The role of herpes simplex virus thymidine kinase in the treatment of solid tumours.

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The role of herpes simplex virus thymidine kinase in the treatment of solid tumours.

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http://www.wikidata.org/entity/Q44113154

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1999 nî lūn-bûn

@nan

1999年の論文

@ja

1999年学术文章

@wuu

1999年学术文章

@zh

1999年学术文章

@zh-cn

1999年学术文章

@zh-hans

1999年学术文章

@zh-my

1999年学术文章

@zh-sg

1999年學術文章

@yue

1999年學術文章

@zh-hant

name

The role of herpes simplex virus thymidine kinase in the treatment of solid tumours.

@en

The role of herpes simplex virus thymidine kinase in the treatment of solid tumours.

@nl

type

Item

label

The role of herpes simplex virus thymidine kinase in the treatment of solid tumours.

@en

The role of herpes simplex virus thymidine kinase in the treatment of solid tumours.

@nl

prefLabel

The role of herpes simplex virus thymidine kinase in the treatment of solid tumours.

@en

The role of herpes simplex virus thymidine kinase in the treatment of solid tumours.

@nl

P1476

The role of herpes simplex virus thymidine kinase in the treatment of solid tumours.

@en

P2093

Dalgleish AG

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Perry MJ

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Todryk SM

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P2860

A "distant" bystander effect of suicide gene therapy: regression of nontransduced tumors together with a distant transduced tumor.

The "bystander effect": association of U-87 cell death with ganciclovir-mediated apoptosis of nearby cells and lack of effect in athymic mice.

In vitro evidence that metabolic cooperation is responsible for the bystander effect observed with HSV tk retroviral gene therapy.

Gap-junctional communication in normal and neoplastic prostate epithelial cells and its regulation by cAMP.

Tumor immunogenicity is determined by the mechanism of cell death via induction of heat shock protein expression.

A reassessment of the role of B7-1 expression in tumor rejection.

Regression of established macroscopic liver metastases after in situ transduction of a suicide gene.

Lack of bystander killing in herpes simplex virus thymidine kinase-transduced colon cell lines due to deficient connexin43 gap junction formation.

In vitro and in vivo antitumor effects of retrovirus-mediated herpes simplex thymidine kinase gene-transfer in human medulloblastoma.

Increased bax expression is associated with cell death induced by ganciclovir in a herpes thymidine kinase gene-expressing glioma cell line.

P304

777-785

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P31

scholarly article

P356

10.1517/13543784.8.6.777

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P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

1999-06-01T00:00:00Z

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P698

15992130

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