Shear stress regulates endothelial nitric-oxide synthase promoter activity through nuclear factor kappaB binding. - Wikidata - awgldk - TriplyDB

Shear stress regulates endothelial nitric-oxide synthase promoter activity through nuclear factor kappaB binding.

Shear stress regulates endothelial nitric-oxide synthase promoter activity through nuclear factor kappaB binding.

http://www.wikidata.org/entity/Q44626801

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Sulfonylurea receptor 1 in central nervous system injury: a focused review S-nitrosylation in cardiovascular signaling Mechanosensitive mechanisms in transcriptional regulation Mechanisms of Amplified Arteriogenesis in Collateral Artery Segments Exposed to Reversed Flow Direction.20-hydroxy-5,8,11,14-eicosatetraenoic acid mediates endothelial dysfunction via IkappaB kinase-dependent endothelial nitric-oxide synthase uncoupling Activation of vascular endothelial nitric oxide synthase and heme oxygenase-1 expression by electrophilic nitro-fatty acids.Hemodynamic disturbed flow induces differential DNA methylation of endothelial Kruppel-Like Factor 4 promoter in vitro and in vivo.Exposure of the thorax to a sublethal blast wave causes a hydrodynamic pulse that leads to perivenular inflammation in the brain.Fluid Mechanics, Arterial Disease, and Gene Expression.Dynamic changes in endothelial cell adhesion molecule nepmucin/CD300LG expression under physiological and pathological conditions.Ox-LDL induces dysfunction of endothelial progenitor cells via activation of NF-κB.Phage-display-guided nanocarrier targeting to atheroprone vasculature.Hemorrhagic progression of a contusion after traumatic brain injury: a review.The role of shear stress in the pathogenesis of atherosclerosis.Relative reduction of endothelial nitric-oxide synthase expression and transcription in atherosclerosis-prone regions of the mouse aorta and in an in vitro model of disturbed flow.Sulfonylurea Receptor 1 in Humans with Post-Traumatic Brain Contusions.Glycocalyx Degradation Induces a Proinflammatory Phenotype and Increased Leukocyte Adhesion in Cultured Endothelial Cells under Flow Systems biology and biomechanical model of heart failure Physiological role of ROCKs in the cardiovascular system Endothelial mechanotransduction, nitric oxide and vascular inflammation.Endothelial Cell Dysfunction and the Pathobiology of Atherosclerosis In silico modeling of shear-stress-induced nitric oxide production in endothelial cells through systems biology NFKB1 promoter variation implicates shear-induced NOS3 gene expression and endothelial function in prehypertensives and stage I hypertensives.Regulation of endothelial nitric oxide synthase and postnatal angiogenesis by Rac1.Role of β-adrenergic receptors and nitric oxide signaling in exercise-mediated cardioprotection.Molecular basis for impaired collateral artery growth in the spontaneously hypertensive rat: insight from microarray analysis Molecular mechanisms responsible for the atheroprotective effects of laminar shear stress.Role of DNA methyltransferase 1 on the altered eNOS expression in human umbilical endothelium from intrauterine growth restricted fetuses.Mechanisms of exercise-induced cardioprotection.miRNAs: roles and clinical applications in vascular disease.Molecular mechanisms of muscle plasticity with exercise.Molecular biology of atherosclerosis.Influence of inflammation on cardiovascular protective effects of cytochrome P450 epoxygenase-derived epoxyeicosatrienoic acids.Shear stress regulated gene expression and angiogenesis in vascular endothelium.Role of NF-kappaB signaling in hepatocyte growth factor/scatter factor-mediated cell protection.Lamins at the crossroads of mechanosignaling Endothelial cell regulation of pulmonary vascular tone, inflammation, and coagulation.Hypoxemia, oxygen content, and the regulation of cerebral blood flow Coordinating Regulation of Gene Expression in Cardiovascular Disease: Interactions between Chromatin Modifiers and Transcription Factors.Activation of p300 histone acetyltransferase activity is an early endothelial response to laminar shear stress and is essential for stimulation of endothelial nitric-oxide synthase mRNA transcription.

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P2860

Shear stress regulates endothelial nitric-oxide synthase promoter activity through nuclear factor kappaB binding.

http://www.wikidata.org/entity/Q44626801

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

@zh-cn

2003年学术文章

@zh-hans

2003年学术文章

@zh-my

2003年学术文章

@zh-sg

2003年學術文章

@yue

2003年學術文章

@zh-hant

name

Shear stress regulates endothe ...... nuclear factor kappaB binding.

@en

Shear stress regulates endothe ...... nuclear factor kappaB binding.

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type

label

Shear stress regulates endothe ...... nuclear factor kappaB binding.

@en

Shear stress regulates endothe ...... nuclear factor kappaB binding.

@nl

prefLabel

Shear stress regulates endothe ...... nuclear factor kappaB binding.

@en

Shear stress regulates endothe ...... nuclear factor kappaB binding.

@nl

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Journal of Biological Chemistry

P1476

Shear stress regulates endothe ...... nuclear factor kappaB binding

@en

P2093

Alexis Cutchins

http://www.w3.org/2001/XMLSchema#string

David G Harrison

http://www.w3.org/2001/XMLSchema#string

Michael E Davis

http://www.w3.org/2001/XMLSchema#string

Tohru Fukai

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P2860

Mapping of the inducible IkappaB phosphorylation sites that signal its ubiquitination and degradation

Accurate transcription initiation by RNA polymerase II in a soluble extract from isolated mammalian nuclei

Signal transduction of eNOS activation

Functional analysis of the human endothelial nitric oxide synthase promoter. Sp1 and GATA factors are necessary for basal transcription in endothelial cells

Transcriptional and posttranscriptional regulation of endothelial nitric oxide synthase expression by hydrogen peroxide

Molecular cloning and characterization of the constitutive bovine aortic endothelial cell nitric oxide synthase.

Up-regulation of endothelial nitric-oxide synthase promoter by the phosphatidylinositol 3-kinase gamma /Janus kinase 2/MEK-1-dependent pathway.

Unidirectional and oscillatory shear stress differentially modulate NOS III gene expression.

Role of the NF-kappaB pathway in the pathogenesis of human disease states.

Databases on transcriptional regulation: TRANSFAC, TRRD and COMPEL

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163-168

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10.1074/JBC.M307528200

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1

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279

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Isabella M Grumbach

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2003-10-21T00:00:00Z

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14570928

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