Effect of CD14 blockade on endotoxin-induced acute lung injury in mice. - Wikidata - awgldk - TriplyDB

Effect of CD14 blockade on endotoxin-induced acute lung injury in mice.

Effect of CD14 blockade on endotoxin-induced acute lung injury in mice.

http://www.wikidata.org/entity/Q44222630

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Polymorphisms in the CD14 gene associated with pulmonary function in farmers Toll-like receptor 4 decoy, TOY, attenuates gram-negative bacterial sepsis.Role of CD14 in a mouse model of acute lung inflammation induced by different lipopolysaccharide chemotypes.Distinct roles of pattern recognition receptors CD14 and Toll-like receptor 4 in acute lung injury Neutrophil extracellular traps directly induce epithelial and endothelial cell death: a predominant role of histones A nonsynonymous polymorphism of IRAK4 associated with increased prevalence of gram-positive infection and decreased response to toll-like receptor ligands Role of toll-like receptor 4 in acute neutrophilic lung inflammation induced by intratracheal bacterial products in mice.Protective role of vascular endothelial growth factor in endotoxin-induced acute lung injury in mice.Commensal Microbe-specific Activation of B2 Cell Subsets Contributes to Atherosclerosis Development Independently of Lipid Metabolism Role of CD14 in responses to clinical isolates of Escherichia coli: effects of K1 capsule expression Endotoxin receptor CD14 in PiZ alpha-1-antitrypsin deficiency individuals.Role of CD14 in lung inflammation and infection.The role of monocytes and inflammation in the pathophysiology of heart failure.Biological therapies in the acute respiratory distress syndrome.Murine macrophage transcriptional responses to Bacillus anthracis infection and intoxication.Lipopolysaccharide interaction with cell surface Toll-like receptor 4-MD-2: higher affinity than that with MD-2 or CD14.Role of soluble receptor for advanced glycation end products on endotoxin-induced lung injury.Effects of inhaled carbon monoxide on acute lung injury in mice.Gene expression of the concentration-sensitive sodium channel is suppressed in lipopolysaccharide-induced acute lung injury in mice.Enhancement of the endotoxin recognition pathway by ventilation with a large tidal volume in rabbits.CD14 is an essential mediator of LPS-induced airway disease.

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P2860

Effect of CD14 blockade on endotoxin-induced acute lung injury in mice.

http://www.wikidata.org/entity/Q44222630

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年學術文章

@yue

2003年學術文章

@zh-hant

name

Effect of CD14 blockade on endotoxin-induced acute lung injury in mice.

@en

Effect of CD14 blockade on endotoxin-induced acute lung injury in mice.

@nl

type

label

Effect of CD14 blockade on endotoxin-induced acute lung injury in mice.

@en

Effect of CD14 blockade on endotoxin-induced acute lung injury in mice.

@nl

prefLabel

Effect of CD14 blockade on endotoxin-induced acute lung injury in mice.

@en

Effect of CD14 blockade on endotoxin-induced acute lung injury in mice.

@nl

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RCMB.2002-0132OC

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American Journal of Respiratory Cell and Molecular Biology

P1476

Effect of CD14 blockade on endotoxin-induced acute lung injury in mice.

@en

P2093

Akitoshi Ishizaka

http://www.w3.org/2001/XMLSchema#string

Hidefumi Koh

http://www.w3.org/2001/XMLSchema#string

Kazuhiro Yamaguchi

http://www.w3.org/2001/XMLSchema#string

Mie Shimizu

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Naoki Hasegawa

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Sadatomo Tasaka

http://www.w3.org/2001/XMLSchema#string

Wakako Yamada

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Yoshiyuki Adachi

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P2860

Resistance to endotoxin shock and reduced dissemination of gram-negative bacteria in CD14-deficient mice

CD14, a receptor for complexes of lipopolysaccharide (LPS) and LPS binding protein

Recognition of gram-negative bacteria and endotoxin by the innate immune system.

Nitric oxide in septic shock.

Lung cytokines and ARDS: Roger S. Mitchell Lecture.

Role of plasma, lipopolysaccharide-binding protein, and CD14 in response of mouse peritoneal exudate macrophages to endotoxin

Critical role of CD14 for production of proinflammatory cytokines and cytokine inhibitors during sepsis with failure to alter morbidity or mortality

Acute respiratory distress in adults.

The toxicology of inhaled nitric oxide.

Function of lipopolysaccharide (LPS)-binding protein (LBP) and CD14, the receptor for LPS/LBP complexes: a short review.

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252-258

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10.1165/RCMB.2002-0132OC

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2

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29

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2003-03-14T00:00:00Z

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12639839

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