Bystander activation of CD4(+) T cells can represent an exclusive means of immunopathology in a virus infection.
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Galectin-9/TIM-3 interaction regulates virus-specific primary and memory CD8 T cell responsePathogenesis of herpes simplex virus-induced ocular immunoinflammatory lesions in B-cell-deficient mice.Tegument-specific, virus-reactive CD4 T cells localize to the cornea in herpes simplex virus interstitial keratitis in humans.Herpes simplex virus-induced keratitis: evaluation of the role of molecular mimicry in lesion pathogenesis.CD154 signaling regulates the Th1 response to herpes simplex virus-1 and inflammation in infected corneasHerpes keratitis.Mapping of genes involved in murine herpes simplex virus keratitis: identification of genes and their modifiers.Cell surface major histocompatibility complex class II proteins are regulated by the products of the gamma(1)34.5 and U(L)41 genes of herpes simplex virus 1.Of mice and not humans: how reliable are animal models for evaluation of herpes CD8(+)-T cell-epitopes-based immunotherapeutic vaccine candidates?Identification of novel immunodominant CD4+ Th1-type T-cell peptide epitopes from herpes simplex virus glycoprotein D that confer protective immunity.Immunodominant "asymptomatic" herpes simplex virus 1 and 2 protein antigens identified by probing whole-ORFome microarrays with serum antibodies from seropositive asymptomatic versus symptomatic individualsThe role of bystander T cells in CNS pathology and pathogen clearance.Viruses and autoimmunity.Herpes simplex virus type 1 DNA is immunostimulatory in vitro and in vivo.CD8 T cells mediate transient herpes stromal keratitis in CD4-deficient miceEnhanced viral immunoinflammatory lesions in mice lacking IL-23 responses.In vitro-generated antigen-specific CD4+ CD25+ Foxp3+ regulatory T cells control the severity of herpes simplex virus-induced ocular immunoinflammatory lesions.Non-mitogenic anti-CD3F(ab')2 monoclonal antibody: a novel approach to control herpetic stromal keratitis.Rebuilding an immune-mediated central nervous system disease: weighing the pathogenicity of antigen-specific versus bystander T cells.Lymphocytes have a role in protection, but not in pathogenesis, during La Crosse Virus infection in mice.Role for 3-O-sulfated heparan sulfate as the receptor for herpes simplex virus type 1 entry into primary human corneal fibroblasts.Herpesvirus Entry Mediator and Ocular Herpesvirus Infection: More than Meets the Eye.Application of FGF-2 to modulate herpetic stromal keratitis.Elucidating the protective and pathologic T cell species in the virus-induced corneal immunoinflammatory condition herpetic stromal keratitis.Bystander stimulation of activated CD4+ T cells of unrelated specificity following a booster vaccination with tetanus toxoid.
P2860
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P2860
Bystander activation of CD4(+) T cells can represent an exclusive means of immunopathology in a virus infection.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh-hant
name
Bystander activation of CD4
@nl
Bystander activation of CD4(+) ...... athology in a virus infection.
@en
type
label
Bystander activation of CD4
@nl
Bystander activation of CD4(+) ...... athology in a virus infection.
@en
prefLabel
Bystander activation of CD4
@nl
Bystander activation of CD4(+) ...... athology in a virus infection.
@en
P2093
P2860
P1476
Bystander activation of CD4(+) ...... athology in a virus infection.
@en
P2093
Deshpande SP
Gangappa S
P2860
P304
P356
10.1002/(SICI)1521-4141(199911)29:11<3674::AID-IMMU3674>3.0.CO;2-7
P407
P577
1999-11-01T00:00:00Z