Changes in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain.
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Phyla- and Subtype-Selectivity of CgNa, a Na Channel Toxin from the Venom of the Giant Caribbean Sea Anemone Condylactis GiganteaRole of acid-sensing ion channel 3 in sub-acute-phase inflammationTemperature dependence of erythromelalgia mutation L858F in sodium channel Nav1.7Synthetic ciguatoxins selectively activate Nav1.8-derived chimeric sodium channels expressed in HEK293 cellsConotoxins That Could Provide Analgesia through Voltage Gated Sodium Channel InhibitionPrimary erythromelalgia: a reviewIon channels as drug targets in central nervous system disordersRoles of Voltage-Gated Tetrodotoxin-Sensitive Sodium Channels NaV1.3 and NaV1.7 in Diabetes and Painful Diabetic NeuropathyChanging channels in pain and epilepsy: Exploiting ion channel gene therapy for disorders of neuronal hyperexcitabilityArthritis and pain. Neurogenic origin of joint painNeurobiology: a channel sets the gain on painCCL2 has similar excitatory effects to TNF-α in a subgroup of inflamed C-fiber axonsInhibition of Inactive States of Tetrodotoxin-Sensitive Sodium Channels Reduces Spontaneous Firing of C-Fiber Nociceptors and Produces Analgesia in Formalin and Complete Freund's Adjuvant Models of PainContinuous delta-opioid receptor activation reduces neuronal voltage-gated sodium channel (NaV1.7) levels through activation of protein kinase C in painful diabetic neuropathySelective spider toxins reveal a role for the Nav1.1 channel in mechanical painEstradiol upregulates voltage-gated sodium channel 1.7 in trigeminal ganglion contributing to hyperalgesia of inflamed TMJ.Voltage-gated Na+ channels in neuropathic pain.Temporal coding by cochlear nucleus bushy cells in DBA/2J mice with early onset hearing loss.A gain-of-function voltage-gated sodium channel 1.8 mutation drives intense hyperexcitability of A- and C-fiber neurons.Nav1.9 channel contributes to mechanical and heat pain hypersensitivity induced by subacute and chronic inflammation.Molecular profiling of murine sensory neurons in the nodose and dorsal root ganglia labeled from the peritoneal cavity.Upregulation of nav1.7 through high salt loading: (mol pain 2013;9:39)Targeting ASIC3 for Relieving Mice Fibromyalgia Pain: Roles of Electroacupuncture, Opioid, and Adenosine.Contribution of the tetrodotoxin-resistant voltage-gated sodium channel NaV1.9 to sensory transmission and nociceptive behavior.Tetrodotoxin (TTX) as a therapeutic agent for pain.Single-cell analysis of sodium channel expression in dorsal root ganglion neuronsMutations in sodium-channel gene SCN9A cause a spectrum of human genetic pain disorders.Are voltage-gated sodium channels on the dorsal root ganglion involved in the development of neuropathic pain?The roles of sodium channels in nociception: Implications for mechanisms of painFunctional upregulation of nav1.8 sodium channels on the membrane of dorsal root Ganglia neurons contributes to the development of cancer-induced bone painHypotonicity modulates tetrodotoxin-sensitive sodium current in trigeminal ganglion neuronsNav1.7 is the predominant sodium channel in rodent olfactory sensory neurons.Sodium channel expression in the ventral posterolateral nucleus of the thalamus after peripheral nerve injuryUp-regulation of tetrodotoxin-sensitive sodium currents by prostaglandin E₂ in type-4 rat dorsal root ganglion cells.Hypersensitivity and hyperinnervation of the rat hind paw following carrageenan-induced inflammationNerve injury induces robust allodynia and ectopic discharges in Nav1.3 null mutant mice.Dexmedetomidine inhibits Tetrodotoxin-resistant Nav1.8 sodium channel activity through Gi/o-dependent pathway in rat dorsal root ganglion neurons.Acid-sensing ion channel 3 mediates peripheral anti-hyperalgesia effects of acupuncture in mice inflammatory pain.Regulation of Nav1.7: A Conserved SCN9A Natural Antisense Transcript Expressed in Dorsal Root Ganglia.Sensitization of dural afferents underlies migraine-related behavior following meningeal application of interleukin-6 (IL-6).
P2860
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P2860
Changes in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
@zh-hans
2004年学术文章
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2004年学术文章
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2004年學術文章
@zh-hant
name
Changes in the expression of t ...... neurons in inflammatory pain.
@en
Changes in the expression of t ...... neurons in inflammatory pain.
@nl
type
label
Changes in the expression of t ...... neurons in inflammatory pain.
@en
Changes in the expression of t ...... neurons in inflammatory pain.
@nl
prefLabel
Changes in the expression of t ...... neurons in inflammatory pain.
@en
Changes in the expression of t ...... neurons in inflammatory pain.
@nl
P2093
P1433
P1476
Changes in the expression of t ...... neurons in inflammatory pain.
@en
P2093
Joel A Black
Masaki Tanaka
Shujun Liu
Stephen G Waxman
Theodore R Cummins
P304
P356
10.1016/J.PAIN.2003.12.035
P407
P577
2004-04-01T00:00:00Z