Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.
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New Pathogenic Concepts and Therapeutic Approaches to Oxidative Stress in Chronic Kidney DiseaseTranscriptomics: A Step behind the Comprehension of the Polygenic Influence on Oxidative Stress, Immune Deregulation, and Mitochondrial Dysfunction in Chronic Kidney DiseasePodocytesMitochondria: a new therapeutic target in chronic kidney diseaseTherapeutic targeting of aldosterone: a novel approach to the treatment of glomerular diseaseRotenone remarkably attenuates oxidative stress, inflammation, and fibrosis in chronic obstructive uropathyPharmacological targeting of GSK3β confers protection against podocytopathy and proteinuria by desensitizing mitochondrial permeability transition.Inhibition of insulin/IGF-1 receptor signaling protects from mitochondria-mediated kidney failure.Huaier Cream Protects against Adriamycin-Induced Nephropathy by Restoring Mitochondrial Function via PGC-1α UpregulationGinsenoside Rg1 protects mouse podocytes from aldosterone-induced injury in vitro.PGC-1α overexpression protects against aldosterone-induced podocyte depletion: role of mitochondria.Mitochondrial dysfunction in the pathophysiology of renal diseases.Fetal programming of chronic kidney disease: the role of maternal smoking, mitochondrial dysfunction, and epigenetic modfification.NLRP3 inflammasome activation contributes to aldosterone-induced podocyte injury.MicroRNA-30e targets BNIP3L to protect against aldosterone-induced podocyte apoptosis and mitochondrial dysfunction.Mitochondrial dysfunction confers albumin-induced NLRP3 inflammasome activation and renal tubular injury.Naturally Occurring Compounds: New Potential Weapons against Oxidative Stress in Chronic Kidney Disease.Protein Kinase A/CREB Signaling Prevents Adriamycin-Induced Podocyte Apoptosis via Upregulation of Mitochondrial Respiratory Chain Complexes.Reactive oxygen species-initiated autophagy opposes aldosterone-induced podocyte injury.The roles of oxidative stress, endoplasmic reticulum stress, and autophagy in aldosterone/mineralocorticoid receptor-induced podocyte injury.Dysfunction of the PGC-1α-mitochondria axis confers adriamycin-induced podocyte injury.Bisphenol A is an exogenous toxin that promotes mitochondrial injury and death in tubular cells.Investigation into the underlying molecular mechanisms of hypertensive nephrosclerosis using bioinformatics analyses.A vital role for myosin-9 in puromycin aminonucleoside-induced podocyte injury by affecting actin cytoskeleton.NLRP3 deletion protects against renal fibrosis and attenuates mitochondrial abnormality in mouse with 5/6 nephrectomy.Protective role of cyclosporine A and minocycline on mitochondrial disequilibrium-related podocyte injury and proteinuria occurrence induced by adriamycin.
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P2860
Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
@yue
2013年學術文章
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2013年學術文章
@zh-hant
name
Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.
@en
Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.
@nl
type
label
Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.
@en
Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.
@nl
prefLabel
Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.
@en
Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.
@nl
P2093
P2860
P1476
Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.
@en
P2093
Aihua Zhang
Asish-Roopchand Dhoopun
Bicheng Liu
Chunhua Zhu
Guixia Ding
Songming Huang
Tianxin Yang
Yanggang Yuan
P2860
P304
P356
10.1152/AJPRENAL.00570.2012
P577
2013-06-12T00:00:00Z