenza-specific, cytolytic T lymphocytesSite-restricted persistent cytomegalovirus infection after selective long-term depletion of CD4+ T lymphocytes.Autoimmune diabetes can be induced in transgenic major histocompatibility complex class II-deficient mice.Distinct types of lung disease caused by functional subsets of antiviral T cells.Upregulation of class I major histocompatibility complex gene expression in primary sensory neurons, satellite cells, and Schwann cells of mice in response to acute but not latent herpes simplex virus infection in vivo.
P2860
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P2860
Different roles for L3T4+ and Lyt 2+ T cell subsets in the control of an acute herpes simplex virus infection of the skin and nervous system.
description
1987 nî lūn-bûn
@nan
1987年の論文
@ja
1987年学术文章
@wuu
1987年学术文章
@zh
1987年学术文章
@zh-cn
1987年学术文章
@zh-hans
1987年学术文章
@zh-my
1987年学术文章
@zh-sg
1987年學術文章
@yue
1987年學術文章
@zh-hant
name
Different roles for L3T4+ and ...... f the skin and nervous system.
@en
Different roles for L3T4+ and ...... f the skin and nervous system.
@nl
type
label
Different roles for L3T4+ and ...... f the skin and nervous system.
@en
Different roles for L3T4+ and ...... f the skin and nervous system.
@nl
prefLabel
Different roles for L3T4+ and ...... f the skin and nervous system.
@en
Different roles for L3T4+ and ...... f the skin and nervous system.
@nl
P2093
P1476
Different roles for L3T4+ and ...... f the skin and nervous system.
@en
P2093
P304
P356
10.1099/0022-1317-68-3-825
P407
P478
68 ( Pt 3)
P577
1987-03-01T00:00:00Z