Immune dysfunction and bacterial coinfections following influenza.
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Harnessing Mechanistic Knowledge on Beneficial Versus Deleterious IFN-I Effects to Design Innovative Immunotherapies Targeting Cytokine Activity to Specific Cell TypesThe Association between Invasive Group A Streptococcal Diseases and Viral Respiratory Tract InfectionsMechanisms of Bacterial Colonization of the Respiratory TractRole of non-conventional T lymphocytes in respiratory infections: the case of the pneumococcusThe potential impact of coinfection on antimicrobial chemotherapy and drug resistanceThe host immune dynamics of pneumococcal colonization: implications for novel vaccine developmentNovel pandemic influenza A (H1N1) and community-associated methicillin-resistant Staphylococcus aureus pneumonia.Vaccination against the M protein of Streptococcus pyogenes prevents death after influenza virus: S. pyogenes super-infection.Influenza promotes pneumococcal growth during coinfection by providing host sialylated substrates as a nutrient source.Applying Convergent Immunity to Innovative Vaccines Targeting Staphylococcus aureus.Regulation of IFN-γ by IL-13 dictates susceptibility to secondary postinfluenza MRSA pneumonia.Enhanced Mucosal Antibody Production and Protection against Respiratory Infections Following an Orally Administered Bacterial Extract.Lethal coinfection of influenza virus and Streptococcus pneumoniae lowers antibody response to influenza virus in lung and reduces numbers of germinal center B cells, T follicular helper cells, and plasma cells in mediastinal lymph Node.Dynamic Virus-Bacterium Interactions in a Porcine Precision-Cut Lung Slice Coinfection Model: Swine Influenza Virus Paves the Way for Streptococcus suis Infection in a Two-Step Process.Clinical differences between respiratory viral and bacterial mono- and dual pathogen detected among Singapore military servicemen with febrile respiratory illness.Plasma gelsolin improves lung host defense against pneumonia by enhancing macrophage NOS3 function.The administration of intranasal live attenuated influenza vaccine induces changes in the nasal microbiota and nasal epithelium gene expression profilesTRAIL+ monocytes and monocyte-related cells cause lung damage and thereby increase susceptibility to influenza-Streptococcus pneumoniae coinfection.Extremes of Interferon-Stimulated Gene Expression Associate with Worse Outcomes in the Acute Respiratory Distress Syndrome.LPAIV H9N2 Drives the Differential Expression of Goose Interferons and Proinflammatory Cytokines in Both In Vitro and In Vivo Studies.Nox2-derived oxidative stress results in inefficacy of antibiotics against post-influenza S. aureus pneumonia.A Critical, Nonlinear Threshold Dictates Bacterial Invasion and Initial Kinetics During Influenza.Influenza infection suppresses NADPH oxidase-dependent phagocytic bacterial clearance and enhances susceptibility to secondary methicillin-resistant Staphylococcus aureus infection.Quantifying the therapeutic requirements and potential for combination therapy to prevent bacterial coinfection during influenza.Adaptation in the innate immune system and heterologous innate immunity.Non-typeable Haemophilus influenzae protects human airway epithelial cells from a subsequent respiratory syncytial virus challenge.Super-infection with Staphylococcus aureus inhibits influenza virus-induced type I IFN signalling through impaired STAT1-STAT2 dimerization.Use of Antibiotics and Antimicrobial Resistance in Veterinary Medicine as Exemplified by the Swine Pathogen Streptococcus suis.Protection against Streptococcus pneumoniae Invasive Pathogenesis by a Protein-Based Vaccine Is Achieved by Suppression of Nasopharyngeal Bacterial Density during Influenza A Virus Coinfection.Mechanisms of Severe Mortality-Associated Bacterial Co-infections Following Influenza Virus Infection.Fatal Fulminant Pneumonia Caused by Methicillin-Sensitive Staphylococcus aureus Negative for Major High-Virulence Factors Following Influenza B Virus Infection.[Influenza and secondary bacterial infections: threats and treatments].Reversal of TREM-1 ectodomain shedding and improved bacterial clearance by intranasal metalloproteinase inhibitors.Co-expressional conservation in virulence and stress related genes of three Gammaproteobacterial species: Escherichia coli, Salmonella enterica and Pseudomonas aeruginosa.Influenza A virus-induced release of interleukin-10 inhibits the anti-microbial activities of invariant natural killer T cells during invasive pneumococcal superinfection.Positive Contribution of Adjuvanted Influenza Vaccines to the Resolution of Bacterial Superinfections.A Perfect Storm: Increased Colonization and Failure of Vaccination Leads to Severe Secondary Bacterial Infection in Influenza Virus-Infected Obese Mice.Proteomic Analysis of IPEC-J2 Cells in Response to Coinfection by Porcine Transmissible Gastroenteritis Virus and Enterotoxigenic Escherichia coli K88.Pathogen-associated molecular patterns alter molecular clock gene expression in mouse splenocytes.Port d’Entrée for Respiratory Infections – Does the Influenza A Virus Pave the Way for Bacteria?
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Immune dysfunction and bacterial coinfections following influenza.
description
article científic
@ca
article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
@pt
artigo científico
@pt-br
artikel ilmiah
@id
artikull shkencor
@sq
artículo científico
@es
name
Immune dysfunction and bacterial coinfections following influenza.
@en
type
label
Immune dysfunction and bacterial coinfections following influenza.
@en
prefLabel
Immune dysfunction and bacterial coinfections following influenza.
@en
P2860
P356
P1476
Immune dysfunction and bacterial coinfections following influenza.
@en
P2093
P2860
P304
P356
10.4049/JIMMUNOL.1301152
P407
P577
2013-09-01T00:00:00Z