Mitochondrial dysfunction mediates aldosterone-induced podocyte damage: a therapeutic target of PPARγ. - Wikidata - awgldk - TriplyDB

Mitochondrial dysfunction mediates aldosterone-induced podocyte damage: a therapeutic target of PPARγ.

Mitochondrial dysfunction mediates aldosterone-induced podocyte damage: a therapeutic target of PPARγ.

http://www.wikidata.org/entity/Q34839549

about

New Pathogenic Concepts and Therapeutic Approaches to Oxidative Stress in Chronic Kidney Disease Podocytes Therapeutic targeting of aldosterone: a novel approach to the treatment of glomerular disease The podocyte power-plant disaster and its contribution to glomerulopathy.Pharmacological targeting of GSK3β confers protection against podocytopathy and proteinuria by desensitizing mitochondrial permeability transition.cAMP signaling prevents podocyte apoptosis via activation of protein kinase A and mitochondrial fusion.Loss of Krüppel-like factor 6 cripples podocyte mitochondrial function.Huaier Cream Protects against Adriamycin-Induced Nephropathy by Restoring Mitochondrial Function via PGC-1α Upregulation Progress in pathogenesis of proteinuria.Rotenone Attenuates Renal Injury in Aldosterone-Infused Rats by Inhibiting Oxidative Stress, Mitochondrial Dysfunction, and Inflammasome Activation.The redox sensitive glycogen synthase kinase 3β suppresses the self-protective antioxidant response in podocytes upon oxidative glomerular injury.Ginsenoside Rg1 protects mouse podocytes from aldosterone-induced injury in vitro.Redox-sensitive glycogen synthase kinase 3β-directed control of mitochondrial permeability transition: rheostatic regulation of acute kidney injury.Contribution of aldosterone to cardiovascular and renal inflammation and fibrosis.New Insights into the PPAR γ Agonists for the Treatment of Diabetic Nephropathy.Diabetes-Induced Reactive Oxygen Species: Mechanism of Their Generation and Role in Renal Injury The renoprotective actions of peroxisome proliferator-activated receptors agonists in diabetes.Role of Rac1-mineralocorticoid-receptor signalling in renal and cardiac disease.Mineralocorticoid receptor activation as an etiological factor in kidney diseases.Association of peroxisome proliferator-activated receptors/retinoic acid receptors with renal diseases.Mitochondrial dysfunction in the pathophysiology of renal diseases.Fetal programming of chronic kidney disease: the role of maternal smoking, mitochondrial dysfunction, and epigenetic modfification.NLRP3 inflammasome activation contributes to aldosterone-induced podocyte injury.MicroRNA-30e targets BNIP3L to protect against aldosterone-induced podocyte apoptosis and mitochondrial dysfunction.Peroxisome proliferator activating receptor-γ and the podocyte.The metabolic syndrome and chronic kidney disease.Pioglitazone Improves Mitochondrial Function in the Remnant Kidney and Protects against Renal Fibrosis in 5/6 Nephrectomized Rats.Dahuang Fuzi Decoction Attenuates Renal Fibrosis and Ameliorates Mitochondrial Dysfunction in Chronic Aristolochic Acid Nephropathy.Peroxisome proliferator-activated receptor γ Pro12Ala polymorphism decrease the risk of diabetic nephropathy in type 2 diabetes: a meta analysis.Mitochondrial dysfunction is an early event in aldosterone-induced podocyte injury.Reactive oxygen species-initiated autophagy opposes aldosterone-induced podocyte injury.Grape seed procyanidin B2 protects podocytes from high glucose-induced mitochondrial dysfunction and apoptosis via the AMPK-SIRT1-PGC-1α axis in vitro.The roles of oxidative stress, endoplasmic reticulum stress, and autophagy in aldosterone/mineralocorticoid receptor-induced podocyte injury.Hyperoside alleviates adriamycin-induced podocyte injury via inhibiting mitochondrial fission.Total flavones of Abelmoschus manihot improve diabetic nephropathy by inhibiting the iRhom2/TACE signalling pathway activity in rats.Urinary mitochondrial DNA level is an indicator of intra-renal mitochondrial depletion and renal scarring in diabetic nephropathy.Sildenafil Prevents Podocyte Injury via PPAR-γ-Mediated TRPC6 Inhibition.The potential signaling pathway between peroxisome proliferator-activated receptor gamma and retinoic acid receptor alpha in renal interstitial fibrosis disease.Contributions of endoplasmic reticulum stress and reactive oxygen species to renal injury in aldosterone/salt-induced rats.Aldosterone Impairs Mitochondrial Function in Human Cardiac Fibroblasts via A-Kinase Anchor Protein 12.

P2860

Q26739475-E0F94FDD-E0FF-4F40-B63D-1EA2970446EB Q26766118-9733500E-5772-4B4B-BCD9-DAC7BF0B8D78 Q26996385-4AB92FCA-2861-46F7-BF8C-2555CB6BF54A Q34699773-88087C02-A0BB-41C7-BD9D-464A6CB49255 Q34994550-DDDFA27C-4FE4-4D7C-A9C0-2482E8E7C870 Q35124052-FD73BFCA-6428-434D-A5A4-DCF87E8035BF Q35183931-F7A08BB9-5522-4BD7-905E-38B42B0C7947 Q35226985-8E66269E-548C-49D1-A340-1F5F20D29C4C Q36011008-72E02C41-830A-4D09-A766-8864E057E268 Q36187559-10C72886-A312-48B2-BAFC-AF40E4B3FB4F Q36545412-2A426B05-1DD5-4965-B973-3A99E4B9BBFC Q36745190-7FDF8EFB-E9AD-43FE-83B4-7D368B52CE93 Q37385047-16DC9160-632A-43E1-9637-E35F903C0A91 Q37580301-7391761E-6CD0-47DE-B64B-00C5CA72B285 Q37590946-CE347DE2-FBDB-4805-83B4-CE436E39E505 Q37597721-294D2CB0-473B-42D1-9A9C-5F458F2044F7 Q37997004-3054603C-916D-4AA6-906B-F2722137A091 Q38072297-4B8BEB32-8131-4AA0-AE7D-7024FC3B5100 Q38119861-3D6031DA-5D4C-4AF6-92DC-5742860F7D54 Q38139624-6799ED80-66AC-4125-AC10-B1473F4DF527 Q38168664-E87A6009-3AF6-4977-B436-3E4956AC1FC2 Q38345872-96DCB837-0AC5-4F97-9222-EA3750087852 Q38722963-4F3935B1-1274-4602-8CE2-A7C89159FD24 Q38726222-B70D54E0-FF44-479B-8824-EC6D73CD01FD Q38815188-00027FCB-87FB-4C6F-AF9C-126711BE8384 Q39056509-9C685F66-2889-4572-8888-158055470879 Q41437002-464415D4-F97E-45DB-91D9-F9193A2C9C38 Q42292920-E1F5A046-BC69-48E3-9F53-BD3F88D81150 Q42364753-86200B8A-A6EF-4961-B717-DAEBF9E934DA Q44067417-99A2ADE1-163B-4CBC-93E9-7DD74150BF40 Q46611165-7E281850-228D-446B-B410-BDABA0C72723 Q46626225-B1544A17-919D-430A-B235-9FF5477C2564 Q46662837-2CC964A2-4A16-4671-BED6-89D574A4C833 Q47148010-84DC4B8A-EA35-491B-B262-AFB11B37E675 Q49995821-1C03AC02-64A4-4662-8F68-01B68BB20DAE Q50192998-6324CC36-A452-4F6F-91EA-B4B5113B74EF Q51299126-E1F6C8F9-CE19-46E8-BF3E-8BFE7287A4FC Q53007399-1CCACBE6-24FD-4F84-ABCF-996EFCE251E2 Q53061760-4B33DFB5-EF2C-45B2-A3A9-7920C0AA51DE Q54979932-1B8091FD-08F1-4143-B134-0D4C2E3B56F8

P2860

Mitochondrial dysfunction mediates aldosterone-induced podocyte damage: a therapeutic target of PPARγ.

http://www.wikidata.org/entity/Q34839549

description

2011 nî lūn-bûn

@nan

2011 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի մայիսին հրատարակված գիտական հոդված

@hy

2011年の論文

@ja

2011年論文

@yue

2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

name

Mitochondrial dysfunction medi ...... a therapeutic target of PPARγ.

@ast

Mitochondrial dysfunction medi ...... a therapeutic target of PPARγ.

@en

Mitochondrial dysfunction medi ...... a therapeutic target of PPARγ.

@nl

type

label

Mitochondrial dysfunction medi ...... a therapeutic target of PPARγ.

@ast

Mitochondrial dysfunction medi ...... a therapeutic target of PPARγ.

@en

Mitochondrial dysfunction medi ...... a therapeutic target of PPARγ.

@nl

prefLabel

Mitochondrial dysfunction medi ...... a therapeutic target of PPARγ.

@ast

Mitochondrial dysfunction medi ...... a therapeutic target of PPARγ.

@en

Mitochondrial dysfunction medi ...... a therapeutic target of PPARγ.

@nl

P1433

Q34839549-92277423-86E8-4B94-A30A-9615AA05548D

P1476

Q34839549-5C7982E3-C5BA-4CE4-9262-435BD71B9841

P2093

Q34839549-073DF79B-FFF7-4FED-9D30-24EB8AB2BB63

Q34839549-2F063181-947D-4402-B440-B307539184DB

Q34839549-50CABC64-5D3C-42D5-800F-8DBCDCB8680E

Q34839549-7C4E01BF-8240-4D21-AC9D-D0ADA0B2E01B

Q34839549-88E5F3A8-3A34-4746-A00D-D8923111A7F2

Q34839549-A643C955-5A4B-4F8E-870F-43C0C4466631

Q34839549-FA0D818D-5BF2-4DB1-B0B0-B6277CAD0D65

Q34839549-FC05B16E-1310-4688-B803-9E157B770580

P2860

Q34839549-049A3CBA-73BB-454D-89F1-F2F078E87227

Q34839549-0D62F162-005C-450F-B26F-F612B87F1B70

Q34839549-0EC0CD55-7C45-42D5-84B6-CA04114F1D1B

Q34839549-14BF76A6-600E-4522-A716-728B3B42F750

Q34839549-1881D447-DAB2-45C2-9AEA-E572BC1AB409

Q34839549-1A00C74B-B508-4439-8790-EDF19B7B6CBA

Q34839549-2078F687-5DD1-4D4E-8E2D-A3725C309A71

Q34839549-291763AC-5277-4303-B8B2-295ACBEF5E8F

Q34839549-2F133A13-2C00-47D0-906C-4D8A2847D422

Q34839549-3307E36C-0BDC-45BD-A6D5-DD08630050EE

P304

Q34839549-F2A77AE6-FE7E-4B5D-8FCA-A0080B96347A

P31

Q34839549-19C5CE7D-27F2-418B-A920-7C5CDF261E76

P356

Q34839549-8ADE017A-90C7-4DF4-8275-EA9296EE93F9

P407

Q34839549-9F22CA56-F460-4026-A13F-25E5C2F879EF

P433

Q34839549-2D10081F-A72B-49B0-A570-E4DD082DD44B

P478

Q34839549-E2DFF515-A70C-435F-9537-83BA0B2AF174

P577

Q34839549-AE319847-6209-40EA-99CF-8316D8818CD5

P5875

Q34839549-4CDA15AD-FE94-4FE4-8661-B8F7535E6EF1

P698

Q34839549-C3CAB703-D98A-4932-B01E-39D96A41215F

P932

Q34839549-15EA6E0C-19C7-4586-85E8-C9282AF3B2F5

P356

J.AJPATH.2011.01.029

P1433

The American Journal of Pathology

P1476

Mitochondrial dysfunction medi ...... a therapeutic target of PPARγ.

@en

P2093

Aihua Zhang

http://www.w3.org/2001/XMLSchema#string

Bicheng Liu

http://www.w3.org/2001/XMLSchema#string

Chunhua Zhu

http://www.w3.org/2001/XMLSchema#string

Guixia Ding

http://www.w3.org/2001/XMLSchema#string

Ronghua Chen

http://www.w3.org/2001/XMLSchema#string

Songming Huang

http://www.w3.org/2001/XMLSchema#string

Tianxin Yang

http://www.w3.org/2001/XMLSchema#string

Yanggang Yuan

http://www.w3.org/2001/XMLSchema#string

P2860

A cold-inducible coactivator of nuclear receptors linked to adaptive thermogenesis

Mechanisms controlling mitochondrial biogenesis and respiration through the thermogenic coactivator PGC-1

Structure and dynamics of the mitochondrial inner membrane cristae

Kidney expression of glutathione peroxidase-1 is not protective against streptozotocin-induced diabetic nephropathy

Hydroperoxide metabolism in mammalian organs

Identification of the HIV-1 gp41 core-binding motif in the scaffolding domain of caveolin-1.

The role of the podocyte in glomerulosclerosis.

Aldosterone as a mediator of progressive renal disease: pathogenetic and clinical implications.

Increased production of reactive oxygen species in hyperglycemic conditions requires dynamic change of mitochondrial morphology.

Insulin resistance and atherosclerosis.

P304

2020-2031

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1016/J.AJPATH.2011.01.029

http://www.w3.org/2001/XMLSchema#string

P407

P433

5

http://www.w3.org/2001/XMLSchema#string

P478

178

http://www.w3.org/2001/XMLSchema#string

P577

2011-05-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

51071488

http://www.w3.org/2001/XMLSchema#string

P698

21514419

http://www.w3.org/2001/XMLSchema#string

P932

3081205

http://www.w3.org/2001/XMLSchema#string