Desynchronization of neocortical networks by asynchronous release of GABA at autaptic and synaptic contacts from fast-spiking interneurons.
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Enhancement of asynchronous release from fast-spiking interneuron in human and rat epileptic neocortexModels of vocal learning in the songbird: Historical frameworks and the stabilizing critic.Lack of parvalbumin in mice leads to behavioral deficits relevant to all human autism core symptoms and related neural morphofunctional abnormalities.Behavioral and neurochemical consequences of cortical oxidative stress on parvalbumin-interneuron maturation in rodent models of schizophrenia.Regulation of the Hippocampal Network by VGLUT3-Positive CCK- GABAergic Basket Cells.PGC-1α provides a transcriptional framework for synchronous neurotransmitter release from parvalbumin-positive interneurons.Vesicles derived via AP-3-dependent recycling contribute to asynchronous release and influence information transferAssortment of GABAergic plasticity in the cortical interneuron melting pot.Downregulation of parvalbumin at cortical GABA synapses reduces network gamma oscillatory activity.Shaping Neuronal Network Activity by Presynaptic MechanismsFunctional alterations in GABAergic fast-spiking interneurons in chronically injured epileptogenic neocortex.A Phenomenological Synapse Model for Asynchronous Neurotransmitter Release.Reduction in parvalbumin expression not loss of the parvalbumin-expressing GABA interneuron subpopulation in genetic parvalbumin and shank mouse models of autismExtrinsic and Intrinsic Brain Network Connectivity Maintains Cognition across the Lifespan Despite Accelerated Decay of Regional Brain Activation.Regulation of epileptiform discharges in rat neocortex by HCN channelsFiring Frequency Maxima of Fast-Spiking Neurons in Human, Monkey, and Mouse Neocortex.Modulation of cortical inhibition by rTMS - findings obtained from animal models.Molecular mechanisms for synchronous, asynchronous, and spontaneous neurotransmitter release.Synaptic Vesicle-Recycling Machinery Components as Potential Therapeutic Targets.Synapsin II desynchronizes neurotransmitter release at inhibitory synapses by interacting with presynaptic calcium channels.Asynchronous GABA Release Is a Key Determinant of Tonic Inhibition and Controls Neuronal Excitability: A Study in the Synapsin II-/- Mouse.Heterogeneity of presynaptic proteins: do not forget isoforms.Neuregulin 1 Type I Overexpression Is Associated with Reduced NMDA Receptor-Mediated Synaptic Signaling in Hippocampal Interneurons Expressing PV or CCK.Alzheimer's Transgenic Model Is Characterized by Very Early Brain Network Alterations and β-CTF Fragment Accumulation: Reversal by β-Secretase Inhibition.Autapses enhance bursting and coincidence detection in neocortical pyramidal cells
P2860
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P2860
Desynchronization of neocortical networks by asynchronous release of GABA at autaptic and synaptic contacts from fast-spiking interneurons.
description
2010 nî lūn-bûn
@nan
2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Desynchronization of neocortic ...... rom fast-spiking interneurons.
@ast
Desynchronization of neocortic ...... rom fast-spiking interneurons.
@en
type
label
Desynchronization of neocortic ...... rom fast-spiking interneurons.
@ast
Desynchronization of neocortic ...... rom fast-spiking interneurons.
@en
prefLabel
Desynchronization of neocortic ...... rom fast-spiking interneurons.
@ast
Desynchronization of neocortic ...... rom fast-spiking interneurons.
@en
P2093
P2860
P50
P1433
P1476
Desynchronization of neocortic ...... from fast-spiking interneurons
@en
P2093
David A Prince
Frédéric Manseau
Silvia Marinelli
P2860
P356
10.1371/JOURNAL.PBIO.1000492
P407
P577
2010-09-28T00:00:00Z