Modulation of the phosphoinositide 3-kinase pathway alters innate resistance to polymicrobial sepsis. - Wikidata - awgldk - TriplyDB

Modulation of the phosphoinositide 3-kinase pathway alters innate resistance to polymicrobial sepsis.

Modulation of the phosphoinositide 3-kinase pathway alters innate resistance to polymicrobial sepsis.

http://www.wikidata.org/entity/Q44701506

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Electroacupuncture Ameliorates Acute Renal Injury in Lipopolysaccharide-Stimulated Rabbits via Induction of HO-1 through the PI3K/Akt/Nrf2 Pathways Phosphoinositide 3-kinase gamma required for lipopolysaccharide-induced transepithelial neutrophil trafficking in the lung.Beta-(1-->3)-D-glucan modulates DNA binding of nuclear factors kappaB, AT and IL-6 leading to an anti-inflammatory shift of the IL-1beta/IL-1 receptor antagonist ratio Interleukin-10 suppression of myeloid cell activation--a continuing puzzle.Genetic analysis of the role of the PI3K-Akt pathway in lipopolysaccharide-induced cytokine and tissue factor gene expression in monocytes/macrophages.Insulin activation of the phosphatidylinositol 3-kinase/protein kinase B (Akt) pathway reduces lipopolysaccharide-induced inflammation in mice TLR2 ligands attenuate cardiac dysfunction in polymicrobial sepsis via a phosphoinositide 3-kinase-dependent mechanism.Genome-wide methylation analysis reveals differentially methylated loci that are associated with an age-dependent increase in bovine fibroblast response to LPS.Atorvastatin improves survival in septic rats: effect on tissue inflammatory pathway and on insulin signaling.TLR2 ligands induce cardioprotection against ischaemia/reperfusion injury through a PI3K/Akt-dependent mechanism A2B adenosine receptors protect against sepsis-induced mortality by dampening excessive inflammation.Activation of myocardial phosphoinositide-3-kinase p110α ameliorates cardiac dysfunction and improves survival in polymicrobial sepsis.β-arrestin 2-mediated immune suppression induced by chronic stress.Differential regulation of interleukin-8 and human beta-defensin 2 in Pseudomonas aeruginosa-infected intestinal epithelial cells Abrogation of antibody-induced arthritis in mice by a self-activating viridin prodrug and association with impaired neutrophil and endothelial cell function HSPA12B inhibits lipopolysaccharide-induced inflammatory response in human umbilical vein endothelial cells.Glucan phosphate attenuates myocardial HMGB1 translocation in severe sepsis through inhibiting NF-κB activation.Astragaloside IV inhibits NF- κ B activation and inflammatory gene expression in LPS-treated mice.PTEN negatively regulates engulfment of apoptotic cells by modulating activation of Rac GTPase.Inhibition of Toll-like receptor 9 attenuates sepsis-induced mortality through suppressing excessive inflammatory response.Alpha-lipoic acid attenuates LPS-induced inflammatory responses by activating the phosphoinositide 3-kinase/Akt signaling pathway Developing animal models for polymicrobial diseases.Attenuation of Cardiac Dysfunction in Polymicrobial Sepsis by MicroRNA-146a Is Mediated via Targeting of IRAK1 and TRAF6 Expression.Regulation of ENaC-mediated alveolar fluid clearance by insulin via PI3K/Akt pathway in LPS-induced acute lung injury The toll-like receptor 9 agonist, CpG-oligodeoxynucleotide 1826, ameliorates cardiac dysfunction after trauma-hemorrhage.Allospecific CD4(+) effector memory T cells do not induce graft-versus-host disease in mice.Scavenger receptor class a plays a central role in mediating mortality and the development of the pro-inflammatory phenotype in polymicrobial sepsis.Rhesus macaque θ-defensin RTD-1 inhibits proinflammatory cytokine secretion and gene expression by inhibiting the activation of NF-κB and MAPK pathways PI3K is critical for the nuclear translocation of IRF-7 and type I IFN production by human plasmacytoid predendritic cells in response to TLR activation.Mucosal adjuvant activity of flagellin in aged mice.Endothelial p110γPI3K Mediates Endothelial Regeneration and Vascular Repair After Inflammatory Vascular Injury Dendritic cells in human aging Toll-like receptor 3 plays a central role in cardiac dysfunction during polymicrobial sepsis.TREM-2 promotes host resistance against Pseudomonas aeruginosa infection by suppressing corneal inflammation via a PI3K/Akt signaling pathway.The Toll-like receptor 9 ligand, CpG oligodeoxynucleotide, attenuates cardiac dysfunction in polymicrobial sepsis, involving activation of both phosphoinositide 3 kinase/Akt and extracellular-signal-related kinase signaling Restraint stress induces lymphocyte reduction through p53 and PI3K/NF-kappaB pathways Chronic restraint stress promotes immune suppression through toll-like receptor 4-mediated phosphoinositide 3-kinase signaling.Modeling the Pro-inflammatory Tumor Microenvironment in Acute Lymphoblastic Leukemia Predicts a Breakdown of Hematopoietic-Mesenchymal Communication Networks Anti-inflammatory effect of procyanidins from wild grape (Vitis amurensis) seeds in LPS-induced RAW 264.7 cells.Leukocyte Dectin-1 expression is differentially regulated in fungal versus polymicrobial sepsis.

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Modulation of the phosphoinositide 3-kinase pathway alters innate resistance to polymicrobial sepsis.

http://www.wikidata.org/entity/Q44701506

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年學術文章

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name

Modulation of the phosphoinosi ...... tance to polymicrobial sepsis.

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Modulation of the phosphoinosi ...... tance to polymicrobial sepsis.

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type

label

Modulation of the phosphoinosi ...... tance to polymicrobial sepsis.

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Modulation of the phosphoinosi ...... tance to polymicrobial sepsis.

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prefLabel

Modulation of the phosphoinosi ...... tance to polymicrobial sepsis.

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Modulation of the phosphoinosi ...... tance to polymicrobial sepsis.

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JIMMUNOL.172.1.449

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Journal of Immunology

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Modulation of the phosphoinosi ...... tance to polymicrobial sepsis.

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Chuanfu Li

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David L Williams

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Johanna Preiszner

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John B Schweitzer

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John H Kalbfleisch

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Kevin Breuel

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Lynne Brooks

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Tammy Ozment-Skelton

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Tuanzhu Ha

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449-456

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