Changes and regulation of the C5a receptor on neutrophils during septic shock in humans. - Wikidata - awgldk - TriplyDB

Changes and regulation of the C5a receptor on neutrophils during septic shock in humans.

Changes and regulation of the C5a receptor on neutrophils during septic shock in humans.

http://www.wikidata.org/entity/Q44402512

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Activated Complement Factors as Disease Markers for Sepsis Moving towards a paradigm: common mechanisms of chemotactic signaling in Dictyostelium and mammalian leukocytes The significance and regulatory mechanisms of innate immune cells in the development of sepsis Pentraxins: structure, function, and role in inflammation Properdin levels in human sepsis.Complement C3 and C5 deficiency affects fracture healing Differential regulation of C5a receptor 1 in innate immune cells during the allergic asthma effector phase Complement 5a receptor-mediated neutrophil dysfunction is associated with a poor outcome in sepsis.Anti-inflammatory interventions-what has worked, not worked, and what may work in the future.Assessment of interleukin-17A, C5a and RANTES for early diagnosis of neonatal sepsis - a preliminary study Complement C5a-Induced Changes in Neutrophil Morphology During Inflammation.Complement C5a Functions as a Master Switch for the pH Balance in Neutrophils Exerting Fundamental Immunometabolic Effects.The immune system's role in sepsis progression, resolution, and long-term outcome.Complement Destabilizes Cardiomyocyte Function In Vivo after Polymicrobial Sepsis and In Vitro.Double blockade of CD14 and complement C5 abolishes the cytokine storm and improves morbidity and survival in polymicrobial sepsis in mice.Complement C5a induces PD-L1 expression and acts in synergy with LPS through Erk1/2 and JNK signaling pathways.Diabetes and Sepsis: Risk, Recurrence, and Ruination.Complement C5a Receptor 1 Exacerbates the Pathophysiology of N. meningitidis Sepsis and Is a Potential Target for Disease Treatment.Auxiliary activation of the complement system and its importance for the pathophysiology of clinical conditions.Extracellular Vesicles: Packages Sent With Complement.Complement system modulation as a target for treatment of arrhythmogenic cardiomyopathy.Neutrophils in Tissue Trauma of the Skin, Bone, and Lung: Two Sides of the Same Coin.Cell-surface signatures of immune dysfunction risk-stratify critically ill patients: INFECT study.Anaphylatoxin Receptors C3aR and C5aR1 Are Important Factors That Influence the Impact of Ethanol on the Adipose Secretome Complement After Trauma: Suturing Innate and Adaptive Immunity

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Changes and regulation of the C5a receptor on neutrophils during septic shock in humans.

http://www.wikidata.org/entity/Q44402512

description

2013 nî lūn-bûn

@nan

2013年の論文

@ja

2013年学术文章

@wuu

2013年学术文章

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2013年学术文章

@zh-cn

2013年学术文章

@zh-hans

2013年学术文章

@zh-my

2013年学术文章

@zh-sg

2013年學術文章

@yue

2013年學術文章

@zh-hant

name

Changes and regulation of the C5a receptor on neutrophils during septic shock in humans.

@en

Changes and regulation of the C5a receptor on neutrophils during septic shock in humans.

@nl

type

label

Changes and regulation of the C5a receptor on neutrophils during septic shock in humans.

@en

Changes and regulation of the C5a receptor on neutrophils during septic shock in humans.

@nl

prefLabel

Changes and regulation of the C5a receptor on neutrophils during septic shock in humans.

@en

Changes and regulation of the C5a receptor on neutrophils during septic shock in humans.

@nl

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JIMMUNOL.1200534

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Journal of Immunology

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Changes and regulation of the C5a receptor on neutrophils during septic shock in humans.

@en

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Florian Gebhard

http://www.w3.org/2001/XMLSchema#string

Heike Unnewehr

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Holger Barth

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J Vidya Sarma

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Manfred Weiss

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Mario Perl

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Marion E Schneider

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Michael A Flierl

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Michael Mihlan

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Peter N Monk

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4215-4225

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scholarly article

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10.4049/JIMMUNOL.1200534

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8

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190

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Markus S Huber-Lang

Daniel Rittirsch

Firas S Zetoune

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2013-03-11T00:00:00Z

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23479227

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