Cutting edge: Toll-like receptor (TLR)2- and TLR4-mediated pathogen recognition in resistance to airborne infection with Mycobacterium tuberculosis.
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Neutrophils Promote Mycobacterial Trehalose Dimycolate-Induced Lung Inflammation via the Mincle PathwayExecution of macrophage apoptosis by PE_PGRS33 of Mycobacterium tuberculosis is mediated by Toll-like receptor 2-dependent release of tumor necrosis factor-alphaNOD2 and toll-like receptors are nonredundant recognition systems of Mycobacterium tuberculosisInnate Immune Defenses in Human Tuberculosis: An Overview of the Interactions between Mycobacterium tuberculosis and Innate Immune CellsTregs in infection and vaccinology: heroes or traitors?Mycobacterium tuberculosis Strains Are Differentially Recognized by TLRs with an Impact on the Immune ResponseSensing of Mycobacterium tuberculosis and consequences to both host and bacillusSuppression of the NF-κB pathway by diesel exhaust particles impairs human antimycobacterial immunityDeletion of the Mycobacterium tuberculosis resuscitation-promoting factor Rv1009 gene results in delayed reactivation from chronic tuberculosisThe SecA2 secretion factor of Mycobacterium tuberculosis promotes growth in macrophages and inhibits the host immune responseToll-like receptor 2 modulates the proinflammatory milieu in Staphylococcus aureus-induced brain abscessA murine DC-SIGN homologue contributes to early host defense against Mycobacterium tuberculosisInvolvement of CD14, toll-like receptors 2 and 4, and MyD88 in the host response to the fungal pathogen Cryptococcus neoformans in vivoFull-exon resequencing reveals toll-like receptor variants contribute to human susceptibility to tuberculosis disease.Innate gene repression associated with Mycobacterium bovis infection in cattle: toward a gene signature of diseaseAntigen stimulation of peripheral blood mononuclear cells from Mycobacterium bovis infected cattle yields evidence for a novel gene expression program.TLR2 polymorphisms, Arg753Gln and Arg677Trp, are not associated with increased burden of tuberculosis in Indian patients.The role of scavenger receptor B1 in infection with Mycobacterium tuberculosis in a murine model.Innate defects of the IL-12/IFN-γ axis in susceptibility to infections by mycobacteria and salmonellaMHC Ib molecule Qa-1 presents Mycobacterium tuberculosis peptide antigens to CD8+ T cells and contributes to protection against infection.Effects of the TLR2 agonists MALP-2 and Pam3Cys in isolated mouse lungsIdentification of an Immunomodulating Agent from Mycobacterium lepraeThe adaptor molecule CARD9 is essential for tuberculosis control.Mycobacterium tuberculosis, macrophages, and the innate immune response: does common variation matter?The role of TLR2, TRL3, TRL4, and TRL9 signaling in the pathogenesis of autoimmune disease in a retinal autoimmunity modelTLR2 engagement on CD4(+) T cells enhances effector functions and protective responses to Mycobacterium tuberculosis.TLR-2 independent recognition of Mycobacterium tuberculosis by CD11c+ pulmonary cells from old miceRegulation of antigen presentation by Mycobacterium tuberculosis: a role for Toll-like receptorsGenome-wide transcriptional profiling of peripheral blood leukocytes from cattle infected with Mycobacterium bovis reveals suppression of host immune genes.Association of TLR polymorphisms with development of tuberculosis in Indonesian females.Human TOLLIP regulates TLR2 and TLR4 signaling and its polymorphisms are associated with susceptibility to tuberculosis.Mycobacterium tuberculosis lipoproteins directly regulate human memory CD4(+) T cell activation via Toll-like receptors 1 and 2.Toll-like receptor 2 is required for inflammatory responses to Francisella tularensis LVSToll-like receptor-induced arginase 1 in macrophages thwarts effective immunity against intracellular pathogens.Transcriptome-based network analysis reveals a spectrum model of human macrophage activation.Inhibition of nuclear factor-kappa B activation decreases survival of Mycobacterium tuberculosis in human macrophages.Genetically determined susceptibility to tuberculosis in mice causally involves accelerated and enhanced recruitment of granulocytes.Development of a secondary immune response to Mycobacterium tuberculosis is independent of Toll-like receptor 2.Host defense and recruitment of Foxp3⁺ T regulatory cells to the lungs in chronic Mycobacterium tuberculosis infection requires toll-like receptor 2Human lung immunity against Mycobacterium tuberculosis: insights into pathogenesis and protection.
P2860
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P2860
Cutting edge: Toll-like receptor (TLR)2- and TLR4-mediated pathogen recognition in resistance to airborne infection with Mycobacterium tuberculosis.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Cutting edge: Toll-like recept ...... th Mycobacterium tuberculosis.
@en
Cutting edge: Toll-like receptor
@nl
type
label
Cutting edge: Toll-like recept ...... th Mycobacterium tuberculosis.
@en
Cutting edge: Toll-like receptor
@nl
prefLabel
Cutting edge: Toll-like recept ...... th Mycobacterium tuberculosis.
@en
Cutting edge: Toll-like receptor
@nl
P2093
P1476
Cutting edge: Toll-like recept ...... th Mycobacterium tuberculosis.
@en
P2093
Alexandra Fehrenbach
Carsten J Kirschning
Christoph Hölscher
Sanna Goyert
Stefan Ehlers
Svenja Kröger
P304
P356
10.4049/JIMMUNOL.169.7.3480
P407
P577
2002-10-01T00:00:00Z