Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury. - Wikidata - awgldk - TriplyDB

Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.

Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.

http://www.wikidata.org/entity/Q44067417

about

New Pathogenic Concepts and Therapeutic Approaches to Oxidative Stress in Chronic Kidney Disease Transcriptomics: A Step behind the Comprehension of the Polygenic Influence on Oxidative Stress, Immune Deregulation, and Mitochondrial Dysfunction in Chronic Kidney Disease Podocytes Mitochondria: a new therapeutic target in chronic kidney disease Therapeutic targeting of aldosterone: a novel approach to the treatment of glomerular disease Rotenone remarkably attenuates oxidative stress, inflammation, and fibrosis in chronic obstructive uropathy Pharmacological targeting of GSK3β confers protection against podocytopathy and proteinuria by desensitizing mitochondrial permeability transition.Inhibition of insulin/IGF-1 receptor signaling protects from mitochondria-mediated kidney failure.Huaier Cream Protects against Adriamycin-Induced Nephropathy by Restoring Mitochondrial Function via PGC-1α Upregulation Ginsenoside Rg1 protects mouse podocytes from aldosterone-induced injury in vitro.PGC-1α overexpression protects against aldosterone-induced podocyte depletion: role of mitochondria.Mitochondrial dysfunction in the pathophysiology of renal diseases.Fetal programming of chronic kidney disease: the role of maternal smoking, mitochondrial dysfunction, and epigenetic modfification.NLRP3 inflammasome activation contributes to aldosterone-induced podocyte injury.MicroRNA-30e targets BNIP3L to protect against aldosterone-induced podocyte apoptosis and mitochondrial dysfunction.Mitochondrial dysfunction confers albumin-induced NLRP3 inflammasome activation and renal tubular injury.Naturally Occurring Compounds: New Potential Weapons against Oxidative Stress in Chronic Kidney Disease.Protein Kinase A/CREB Signaling Prevents Adriamycin-Induced Podocyte Apoptosis via Upregulation of Mitochondrial Respiratory Chain Complexes.Reactive oxygen species-initiated autophagy opposes aldosterone-induced podocyte injury.The roles of oxidative stress, endoplasmic reticulum stress, and autophagy in aldosterone/mineralocorticoid receptor-induced podocyte injury.Dysfunction of the PGC-1α-mitochondria axis confers adriamycin-induced podocyte injury.Bisphenol A is an exogenous toxin that promotes mitochondrial injury and death in tubular cells.Investigation into the underlying molecular mechanisms of hypertensive nephrosclerosis using bioinformatics analyses.A vital role for myosin-9 in puromycin aminonucleoside-induced podocyte injury by affecting actin cytoskeleton.NLRP3 deletion protects against renal fibrosis and attenuates mitochondrial abnormality in mouse with 5/6 nephrectomy.Protective role of cyclosporine A and minocycline on mitochondrial disequilibrium-related podocyte injury and proteinuria occurrence induced by adriamycin.

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P2860

Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.

http://www.wikidata.org/entity/Q44067417

description

2013 nî lūn-bûn

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2013年の論文

@ja

2013年学术文章

@wuu

2013年学术文章

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2013年学术文章

@zh-hans

2013年学术文章

@zh-my

2013年学术文章

@zh-sg

2013年學術文章

@yue

2013年學術文章

@zh

2013年學術文章

@zh-hant

name

Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.

@en

Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.

@nl

type

label

Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.

@en

Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.

@nl

prefLabel

Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.

@en

Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.

@nl

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AJPRENAL.00570.2012

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American Journal of Physiology - Renal Physiology

P1476

Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.

@en

P2093

Aihua Zhang

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Asish-Roopchand Dhoopun

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Bicheng Liu

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Chunhua Zhu

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Guixia Ding

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Min Su

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Songming Huang

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Tianxin Yang

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Yanggang Yuan

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P2860

Architectural role of mitochondrial transcription factor A in maintenance of human mitochondrial DNA

Mitochondrial transcription factor A is necessary for mtDNA maintenance and embryogenesis in mice

Podocyte glutamatergic signaling contributes to the function of the glomerular filtration barrier.

Proteinuria: an enzymatic disease of the podocyte?

The role of oxidative damage in mitochondria during aging: a review.

Mitochondrial dysfunction mediates aldosterone-induced podocyte damage: a therapeutic target of PPARγ.

Mitochondrial oxidative stress in cancer-associated fibroblasts drives lactate production, promoting breast cancer tumor growth: understanding the aging and cancer connection

Mitochondrial transcription factor A (TFAM): roles in maintenance of mtDNA and cellular functions.

The role of podocytes in proteinuria.

Pathogenesis of the podocytopathy and proteinuria in diabetic glomerulopathy.

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