Infection of neutrophil granulocytes with Leishmania major activates ERK 1/2 and modulates multiple apoptotic pathways to inhibit apoptosis. - Wikidata - awgldk - TriplyDB

Infection of neutrophil granulocytes with Leishmania major activates ERK 1/2 and modulates multiple apoptotic pathways to inhibit apoptosis.

Infection of neutrophil granulocytes with Leishmania major activates ERK 1/2 and modulates multiple apoptotic pathways to inhibit apoptosis.

http://www.wikidata.org/entity/Q44497128

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Rapid Sequestration of Leishmania mexicana by Neutrophils Contributes to the Development of Chronic Lesion Lipophosphoglycans from Leishmania amazonensis Strains Display Immunomodulatory Properties via TLR4 and Do Not Affect Sand Fly Infection Influence of Microbes on Neutrophil Life and Death.Deception and manipulation: the arms of leishmania, a successful parasite Exposure to Leishmania braziliensis triggers neutrophil activation and apoptosis.Simultaneous transcriptional profiling of Leishmania major and its murine macrophage host cell reveals insights into host-pathogen interactions.Increased leucocyte apoptosis in transfused β-thalassaemia patients.Enhanced survival of Leishmania major in neutrophil granulocytes in the presence of apoptotic cells.Exploring the role of medicinal plant-based immunomodulators for effective therapy of leishmaniasis.Regulation of human neutrophil apoptosis and lifespan in health and disease.Annexin A1 Is Involved in the Resolution of Inflammatory Responses during Leishmania braziliensis Infection.Leishmania donovani infection drives the priming of human monocyte-derived dendritic cells during Plasmodium falciparum co-infections.The anti-apoptotic Bcl-2 family protein A1/Bfl-1 regulates neutrophil survival and homeostasis and is controlled via PI3K and JAK/STAT signaling.Leishmania mexicana amastigotes inhibit p38 and JNK and activate PI3K/AKT: role in the inhibition of apoptosis of dendritic cells.Neutrophil extracellular traps release induced by Leishmania: role of PI3Kγ, ERK, PI3Kσ, PKC, and [Ca2+].Survival Mechanisms Used by Some Leishmania Species to Escape Neutrophil Killing.NOX2-Derived Reactive Oxygen Species Control Inflammation during Leishmania amazonensis Infection by Mediating Infection-Induced Neutrophil Apoptosis.Caffeic acid combined with autoclaved Leishmania major boosted the protection of infected BALB/c mice by enhancing IgG2 production, IFN-γ/TGF-β and iNO synthase/arginase1 ratios, and the death of infected phagocytes.Leishmania Hijacks Myeloid Cells for Immune Escape.

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Infection of neutrophil granulocytes with Leishmania major activates ERK 1/2 and modulates multiple apoptotic pathways to inhibit apoptosis.

http://www.wikidata.org/entity/Q44497128

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2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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Infection of neutrophil granul ...... pathways to inhibit apoptosis.

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Infection of neutrophil granul ...... pathways to inhibit apoptosis.

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Infection of neutrophil granul ...... pathways to inhibit apoptosis.

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Infection of neutrophil granul ...... pathways to inhibit apoptosis.

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Infection of neutrophil granul ...... pathways to inhibit apoptosis.

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Infection of neutrophil granul ...... pathways to inhibit apoptosis.

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Medical Microbiology and Immunology

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Infection of neutrophil granul ...... pathways to inhibit apoptosis.

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Arup Sarkar

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Asima Bhattacharyya

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Eresso Aga

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Lars Hellberg

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Martina Behnen

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Sonja Möller

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Tamás Laskay

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Uta Bussmeyer

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P2860

Akt promotes cell survival by phosphorylating and inhibiting a Forkhead transcription factor

Toxoplasma gondii infection confers resistance against BimS-induced apoptosis by preventing the activation and mitochondrial targeting of pro-apoptotic Bax

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

Aspirin-triggered lipoxins override the apoptosis-delaying action of serum amyloid A in human neutrophils: a novel mechanism for resolution of inflammation

Insights into the regulatory mechanism for caspase-8 activation

MAPK-activated protein kinase-2 participates in p38 MAPK-dependent and ERK-dependent functions in human neutrophils

Bid induces the oligomerization and insertion of Bax into the outer mitochondrial membrane

The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

BH3 domains of BH3-only proteins differentially regulate Bax-mediated mitochondrial membrane permeabilization both directly and indirectly

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