Increased kidney metabolism as a pathway to kidney tissue hypoxia and damage: effects of triiodothyronine and dinitrophenol in normoglycemic rats.
about
Hypoxia: The Force that Drives Chronic Kidney DiseaseHypertension: a problem of organ blood flow supply-demand mismatch.Targeted mitochondrial therapy using MitoQ shows equivalent renoprotection to angiotensin converting enzyme inhibition but no combined synergy in diabetes.Renal transplantation induces mitochondrial uncoupling, increased kidney oxygen consumption, and decreased kidney oxygen tension.Renal hypoxia in kidney disease: Cause or consequence?
P2860
Increased kidney metabolism as a pathway to kidney tissue hypoxia and damage: effects of triiodothyronine and dinitrophenol in normoglycemic rats.
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2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
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name
Increased kidney metabolism as ...... ophenol in normoglycemic rats.
@en
Increased kidney metabolism as ...... ophenol in normoglycemic rats.
@nl
type
label
Increased kidney metabolism as ...... ophenol in normoglycemic rats.
@en
Increased kidney metabolism as ...... ophenol in normoglycemic rats.
@nl
prefLabel
Increased kidney metabolism as ...... ophenol in normoglycemic rats.
@en
Increased kidney metabolism as ...... ophenol in normoglycemic rats.
@nl
P2093
P50
P1476
Increased kidney metabolism as ...... rophenol in normoglycemic rats
@en
P2093
Angelica Fasching
Fredrik Palm
Malou Friederich-Persson
P356
10.1007/978-1-4614-7411-1_2
P407
P577
2013-01-01T00:00:00Z