about
Estradiol activates beta-catenin dependent transcription in neurons.Anesthetics rapidly promote synaptogenesis during a critical period of brain development.Desynchronization of neocortical networks by asynchronous release of GABA at autaptic and synaptic contacts from fast-spiking interneurons.Activity-dependent inhibitory synapse remodeling through gephyrin phosphorylation.Assortment of GABAergic plasticity in the cortical interneuron melting pot.Interdependence of oestrogen and insulin-like growth factor-I in the brain: potential for analysing neuroprotective mechanisms.Cross-talk between estrogen receptors and insulin-like growth factor-I receptor in the brain: cellular and molecular mechanisms.Contribution of estrogen receptors alpha and beta to the effects of estradiol in the brain.Nitric oxide mediates local activity-dependent excitatory synapse development.Interaction of estrogen receptors with insulin-like growth factor-I and Wnt signaling in the nervous system.Role of NCAM in spine dynamics and synaptogenesis.Shaping inhibition: activity dependent structural plasticity of GABAergic synapses.Phosphatidylinositol 3-kinase and glycogen synthase kinase 3 regulate estrogen receptor-mediated transcription in neuronal cells.Reversal of activity-mediated spine dynamics and learning impairment in a mouse model of Fragile X syndrome.Direct alteration of a specific inhibitory circuit of the hippocampus by antidepressants.Rapid stimulation of the PI3-kinase/Akt signalling pathway in developing midbrain neurones by oestrogen.Synergistic interaction of estradiol and insulin-like growth factor-I in the activation of PI3K/Akt signaling in the adult rat hypothalamus.Estrogen receptor alpha forms estrogen-dependent multimolecular complexes with insulin-like growth factor receptor and phosphatidylinositol 3-kinase in the adult rat brain.Implication of the phosphatidylinositol-3 kinase/protein kinase B signaling pathway in the neuroprotective effect of estradiol in the striatum of 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine mice.Hippocampal Somatostatin Interneurons Control the Size of Neuronal Memory Ensembles.Activity-dependent PSD formation and stabilization of newly formed spines in hippocampal slice cultures.Cation-chloride cotransporters and GABA-ergic innervation in the human epileptic hippocampus.The distribution and mechanism of action of ghrelin in the CNS demonstrates a novel hypothalamic circuit regulating energy homeostasis.Homeostatic Plasticity in the Hippocampus Facilitates Memory Extinction.Interactions of estrogens and insulin-like growth factor-I in the brain: implications for neuroprotectionEstradiol promotes spine growth and synapse formation without affecting pre-established networks
P50
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P50
description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Pablo Mendez
@ast
Pablo Mendez
@en
Pablo Mendez
@es
Pablo Mendez
@nl
Pablo Mendez
@sl
type
label
Pablo Mendez
@ast
Pablo Mendez
@en
Pablo Mendez
@es
Pablo Mendez
@nl
Pablo Mendez
@sl
prefLabel
Pablo Mendez
@ast
Pablo Mendez
@en
Pablo Mendez
@es
Pablo Mendez
@nl
Pablo Mendez
@sl
P108
P1053
J-7552-2017
P106
P108
P21
P2798
P31
P3829
P496
0000-0001-9862-6818