Elevated MnSOD is not required for exercise-induced cardioprotection against myocardial stunning. - Wikidata - awgldk - TriplyDB

Elevated MnSOD is not required for exercise-induced cardioprotection against myocardial stunning.

Elevated MnSOD is not required for exercise-induced cardioprotection against myocardial stunning.

http://www.wikidata.org/entity/Q44805460

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Exercise Modulates Oxidative Stress and Inflammation in Aging and Cardiovascular Diseases Effects of Methane-Rich Saline on the Capability of One-Time Exhaustive Exercise in Male SD Rats Subcellular Energetics and Metabolism: Potential Therapeutic Applications.Effects of exercise and L-arginine on ventricular remodeling and oxidative stress.Mitochondrial KATP channel inhibition blunts arrhythmia protection in ischemic exercised hearts.Exercise training combined with angiotensin II receptor blockade reduces oxidative stress after myocardial infarction in rats.Acute exercise increases resistance to oxidative stress in young but not older adults.Delta opioid receptors: the link between exercise and cardioprotection.Molecular mechanisms in exercise-induced cardioprotection Cardiac remodeling and physical training post myocardial infarction Mechanisms Involved in Exercise-Induced Cardioprotection: A Systematic Review.Exercise-induced protection against reperfusion arrhythmia involves stabilization of mitochondrial energetics Evaluation of arrhythmia scoring systems and exercise-induced cardioprotection Ischemia reperfusion injury, KATP channels, and exercise-induced cardioprotection against apoptosis Exercise-induced protection against myocardial apoptosis and necrosis: MnSOD, calcium-handling proteins, and calpain.Exercise and cardiac preconditioning against ischemia reperfusion injury.Exercise-induced improvements in myocardial antioxidant capacity: the antioxidant players and cardioprotection.Mitochondria in the middle: exercise preconditioning protection of striated muscle.Involvement of the δ-opioid receptor in exercise-induced cardioprotection.MnSOD in mouse heart: acute responses to ischemic preconditioning and ischemia-reperfusion injury.Increased temperature, not cardiac load, activates heat shock transcription factor 1 and heat shock protein 72 expression in the heart.Short-term exercise preserves myocardial glutathione and decreases arrhythmias after thiol oxidation and ischemia in isolated rat hearts.Exercise training improves myocardial tolerance to ischemia in male but not in female rats.Interleukin-6 mediates exercise preconditioning against myocardial ischemia reperfusion injury.

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P2860

Elevated MnSOD is not required for exercise-induced cardioprotection against myocardial stunning.

http://www.wikidata.org/entity/Q44805460

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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name

Elevated MnSOD is not required ...... n against myocardial stunning.

@en

Elevated MnSOD is not required ...... n against myocardial stunning.

@nl

type

label

Elevated MnSOD is not required ...... n against myocardial stunning.

@en

Elevated MnSOD is not required ...... n against myocardial stunning.

@nl

prefLabel

Elevated MnSOD is not required ...... n against myocardial stunning.

@en

Elevated MnSOD is not required ...... n against myocardial stunning.

@nl

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AJPHEART.01208.2003

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American Journal of Physiology - Heart and Circulatory Physiology

P1476

Elevated MnSOD is not required ...... n against myocardial stunning.

@en

P2093

Jay L Mehta

http://www.w3.org/2001/XMLSchema#string

Jeffrey Hughes

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Joel P French

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John C Quindry

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Karyn L Hamilton

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Scott K Powers

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Shannon L Lennon

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P2860

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

Exercise provides direct biphasic cardioprotection via manganese superoxide dismutase activation

Oxidative injury due to chronic nitric oxide synthase inhibition in rat: effect of regular exercise on the heart

Catalase in vitro

Direct evidence that oxygen-derived free radicals contribute to postischemic myocardial dysfunction in the intact dog.

The isolated blood and perfusion fluid perfused heart.

Demonstration of free radical generation in "stunned" myocardium of intact dogs with the use of the spin trap alpha-phenyl N-tert-butyl nitrone

Overexpression of MnSOD protects against myocardial ischemia/reperfusion injury in transgenic mice.

The early and late phases of preconditioning against myocardial stunning and the essential role of oxyradicals in the late phase: an overview.

Improvement of postischemic myocardial function and metabolism induced by administration of deferoxamine at the time of reflow: the role of iron in the pathogenesis of reperfusion injury.

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10.1152/AJPHEART.01208.2003

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15031126

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