The mitochondrial K(ATP) channel opener BMS-191095 reduces neuronal damage after transient focal cerebral ischemia in rats.
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Neuroprotective role of ATP-sensitive potassium channels in cerebral ischemiaAcute treatment with rosuvastatin protects insulin resistant (C57BL/6J ob/ob) mice against transient cerebral ischemia.Delayed neuronal preconditioning by NS1619 is independent of calcium activated potassium channels.Is there a place for cerebral preconditioning in the clinic?Diversity of mitochondria-dependent dilator mechanisms in vascular smooth muscle of cerebral arteries from normal and insulin-resistant rats.Mitochondria: the missing link between preconditioning and neuroprotection.Mitochondrial mechanisms in cerebral vascular control: shared signaling pathways with preconditioning.Depolarization of mitochondria in endothelial cells promotes cerebral artery vasodilation by activation of nitric oxide synthaseMolecular physiology of preconditioning-induced brain tolerance to ischemia.Mitochondrial-mediated suppression of ROS production upon exposure of neurons to lethal stress: mitochondrial targeted preconditioning.Immediate neuronal preconditioning by NS1619.Preconditioning provides neuroprotection in models of CNS disease: paradigms and clinical significance.Pharmacology of mitochondrial potassium channels: dark side of the field.Mitochondrial preconditioning: a potential neuroprotective strategy.Neuron specific metabolic adaptations following multi-day exposures to oxygen glucose deprivation.ROS-independent preconditioning in neurons via activation of mitoK(ATP) channels by BMS-191095.The mechanistic target of rapamycin (mTOR) pathway and S6 Kinase mediate diazoxide preconditioning in primary rat cortical neurons.Modulation of mitochondrial respiratory function and ROS production by novel benzopyran analogues.Role of Mitochondria in Cerebral Vascular Function: Energy Production, Cellular Protection, and Regulation of Vascular Tone.Neurovascular protection by ischaemic tolerance: role of nitric oxide.
P2860
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P2860
The mitochondrial K(ATP) channel opener BMS-191095 reduces neuronal damage after transient focal cerebral ischemia in rats.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
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2006年学术文章
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2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
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2006年學術文章
@zh-hant
name
The mitochondrial K
@nl
The mitochondrial K(ATP) chann ...... cal cerebral ischemia in rats.
@en
type
label
The mitochondrial K
@nl
The mitochondrial K(ATP) chann ...... cal cerebral ischemia in rats.
@en
prefLabel
The mitochondrial K
@nl
The mitochondrial K(ATP) chann ...... cal cerebral ischemia in rats.
@en
P2093
P2860
P356
P1476
The mitochondrial K(ATP) chann ...... ocal cerebral ischemia in rats
@en
P2093
David W Busija
Keita Mayanagi
Tamás Gáspár
P2860
P304
P356
10.1038/SJ.JCBFM.9600345
P577
2006-05-31T00:00:00Z