Single-cell analysis of sodium channel expression in dorsal root ganglion neurons - Wikidata - awgldk - TriplyDB

Single-cell analysis of sodium channel expression in dorsal root ganglion neurons

Single-cell analysis of sodium channel expression in dorsal root ganglion neurons

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Electrophysiological properties of embryonic stem cell-derived neurons Subtype-Selective Small Molecule Inhibitors Reveal a Fundamental Role for Nav1.7 in Nociceptor Electrogenesis, Axonal Conduction and Presynaptic Release.Voltage-gated sodium channel expression in mouse DRG after SNI leads to re-evaluation of projections of injured fibers.Enhanced excitability of primary sensory neurons and altered gene expression of neuronal ion channels in dorsal root ganglion in paclitaxel-induced peripheral neuropathy Voltage-gated Na+ currents in human dorsal root ganglion neurons.Modulation of neuronal sodium channels by the sea anemone peptide BDS-I.Dysregulation of voltage-gated sodium channels by ubiquitin ligase NEDD4-2 in neuropathic pain.Sea-anemone toxin ATX-II elicits A-fiber-dependent pain and enhances resurgent and persistent sodium currents in large sensory neurons.Inhibition of tetrodotoxin-resistant sodium current in dorsal root ganglia neurons mediated by D1/D5 dopamine receptors Silk-tropoelastin protein films for nerve guidance.Nav1.7 protein and mRNA expression in the dorsal root ganglia of rats with chronic neuropathic pain.The venom of the spider Selenocosmia jiafu contains various neurotoxins acting on voltage-gated ion channels in rat dorsal root ganglion neurons.Increased expression of sodium channel subunit Nav1.1 in the injured dorsal root ganglion after peripheral nerve injury In vivo serial imaging of regenerating corneal nerves after surgical transection in transgenic thy1-YFP mice.Regulatory Role of Voltage-Gated Na Channel β Subunits in Sensory Neurons Regulation of Nav1.6 and Nav1.8 peripheral nerve Na+ channels by auxiliary β-subunits.Novel sodium channel antagonists in the treatment of neuropathic pain.Differential expression of sodium channel β subunits in dorsal root ganglion sensory neurons.Navβ4 regulates fast resurgent sodium currents and excitability in sensory neurons SCN10A Mutation in a Patient with Erythromelalgia Enhances C-Fiber Activity Dependent Slowing.Post-translational modifications of voltage-gated sodium channels in chronic pain syndromes Knockout of Slo2.2 enhances itch, abolishes KNa current, and increases action potential firing frequency in DRG neurons.Tetrodotoxin-resistant voltage-dependent sodium channels in identified muscle afferent neurons Lack of transient receptor potential vanilloid 1 channel modulates the development of neurogenic bladder dysfunction induced by cross-sensitization in afferent pathways Pharmacological fractionation of tetrodotoxin-sensitive sodium currents in rat dorsal root ganglion neurons by μ-conotoxins Sodium channel diversity in the vestibular ganglion: NaV1.5, NaV1.8, and tetrodotoxin-sensitive currents.β1- and β3- voltage-gated sodium channel subunits modulate cell surface expression and glycosylation of Nav1.7 in HEK293 cells.MicroRNA-30b regulates expression of the sodium channel Nav1.7 in nerve injury-induced neuropathic pain in the rat Reactive species modify NaV1.8 channels and affect action potentials in murine dorsal root ganglion neurons.Neuronal electrophysiological function and control of neurite outgrowth on electrospun polymer nanofibers are cell type dependent Recent developments regarding voltage-gated sodium channel blockers for the treatment of inherited and acquired neuropathic pain syndromes.Modality-specific axonal regeneration: toward selective regenerative neural interfaces.FHF2 isoforms differentially regulate Nav1.6-mediated resurgent sodium currents in dorsal root ganglion neurons.Sodium Channel Nav1.8 Underlies TTX-Resistant Axonal Action Potential Conduction in Somatosensory C-Fibers of Distal Cutaneous Nerves.Functional up-regulation of Nav1.8 sodium channel in Aβ afferent fibers subjected to chronic peripheral inflammation.Switching off pain at the source: is this the end for opioid pain relief?Cooperativity between remote sites of ectopic spiking allows afterdischarge to be initiated and maintained at different locations CC chemokine ligand 2 upregulates the current density and expression of TRPV1 channels and Nav1.8 sodium channels in dorsal root ganglion neurons.Correlation of the electrophysiological profiles and sodium channel transcripts of individual rat dorsal root ganglia neurons.Histamine modulation of acute nociception involves regulation of Nav 1.8 in primary afferent neurons in mice.

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Single-cell analysis of sodium channel expression in dorsal root ganglion neurons

http://www.wikidata.org/entity/Q34422391

description

2010 nî lūn-bûn

@nan

2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

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2010年論文

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2010年论文

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name

Single-cell analysis of sodium channel expression in dorsal root ganglion neurons

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Single-cell analysis of sodium channel expression in dorsal root ganglion neurons

@en

Single-cell analysis of sodium channel expression in dorsal root ganglion neurons

@nl

type

label

Single-cell analysis of sodium channel expression in dorsal root ganglion neurons

@ast

Single-cell analysis of sodium channel expression in dorsal root ganglion neurons

@en

Single-cell analysis of sodium channel expression in dorsal root ganglion neurons

@nl

prefLabel

Single-cell analysis of sodium channel expression in dorsal root ganglion neurons

@ast

Single-cell analysis of sodium channel expression in dorsal root ganglion neurons

@en

Single-cell analysis of sodium channel expression in dorsal root ganglion neurons

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J.MCN.2010.08.017

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Molecular and Cellular Neuroscience

P1476

Single-cell analysis of sodium channel expression in dorsal root ganglion neurons

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P2093

Cojen Ho

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Michael E O'Leary

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P2860

NaN/Nav1.9: a sodium channel with unique properties

A novel persistent tetrodotoxin-resistant sodium current in SNS-null and wild-type small primary sensory neurons

Identification of PN1, a predominant voltage-dependent sodium channel expressed principally in peripheral neurons

A tetrodotoxin-resistant voltage-gated sodium channel expressed by sensory neurons

Nectin-like proteins mediate axon Schwann cell interactions along the internode and are essential for myelination

Gating properties of Na(v)1.7 and Na(v)1.8 peripheral nerve sodium channels.

Nectin-like molecule-1/TSLL1/SynCAM3: a neural tissue-specific immunoglobulin-like cell-cell adhesion molecule localizing at non-junctional contact sites of presynaptic nerve terminals, axons and glia cell processes.

Distribution of the tetrodotoxin-resistant sodium channel PN3 in rat sensory neurons in normal and neuropathic conditions.

A central role for Necl4 (SynCAM4) in Schwann cell-axon interaction and myelination.

Contribution of the tetrodotoxin-resistant voltage-gated sodium channel NaV1.9 to sensory transmission and nociceptive behavior.

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159-166

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10.1016/J.MCN.2010.08.017

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1

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