Replication and plaque formation of mouse hepatitis virus (MHV-2) in mouse cell line DBT culture.
about
Cooperation of an RNA packaging signal and a viral envelope protein in coronavirus RNA packagingRole of polypyrimidine tract binding protein in the function of the hepatitis B virus posttranscriptional regulatory element.Role of Endocytosis and Low pH in Murine Hepatitis Virus Strain A59 Cell EntryA pregnancy-specific glycoprotein is expressed in the brain and serves as a receptor for mouse hepatitis virusSYNCRIP, a member of the heterogeneous nuclear ribonucleoprotein family, is involved in mouse hepatitis virus RNA synthesisRNase L-independent specific 28S rRNA cleavage in murine coronavirus-infected cellsPolypyrimidine tract-binding protein binds to the leader RNA of mouse hepatitis virus and serves as a regulator of viral transcriptionInduction of apoptosis in murine coronavirus-infected cultured cells and demonstration of E protein as an apoptosis inducer.Mouse hepatitis virus utilizes two carcinoembryonic antigens as alternative receptorsResistance to murine hepatitis virus strain 3 is dependent on production of nitric oxideImportance of the positive-strand RNA secondary structure of a murine coronavirus defective interfering RNA internal replication signal in positive-strand RNA synthesis.Coronavirus transcription early in infectionSubgenomic negative-strand RNA function during mouse hepatitis virus infection.Characterization of the coronavirus M protein and nucleocapsid interaction in infected cells.Polypyrimidine tract-binding protein binds to the complementary strand of the mouse hepatitis virus 3' untranslated region, thereby altering RNA conformation.Heterogeneous nuclear ribonucleoprotein a1 binds to the 3'-untranslated region and mediates potential 5'-3'-end cross talks of mouse hepatitis virus RNA.Resistance to highly virulent mouse hepatitis virus acquired by mice after low-virulence infection: enhanced antiviral activity of macrophages.Murine coronavirus requires lipid rafts for virus entry and cell-cell fusion but not for virus release.Murine coronavirus replication induces cell cycle arrest in G0/G1 phase.Murine coronavirus receptors are differentially expressed in the central nervous system and play virus strain-dependent roles in neuronal spreadRole of the mitochondrial signaling pathway in murine coronavirus-induced oligodendrocyte apoptosis.Coronaviruses resistant to a 3C-like protease inhibitor are attenuated for replication and pathogenesis, revealing a low genetic barrier but high fitness cost of resistanceSoluble receptor potentiates receptor-independent infection by murine coronavirus.RNA recombination of coronaviruses: localization of neutralizing epitopes and neuropathogenic determinants on the carboxyl terminus of peplomers.Heterogeneous nuclear ribonucleoprotein A1 regulates RNA synthesis of a cytoplasmic virusLCMV glycosylation modulates viral fitness and cell tropism.Nucleocapsid-independent specific viral RNA packaging via viral envelope protein and viral RNA signal.Simultaneous detection of antibodies to mouse hepatitis virus recombinant structural proteins by a microsphere-based multiplex fluorescence immunoassay.Comparative analysis of RNA genomes of mouse hepatitis viruses.Suppression of coronavirus replication by inhibition of the MEK signaling pathway.Induction of transcription factor Egr-1 gene expression in astrocytoma cells by Murine coronavirus infection.Murine coronavirus-induced oligodendrocyte apoptosis is mediated through the activation of the Fas signaling pathway.The effect of two closely inserted transcription consensus sequences on coronavirus transcription.Interaction of mouse hepatitis virus (MHV) spike glycoprotein with receptor glycoprotein MHVR is required for infection with an MHV strain that expresses the hemagglutinin-esterase glycoproteinInteractions between the cytoplasmic proteins and the intergenic (promoter) sequence of mouse hepatitis virus RNA: correlation with the amounts of subgenomic mRNA transcribed.Two murine coronavirus genes suffice for viral RNA synthesis.Characterization of a murine coronavirus defective interfering RNA internal cis-acting replication signal.The S2 subunit of the murine coronavirus spike protein is not involved in receptor binding.A 5'-proximal RNA sequence of murine coronavirus as a potential initiation site for genomic-length mRNA transcriptionMolecular anatomy of mouse hepatitis virus persistence: coevolution of increased host cell resistance and virus virulence.
P2860
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P2860
Replication and plaque formation of mouse hepatitis virus (MHV-2) in mouse cell line DBT culture.
description
1974 nî lūn-bûn
@nan
1974年の論文
@ja
1974年学术文章
@wuu
1974年学术文章
@zh
1974年学术文章
@zh-cn
1974年学术文章
@zh-hans
1974年学术文章
@zh-my
1974年学术文章
@zh-sg
1974年學術文章
@yue
1974年學術文章
@zh-hant
name
Replication and plaque formati ...... n mouse cell line DBT culture.
@en
Replication and plaque formation of mouse hepatitis virus
@nl
type
label
Replication and plaque formati ...... n mouse cell line DBT culture.
@en
Replication and plaque formation of mouse hepatitis virus
@nl
prefLabel
Replication and plaque formati ...... n mouse cell line DBT culture.
@en
Replication and plaque formation of mouse hepatitis virus
@nl
P2093
P356
P1476
Replication and plaque formati ...... in mouse cell line DBT culture
@en
P2093
P304
P356
10.1007/BF01240618
P577
1974-01-01T00:00:00Z