Sublethal heat treatment promotes epithelial-mesenchymal transition and enhances the malignant potential of hepatocellular carcinoma. - Wikidata - awgldk - TriplyDB

Sublethal heat treatment promotes epithelial-mesenchymal transition and enhances the malignant potential of hepatocellular carcinoma.

Sublethal heat treatment promotes epithelial-mesenchymal transition and enhances the malignant potential of hepatocellular carcinoma.

http://www.wikidata.org/entity/Q44536329

about

Incomplete radiofrequency ablation enhances invasiveness and metastasis of residual cancer of hepatocellular carcinoma cell HCCLM3 via activating β-catenin signaling Radiofrequency Ablation versus Liver Resection for Colorectal Cancer Liver Metastasis: An Updated Systematic Review and Meta-analysis Activated hepatic stellate cells secrete periostin to induce stem cell-like phenotype of residual hepatocellular carcinoma cells after heat treatment.Long noncoding RNA expression profiles in sub-lethal heat-treated hepatoma carcinoma cells.Aging impairs induction of redox factor-1 after heat stress: a potential mechanism for heat-induced liver injury.Sorafenib suppresses the epithelial-mesenchymal transition of hepatocellular carcinoma cells after insufficient radiofrequency ablation.A Comparison between Three-Dimensional Visualization Guided Hepatectomy and Ultrasonography Guided Radiofrequency Ablation in the Treatment of Small Hepatocellular Carcinoma within the Milan Criteria.Elevated MTSS1 expression associated with metastasis and poor prognosis of residual hepatitis B-related hepatocellular carcinoma.Utility of the dual-specificity protein kinase TTK as a therapeutic target for intrahepatic spread of liver cancer Insufficient radiofrequency ablation promotes epithelial-mesenchymal transition of hepatocellular carcinoma cells through Akt and ERK signaling pathways.Radiofrequency hyperthermia-enhanced herpes simplex virus-thymidine kinase/ganciclovir direct intratumoral gene therapy of hepatocellular carcinoma.Pathophysiology of recurrent hepatocellular carcinoma after radiofrequency ablation.Inflammation and cancer: inhibiting the progression of residual hepatic VX2 carcinoma by anti-inflammatory drug after incomplete radiofrequency ablation.Increased matrix stiffness promotes tumor progression of residual hepatocellular carcinoma after insufficient heat treatment.Effect of a hypoxic microenvironment after radiofrequency ablation on residual hepatocellular cell migration and invasion.microRNA-526b servers as a prognostic factor and exhibits tumor suppressive property by targeting Sirtuin 7 in hepatocellular carcinoma.Tumour promotion versus tumour suppression in chronic hepatic iron overload.Insufficient microwave ablation-induced promotion of distant metastasis is suppressed by β-catenin pathway inhibition in breast cancer.Orthotopic hepatic cancer: radiofrequency hyperthermia-enhanced intratumoral herpes simplex virus-thymidine kinase gene therapy.Tumour eradication using synchronous thermal ablation and Hsp90 chemotherapy with protein engineered triblock biopolymer-geldanamycin conjugates.Extracellular matrix collagen I promotes the tumor progression of residual hepatocellular carcinoma after heat treatment

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P2860

Sublethal heat treatment promotes epithelial-mesenchymal transition and enhances the malignant potential of hepatocellular carcinoma.

http://www.wikidata.org/entity/Q44536329

description

2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

@zh-my

2013年学术文章

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2013年學術文章

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2013年學術文章

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name

Sublethal heat treatment promo ...... l of hepatocellular carcinoma.

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Sublethal heat treatment promo ...... l of hepatocellular carcinoma.

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type

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Sublethal heat treatment promo ...... l of hepatocellular carcinoma.

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Sublethal heat treatment promo ...... l of hepatocellular carcinoma.

@nl

prefLabel

Sublethal heat treatment promo ...... l of hepatocellular carcinoma.

@en

Sublethal heat treatment promo ...... l of hepatocellular carcinoma.

@nl

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Sublethal heat treatment promo ...... l of hepatocellular carcinoma.

@en

P2093

Detlef Schuppan

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Moritz Schmelzle

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Naoki Ikenaga

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Ryota Masuzaki

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Shuhei Yoshida

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Simon C Robson

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Yan Wu

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P2860

Use of signal specific receptor tyrosine kinase oncoproteins reveals that pathways downstream from Grb2 or Shc are sufficient for cell transformation and metastasis

Tetraspanin CD81 is linked to ERK/MAPKinase signaling by Shc in liver tumor cells

A positive feedback loop of ER-α36/EGFR promotes malignant growth of ER-negative breast cancer cells

Induction of EMT by twist proteins as a collateral effect of tumor-promoting inactivation of premature senescence

A novel transforming protein (SHC) with an SH2 domain is implicated in mitogenic signal transduction

Management of hepatocellular carcinoma: An update

Epithelial-mesenchymal transitions in development and disease

Insufficient radiofrequency ablation promotes angiogenesis of residual hepatocellular carcinoma via HIF-1α/VEGFA

The p66shc adaptor protein controls oxidative stress response and life span in mammals

Association of the Shc and Grb2/Sem5 SH2-containing proteins is implicated in activation of the Ras pathway by tyrosine kinases

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1667-1680

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10.1002/HEP.26526

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5

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58

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Miroslaw Kornek

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2013-09-19T00:00:00Z

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237013675

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23729316

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