Primary fibrinolysis is integral in the pathogenesis of the acute coagulopathy of trauma. - Wikidata - awgldk - TriplyDB

Primary fibrinolysis is integral in the pathogenesis of the acute coagulopathy of trauma.

Primary fibrinolysis is integral in the pathogenesis of the acute coagulopathy of trauma.

http://www.wikidata.org/entity/Q44405049

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Viscoelastic Methods of Blood Clotting Assessment - A Multidisciplinary Review Early and individualized goal-directed therapy for trauma-induced coagulopathy Thrombelastography (TEG®): practical considerations on its clinical use in trauma resuscitation Bleeding related to disturbed fibrinolysis TACTIC: Trans-Agency Consortium for Trauma-Induced Coagulopathy Discrepant fibrinolytic response in plasma and whole blood during experimental endotoxemia in healthy volunteers Effect of thromboelastography (TEG®) and rotational thromboelastometry (ROTEM®) on diagnosis of coagulopathy, transfusion guidance and mortality in trauma: descriptive systematic review Criteria for empiric treatment of hyperfibrinolysis after trauma.Updated concepts on the pathophysiology and the clinical management of trauma hemorrhage and coagulopathy.Fibrinolysis greater than 3% is the critical value for initiation of antifibrinolytic therapy.Hyperfibrinolysis and Hypofibrinogenemia Diagnosed With Rotational Thromboelastometry in Dogs Naturally Infected With Angiostrongylus vasorum.Kinetic model facilitates analysis of fibrin generation and its modulation by clotting factors: implications for hemostasis-enhancing therapies.Trauma-Induced Coagulopathy: An Institution's 35 Year Perspective on Practice and Research.Trauma hemostasis and oxygenation research position paper on remote damage control resuscitation: definitions, current practice, and knowledge gaps Impact of β-adrenoceptor blockade on systemic inflammation and coagulation disturbances in rats with acute traumatic coagulopathy Thrombelastographic pattern recognition in renal disease and trauma Hyperfibrinolysis, physiologic fibrinolysis, and fibrinolysis shutdown: the spectrum of postinjury fibrinolysis and relevance to antifibrinolytic therapy Reduced clot strength upon admission, evaluated by thrombelastography (TEG), in trauma patients is independently associated with increased 30-day mortality Plasma is the physiologic buffer of tissue plasminogen activator-mediated fibrinolysis: rationale for plasma-first resuscitation after life-threatening hemorrhage.Thrombelastography and biomarker profiles in acute coagulopathy of trauma: a prospective study.Postinjury fibrinolysis shutdown: Rationale for selective tranexamic acid Thromboelastography values from pigtail macaques ( Macaca nemestrina): effects of age and sex Early platelet dysfunction: an unrecognized role in the acute coagulopathy of trauma.Bleeding complications during cardiac electronic device implantation in patients receiving antithrombotic therapy: is there any value of local tranexamic acid?FIBTEM provides early prediction of massive transfusion in trauma Clinical review: Canadian National Advisory Committee on Blood and Blood Products--Massive transfusion consensus conference 2011: report of the panel.Whole blood thromboelastometry: another Knight at the Roundtable?TEG® and ROTEM® in trauma: similar test but different results?Design of the Study of Tranexamic Acid during Air Medical Prehospital Transport (STAAMP) Trial: Addressing the Knowledge Gaps.The "Death Diamond": Rapid thrombelastography identifies lethal hyperfibrinolysis.Shock-induced systemic hyperfibrinolysis is attenuated by plasma-first resuscitation.Overwhelming tPA release, not PAI-1 degradation, is responsible for hyperfibrinolysis in severely injured trauma patients.Base deficit as a marker of survival after traumatic injury: consistent across changing patient populations and resuscitation paradigms Viscoelastic measurements of platelet function, not fibrinogen function, predicts sensitivity to tissue-type plasminogen activator in trauma patients.Blockade of Extracellular High-Mobility Group Box 1 Attenuates Systemic Inflammation and Coagulation Abnormalities in Rats with Acute Traumatic Coagulopathy.Impact of fibrinogen concentrate alone or with prothrombin complex concentrate (+/- fresh frozen plasma) on plasma fibrinogen level and fibrin-based clot strength (FIBTEM) in major trauma: a retrospective study Impact of kidney donor hemostasis on risk of complications after transplantation--preliminary outcomes.Activated Protein C Drives the Hyperfibrinolysis of Acute Traumatic Coagulopathy.Targeted Thromboelastographic (TEG) Blood Component and Pharmacologic Hemostatic Therapy in Traumatic and Acquired Coagulopathy.Current management of massive hemorrhage in trauma.

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P2860

Primary fibrinolysis is integral in the pathogenesis of the acute coagulopathy of trauma.

http://www.wikidata.org/entity/Q44405049

description

2010 nî lūn-bûn

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2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

@zh-my

2010年学术文章

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2010年學術文章

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2010年學術文章

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name

Primary fibrinolysis is integral in the pathogenesis of the acute coagulopathy of trauma.

@en

Primary fibrinolysis is integral in the pathogenesis of the acute coagulopathy of trauma.

@nl

type

label

Primary fibrinolysis is integral in the pathogenesis of the acute coagulopathy of trauma.

@en

Primary fibrinolysis is integral in the pathogenesis of the acute coagulopathy of trauma.

@nl

prefLabel

Primary fibrinolysis is integral in the pathogenesis of the acute coagulopathy of trauma.

@en

Primary fibrinolysis is integral in the pathogenesis of the acute coagulopathy of trauma.

@nl

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Annals of Surgery

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Primary fibrinolysis is integral in the pathogenesis of the acute coagulopathy of trauma.

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Angela Sauaia

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Carlton Barnett

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Clay C Burlew

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Ernest E Moore

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Jeffrey L Johnson

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Jeffry L Kashuk

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Max Wohlauer

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Michael Pezold

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Michael Sawyer

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Walter L Biffl

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P304

434-42; discussion 443-4

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scholarly article

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10.1097/SLA.0B013E3181F09191

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3

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252

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2010-09-01T00:00:00Z

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20739843

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