Interleukin-1 receptor antagonist attenuates airway hyperresponsiveness following exposure to ozone.
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Inflammasome, IL-1 and inflammation in ozone-induced lung injuryAssociation between polymorphism of interleukin-1 beta and interleukin-1 receptor antagonist gene and asthma risk: a meta-analysisVgamma1+ T cells and tumor necrosis factor-alpha in ozone-induced airway hyperresponsivenessOzone exposure of macrophages induces an alveolar epithelial chemokine response through IL-1alphaAvian-type receptor-binding ability can increase influenza virus pathogenicity in macaques.Characterization in vitro and in vivo of pandemic (H1N1) 2009 influenza viruses isolated from patients.Interleukin-1 receptor antagonist prevents murine bronchopulmonary dysplasia induced by perinatal inflammation and hyperoxia.Maternal exposure to particulate matter increases postnatal ozone-induced airway hyperreactivity in juvenile mice.TLR4 is necessary for hyaluronan-mediated airway hyperresponsiveness after ozone inhalation.Hyaluronan signaling during ozone-induced lung injury requires TLR4, MyD88, and TIRAP.Atopic asthmatic subjects but not atopic subjects without asthma have enhanced inflammatory response to ozone.Association of cytokine gene polymorphism with susceptibility and clinical types of leprosy.The role of the extracellular matrix protein mindin in airway response to environmental airways injuryOzone exposure initiates a sequential signaling cascade in airways involving interleukin-1beta release, nerve growth factor secretion, and substance P upregulationSignal transduction pathways of tumor necrosis factor--mediated lung injury induced by ozone in mice.Pneumocystis elicits a STAT6-dependent, strain-specific innate immune response and airway hyperresponsiveness.Effects of Exposure to Ozone on the Ocular Surface in an Experimental Model of Allergic Conjunctivitis.Resistin deficiency in mice has no effect on pulmonary responses induced by acute ozone exposure.Type I interleukin-1 receptor is required for pulmonary responses to subacute ozone exposure in mice.Ozone-induced IL-17A and neutrophilic airway inflammation is orchestrated by the caspase-1-IL-1 cascade.Ozone exposure in a mouse model induces airway hyperreactivity that requires the presence of natural killer T cells and IL-17Hyaluronan activation of the Nlrp3 inflammasome contributes to the development of airway hyperresponsiveness.Ozone enhances pulmonary innate immune response to a Toll-like receptor-2 agonist.Interleukin-1R antagonist gene and pre-natal smoke exposure are associated with childhood asthma.Targeting the networks that underpin contiguous immunity in asthma and chronic obstructive pulmonary disease.Vagotomy reverses established allergen-induced airway hyperreactivity to methacholine in the mouseOzone and pulmonary innate immunityNo effect of metformin on the innate airway hyperresponsiveness and increased responses to ozone observed in obese miceIL-1 receptors mediate persistent, but not acute, airway hyperreactivity to ozone in guinea pigs.Endogenous osteopontin promotes ozone-induced neutrophil recruitment to the lungs and airway hyperresponsiveness to methacholinePrimary murine airway smooth muscle cells exposed to poly(I,C) or tunicamycin synthesize a leukocyte-adhesive hyaluronan matrixAmbient ozone and pulmonary innate immunity.Differential expression of pro-inflammatory and oxidative stress mediators induced by nitrogen dioxide and ozone in primary human bronchial epithelial cellsPlasminogen activator inhibitor-1 does not contribute to the pulmonary pathology induced by acute exposure to ozone.Immunogenetics of drug-induced skin blistering disorders. Part II: synthesis.Effect of obesity on pulmonary inflammation induced by acute ozone exposure: role of interleukin-6.Increased pulmonary responses to acute ozone exposure in obese db/db mice.The effects of ozone on bacterial growth and thiol-disulphide homeostasis in vascular graft infection caused by MRSA in rats.CXCR2 is essential for maximal neutrophil recruitment and methacholine responsiveness after ozone exposure.A critical role for the IL-1 receptor in lung injury induced in neonatal rats by 60% O2.
P2860
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P2860
Interleukin-1 receptor antagonist attenuates airway hyperresponsiveness following exposure to ozone.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
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@yue
2004年學術文章
@zh-hant
name
Interleukin-1 receptor antagon ...... s following exposure to ozone.
@en
Interleukin-1 receptor antagon ...... s following exposure to ozone.
@nl
type
label
Interleukin-1 receptor antagon ...... s following exposure to ozone.
@en
Interleukin-1 receptor antagon ...... s following exposure to ozone.
@nl
prefLabel
Interleukin-1 receptor antagon ...... s following exposure to ozone.
@en
Interleukin-1 receptor antagon ...... s following exposure to ozone.
@nl
P2093
P2860
P50
P356
P1476
Interleukin-1 receptor antagon ...... s following exposure to ozone.
@en
P2093
Annette Balhorn
Anthony Joetham
Christian Taube
Christina Swasey
Corrie B Allen
Jung-Won Park
Katsuyuki Takeda
Nobuaki Miyahara
Soo-Hyun Kim
Taku Kodama
P2860
P304
P356
10.1165/RCMB.2003-0373OC
P577
2004-01-30T00:00:00Z