RNase L-independent specific 28S rRNA cleavage in murine coronavirus-infected cells
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ICP0 prevents RNase L-independent rRNA cleavage in herpes simplex virus type 1-infected cellsViral encounters with 2',5'-oligoadenylate synthetase and RNase L during the interferon antiviral responseTowards standardization of RNA quality assessment using user-independent classifiers of microcapillary electrophoresis tracesRegulation of Stress Responses and Translational Control by CoronavirusRNase L plays a role in the antiviral response to West Nile virus.Coronavirus Infection Modulates the Unfolded Protein Response and Mediates Sustained Translational RepressionViral and Cellular mRNA Translation in Coronavirus-Infected Cells.Equine torovirus (BEV) induces caspase-mediated apoptosis in infected cellsMurine coronavirus replication induces cell cycle arrest in G0/G1 phase.Murine coronavirus replication-induced p38 mitogen-activated protein kinase activation promotes interleukin-6 production and virus replication in cultured cells.RNA disruption is associated with response to multiple classes of chemotherapy drugs in tumor cell lines.Ribonuclease L and metal-ion-independent endoribonuclease cleavage sites in host and viral RNAs.Targets and intracellular signaling mechanisms for deoxynivalenol-induced ribosomal RNA cleavage.Novel small-molecule inhibitors of transmissible gastroenteritis virus.Apoptotic signals induce specific degradation of ribosomal RNA in yeast.A two-pronged strategy to suppress host protein synthesis by SARS coronavirus Nsp1 protein.Tinkering with translation: protein synthesis in virus-infected cells.Inhibition of protein kinase R activation and upregulation of GADD34 expression play a synergistic role in facilitating coronavirus replication by maintaining de novo protein synthesis in virus-infected cells.Mouse hepatitis coronavirus A59 nucleocapsid protein is a type I interferon antagonist.Characterization of the antiviral effects of interferon-alpha against a SARS-like coronoavirus infection in vitro.Chronic fatigue and immune deficiency syndrome (CFIDS), cellular metabolism, and ionizing radiation: a review of contemporary scientific literature and suggested directions for future research.
P2860
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P2860
RNase L-independent specific 28S rRNA cleavage in murine coronavirus-infected cells
description
2000 nî lūn-bûn
@nan
2000 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
RNase L-independent specific 28S rRNA cleavage in murine coronavirus-infected cells
@ast
RNase L-independent specific 28S rRNA cleavage in murine coronavirus-infected cells
@en
type
label
RNase L-independent specific 28S rRNA cleavage in murine coronavirus-infected cells
@ast
RNase L-independent specific 28S rRNA cleavage in murine coronavirus-infected cells
@en
prefLabel
RNase L-independent specific 28S rRNA cleavage in murine coronavirus-infected cells
@ast
RNase L-independent specific 28S rRNA cleavage in murine coronavirus-infected cells
@en
P2093
P2860
P1433
P1476
RNase L-independent specific 28S rRNA cleavage in murine coronavirus-infected cells
@en
P2093
P2860
P304
P356
10.1128/JVI.74.19.8793-8802.2000
P577
2000-10-01T00:00:00Z