Lack of CD200 enhances pathological T cell responses during influenza infection. - Wikidata - awgldk - TriplyDB

Lack of CD200 enhances pathological T cell responses during influenza infection.

Lack of CD200 enhances pathological T cell responses during influenza infection.

http://www.wikidata.org/entity/Q44745579

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CD200 receptor controls sex-specific TLR7 responses to viral infection How Neutrophils Shape Adaptive Immune Responses Chronic infection drives expression of the inhibitory receptor CD200R, and its ligand CD200, by mouse and human CD4 T cells Pneumovirus-Induced Lung Disease in Mice Is Independent of Neutrophil-Driven Inflammation Host response to influenza virus: protection versus immunopathology.T cell responses during influenza infection: getting and keeping control.Recent insights into pulmonary repair following virus-induced inflammation of the respiratory tract.The CD200-CD200 receptor inhibitory axis controls arteriogenesis and local T lymphocyte influx.Constrained release of lamina-associated enhancers and genes from the nuclear envelope during T-cell activation facilitates their association in chromosome compartments.The rhesus rhadinovirus CD200 homologue affects immune responses and viral loads during in vivo infection Altered gene expression in blood and sputum in COPD frequent exacerbators in the ECLIPSE cohort.Genome-wide association study of antibody response to Newcastle disease virus in chicken.IFN-γ-stimulated neutrophils suppress lymphocyte proliferation through expression of PD-L1.Lowering the threshold of lung innate immune cell activation alters susceptibility to secondary bacterial superinfection CD200 receptor restriction of myeloid cell responses antagonizes antiviral immunity and facilitates cytomegalovirus persistence within mucosal tissue.The Inhibitory Receptor NKG2A Sustains Virus-Specific CD8⁺ T Cells in Response to a Lethal Poxvirus Infection Understanding the neurobiology of CD200 and the CD200 receptor: a therapeutic target for controlling inflammation in human brains?Respiratory viral infection in neonatal piglets causes marked microglia activation in the hippocampus and deficits in spatial learning.Immune inhibitory receptors: essential regulators of phagocyte function.Cellular immune reactions in the lung.Macrophage-mediated inflammation and disease: a focus on the lung.Use of an anti-CD200 antibody for prolonging the survival of allografts: a patent evaluation of WO2012106634A1.Immune dysfunction and bacterial coinfections following influenza.BamHI-A rightward frame 1, an Epstein-Barr virus-encoded oncogene and immune modulator.Human CD8(+) T Cells Damage Non-infected Epithelial Cells During Influenza Virus Infection In Vitro.Unravelling the networks dictating host resistance versus tolerance during pulmonary infections.Soluble CD200 Correlates With Interleukin-6 Levels in Sera of COPD Patients: Potential Implication of the CD200/CD200R Axis in the Disease Course.Differentiating Staphylococcus aureus from Escherichia coli mastitis: S. aureus triggers unbalanced immune-dampening and host cell invasion immediately after udder infection.A Critical Role for CD200R Signaling in Limiting the Growth and Metastasis of CD200+ Melanoma.Critical role of natural killer cells in lung immunopathology during influenza infection in mice.CD200 in growing rat lungs: developmental expression and control by dexamethasone.Association of peripheral blood dendritic cells with recurrent pregnancy loss: a case-controlled study.Centrin-Deleted Leishmania donovani Parasites Help CD4+ T Cells to Acquire Th1 Phenotype and Multi-Functionality Through Downregulation of CD200-CD200R Immune Inhibitory Axis.Inhibitory receptor expression on neonatal immune cells

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P2860

Lack of CD200 enhances pathological T cell responses during influenza infection.

http://www.wikidata.org/entity/Q44745579

description

2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

@zh-my

2009年学术文章

@zh-sg

2009年學術文章

@yue

2009年學術文章

@zh-hant

name

Lack of CD200 enhances pathological T cell responses during influenza infection.

@en

Lack of CD200 enhances pathological T cell responses during influenza infection.

@nl

type

label

Lack of CD200 enhances pathological T cell responses during influenza infection.

@en

Lack of CD200 enhances pathological T cell responses during influenza infection.

@nl

prefLabel

Lack of CD200 enhances pathological T cell responses during influenza infection.

@en

Lack of CD200 enhances pathological T cell responses during influenza infection.

@nl

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JIMMUNOL.0900252

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Journal of Immunology

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Lack of CD200 enhances pathological T cell responses during influenza infection.

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Anton M van Loon

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Ellen H Stolte

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Eva S K Rijkers

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Frank E Coenjaerts

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Kiki Tesselaar

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Louis Boon

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Martin van der Valk

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Robert M Hoek

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Tomasz P Rygiel

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10.4049/JIMMUNOL.0900252

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Guus Rimmelzwaan

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