Effects of mitochondrial K(ATP) modulators on cardioprotection induced by chronic high altitude hypoxia in rats. - Wikidata - awgldk - TriplyDB

Effects of mitochondrial K(ATP) modulators on cardioprotection induced by chronic high altitude hypoxia in rats.

Effects of mitochondrial K(ATP) modulators on cardioprotection induced by chronic high altitude hypoxia in rats.

http://www.wikidata.org/entity/Q44088301

about

Improved energy supply regulation in chronic hypoxic mouse counteracts hypoxia-induced altered cardiac energetics Chronic intermittent hypoxia alters Ca2+ handling in rat cardiomyocytes by augmented Na+/Ca2+ exchange and ryanodine receptor activities in ischemia-reperfusion Role of oxidative stress in PKC-delta upregulation and cardioprotection induced by chronic intermittent hypoxia Diversity of mitochondria-dependent dilator mechanisms in vascular smooth muscle of cerebral arteries from normal and insulin-resistant rats.Mitochondrial energy metabolism plays a critical role in the cardioprotection afforded by intermittent hypobaric hypoxia.Mitochondrial BKCa channels contribute to protection of cardiomyocytes isolated from chronically hypoxic rats.Hypoxia. 4. Hypoxia and ion channel function Therapeutic potential of intermittent hypoxia: a matter of dose.Cardiac response to chronic intermittent hypoxia with a transition from adaptation to maladaptation: the role of hydrogen peroxide.Depolarization of mitochondria in endothelial cells promotes cerebral artery vasodilation by activation of nitric oxide synthase Chronic intermittent hypoxia affects the cytosolic phospholipase A2α/cyclooxygenase 2 pathway via β2-adrenoceptor-mediated ERK/p38 stimulation.Mitochondria as a target of cardioprotection in models of preconditioning.Effect of sustained hypobaric hypoxia during maturation and aging on rat myocardium. I. Mechanical activity.Intermittent hypobaric hypoxia applicability in myocardial infarction prevention and recovery.Mitochondrial KATP opening confers protection against lethal myocardial injury and ischaemia-induced arrhythmias in the rat heart via PI3K/Akt-dependent and -independent mechanisms.Increased expression and altered subcellular distribution of PKC-delta in chronically hypoxic rat myocardium: involvement in cardioprotection.Chronic intermittent hypoxia increases infarction in the isolated rat heart.Inducible nitric oxide synthase contributes to intermittent hypoxia against ischemia/reperfusion injury.Antioxidant tempol suppresses heart cytosolic phospholipase A2α stimulated by chronic intermittent hypoxia.Cardioprotective adaptation of rats to intermittent hypobaric hypoxia is accompanied by the increased association of hexokinase with mitochondria.Cardioprotective and nonprotective regimens of chronic hypoxia diversely affect the myocardial antioxidant systems.Modulation of mitochondrial respiratory function and ROS production by novel benzopyran analogues.The mitochondrial K(ATP) channel opener BMS-191095 reduces neuronal damage after transient focal cerebral ischemia in rats.Cardioprotection by Intermittent Hypoxia Conditioning: Evidence, Mechanisms and Therapeutic Potential.Activation of α1B-adrenoceptors contributes to intermittent hypobaric hypoxia-improved postischemic myocardial performance via inhibiting MMP-2 activation.

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P2860

Effects of mitochondrial K(ATP) modulators on cardioprotection induced by chronic high altitude hypoxia in rats.

http://www.wikidata.org/entity/Q44088301

description

2002 nî lūn-bûn

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2002年の論文

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年學術文章

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2002年學術文章

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Effects of mitochondrial K

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Effects of mitochondrial K(ATP ...... high altitude hypoxia in rats.

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Effects of mitochondrial K

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Effects of mitochondrial K(ATP ...... high altitude hypoxia in rats.

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Effects of mitochondrial K

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Effects of mitochondrial K(ATP ...... high altitude hypoxia in rats.

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S0008-6363(02)00456-X

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Cardiovascular Research

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Effects of mitochondrial K(ATP ...... high altitude hypoxia in rats.

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Bohuslav Ost'ádal

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Frantisek Kolár

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Frantisek Papousek

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Gary J Grover

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Jan Neckár

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567-575

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scholarly article

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10.1016/S0008-6363(02)00456-X

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55

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