Alternative splicing and differential expression of the islet autoantigen IGRP between pancreas and thymus contributes to immunogenicity of pancreatic islets but not diabetogenicity in humans.
about
Where, How, and When: Positioning Posttranslational Modification Within Type 1 Diabetes PathogenesisPositive and negative selection of the T cell repertoire: what thymocytes see (and don't see)The IL-2/IL-2R system: from basic science to therapeutic applications to enhance immune regulation.Self-antigen expression in the peripheral immune system: roles in self-tolerance and type 1 diabetes pathogenesis.Autoreactive T cells in type 1 diabetes.
P2860
Alternative splicing and differential expression of the islet autoantigen IGRP between pancreas and thymus contributes to immunogenicity of pancreatic islets but not diabetogenicity in humans.
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2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
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2013年学术文章
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name
Alternative splicing and diffe ...... not diabetogenicity in humans.
@en
Alternative splicing and diffe ...... not diabetogenicity in humans.
@nl
type
label
Alternative splicing and diffe ...... not diabetogenicity in humans.
@en
Alternative splicing and diffe ...... not diabetogenicity in humans.
@nl
prefLabel
Alternative splicing and diffe ...... not diabetogenicity in humans.
@en
Alternative splicing and diffe ...... not diabetogenicity in humans.
@nl
P2093
P2860
P1433
P1476
Alternative splicing and diffe ...... not diabetogenicity in humans.
@en
P2093
Annemarie A Verrijn Stuart
Bart O Roep
Bianca Blom
Frank J T Staal
Frans K Gorus
Joana R F Abreu
Katrijn Verhaeghen
Marten A Engelse
V Martijn de Jong
P2860
P2888
P304
P356
10.1007/S00125-013-3034-6
P577
2013-09-13T00:00:00Z