Fast-twitch skeletal muscles of dystrophic mouse pups are resistant to injury from acute mechanical stress.
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Skeletal muscle expression of the adhesion-GPCR CD97: CD97 deletion induces an abnormal structure of the sarcoplasmatic reticulum but does not impair skeletal muscle functionAbsence of alpha 7 integrin in dystrophin-deficient mice causes a myopathy similar to Duchenne muscular dystrophyThe SH3 and cysteine-rich domain 3 (Stac3) gene is important to growth, fiber composition, and calcium release from the sarcoplasmic reticulum in postnatal skeletal muscle.Microtubules underlie dysfunction in duchenne muscular dystrophy.Blastocyst injection of wild type embryonic stem cells induces global corrections in mdx mice.Functional muscle analysis of the Tcap knockout mouse.Influence of longitudinal radiation exposure from microcomputed tomography scanning on skeletal muscle function and metabolic activity in female CD-1 miceDifferential requirement for utrophin in the induced pluripotent stem cell correction of muscle versus fat in muscular dystrophy mice.Neuronal nitric oxide synthase-rescue of dystrophin/utrophin double knockout mice does not require nNOS localization to the cell membraneMonitoring murine skeletal muscle function for muscle gene therapy.TAT-μUtrophin mitigates the pathophysiology of dystrophin and utrophin double-knockout mice.Adaptive strength gains in dystrophic muscle exposed to repeated bouts of eccentric contractionChronic administration of a leupeptin-derived calpain inhibitor fails to ameliorate severe muscle pathology in a canine model of duchenne muscular dystrophyFoxj3 transcriptionally activates Mef2c and regulates adult skeletal muscle fiber type identity.Selective Connexin43 Inhibition Prevents Isoproterenol-Induced Arrhythmias and Lethality in Muscular Dystrophy Mice.Adaptive and nonadaptive responses to voluntary wheel running by mdx miceClassical and adaptive control of ex vivo skeletal muscle contractions using Functional Electrical Stimulation (FES)Challenges and opportunities in dystrophin-deficient cardiomyopathy gene therapy.Evaluation of muscle function of the extensor digitorum longus muscle ex vivo and tibialis anterior muscle in situ in mice.Enzyme replacement therapy rescues weakness and improves muscle pathology in mice with X-linked myotubular myopathy.Endurance capacity in maturing mdx mice is markedly enhanced by combined voluntary wheel running and green tea extractC-terminal-truncated microdystrophin recruits dystrobrevin and syntrophin to the dystrophin-associated glycoprotein complex and reduces muscular dystrophy in symptomatic utrophin/dystrophin double-knockout miceAdeno-associated virus-mediated microdystrophin expression protects young mdx muscle from contraction-induced injury.Role of dystroglycan in limiting contraction-induced injury to the sarcomeric cytoskeleton of mature skeletal muscle.Malformed mdx myofibers have normal cytoskeletal architecture yet altered EC coupling and stress-induced Ca2+ signaling.Genomic removal of a therapeutic mini-dystrophin gene from adult mice elicits a Duchenne muscular dystrophy-like phenotypeTargeting RNA to treat neuromuscular disease.The dystrophin-glycoprotein complex in the prevention of muscle damage.Membrane Injury and Repair in the Muscular Dystrophies.Efficient single muscle fiber isolation from alcohol-fixed adult muscle following β-galactosidase staining for satellite cell detectionMyosin light chain phosphorylation is required for peak power output of mouse fast skeletal muscle in vitro.Defective excitation-contraction coupling is partially responsible for impaired contractility in hindlimb muscles of Stac3 knockout mice.Catalase overexpression does not impair extensor digitorum longus muscle function in normal mice.Disruption of action potential and calcium signaling properties in malformed myofibers from dystrophin-deficient mice.Reducing sarcolipin expression mitigates Duchenne muscular dystrophy and associated cardiomyopathy in mice.Passive mechanical properties of maturing extensor digitorum longus are not affected by lack of dystrophin.A novel SNaPshot assay to detect the mdx mutation.Ryanodine channel complex stabilizer compound S48168/ARM210 as a disease modifier in dystrophin-deficient mdx mice: proof-of-concept study and independent validation of efficacy.Non-invasive assessment of muscle injury in healthy and dystrophic animals with electrical impedance myography.Nutritional Health Considerations for Persons with Spinal Cord Injury.
P2860
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P2860
Fast-twitch skeletal muscles of dystrophic mouse pups are resistant to injury from acute mechanical stress.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh
2002年學術文章
@zh-hant
name
Fast-twitch skeletal muscles o ...... from acute mechanical stress.
@en
Fast-twitch skeletal muscles o ...... from acute mechanical stress.
@nl
type
label
Fast-twitch skeletal muscles o ...... from acute mechanical stress.
@en
Fast-twitch skeletal muscles o ...... from acute mechanical stress.
@nl
prefLabel
Fast-twitch skeletal muscles o ...... from acute mechanical stress.
@en
Fast-twitch skeletal muscles o ...... from acute mechanical stress.
@nl
P2093
P1476
Fast-twitch skeletal muscles o ...... from acute mechanical stress.
@en
P2093
James T Stull
Krista M Marschner
Robert J Talmadge
Robert W Grange
Thomas G Gainer
P304
P356
10.1152/AJPCELL.00450.2001
P577
2002-10-01T00:00:00Z