[Endoplasmic reticulum stress: from physiology to pathogenesis of type 2 diabetes].

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[Interaction between environment and genetic background in type 2 diabetes: lessons from animal models].Extracellular deposition of mouse senile AApoAII amyloid fibrils induced different unfolded protein responses in the liver, kidney, and heart.

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[Endoplasmic reticulum stress: from physiology to pathogenesis of type 2 diabetes].

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2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年學術文章

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2013年學術文章

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name

[Endoplasmic reticulum stress: from physiology to pathogenesis of type 2 diabetes].

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[Endoplasmic reticulum stress: from physiology to pathogenesis of type 2 diabetes].

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type

Item

label

[Endoplasmic reticulum stress: from physiology to pathogenesis of type 2 diabetes].

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[Endoplasmic reticulum stress: from physiology to pathogenesis of type 2 diabetes].

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[Endoplasmic reticulum stress: from physiology to pathogenesis of type 2 diabetes].

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[Endoplasmic reticulum stress: from physiology to pathogenesis of type 2 diabetes].

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P1476

[Endoplasmic reticulum stress: from physiology to pathogenesis of type 2 diabetes].

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P2093

Mélissa Flamment

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P2860

Coupling of stress in the ER to activation of JNK protein kinases by transmembrane protein kinase IRE1

Endoplasmic reticulum stress and type 2 diabetes

EIF2AK3, encoding translation initiation factor 2-alpha kinase 3, is mutated in patients with Wolcott-Rallison syndrome

Translational regulation of proinsulin biosynthesis and proinsulin conversion in the pancreatic beta-cell

Translational control is required for the unfolded protein response and in vivo glucose homeostasis

Hepatic steatosis: a role for de novo lipogenesis and the transcription factor SREBP-1c

Stimulation of autophagy improves endoplasmic reticulum stress-induced diabetes

Regulation of hepatic lipogenesis by the transcription factor XBP1

Endoplasmic reticulum stress links obesity, insulin action, and type 2 diabetes

Selective inhibition of eukaryotic translation initiation factor 2 alpha dephosphorylation potentiates fatty acid-induced endoplasmic reticulum stress and causes pancreatic beta-cell dysfunction and apoptosis

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756-764

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scholarly article

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10.1051/MEDSCI/2013298015

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Af-Faransiis

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8-9

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Fabienne Foufelle

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2013-08-01T00:00:00Z

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256449239

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24005631

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type-2 diabetes

endoplasmic reticulum

[Endoplasmic reticulum stress: from physiology to pathogenesis of type 2 diabetes].

about

P2860

P2860

[Endoplasmic reticulum stress: from physiology to pathogenesis of type 2 diabetes].

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