Absence of glial fibrillary acidic protein and vimentin prevents hypertrophy of astrocytic processes and improves post-traumatic regeneration. - Wikidata - awgldk - TriplyDB

Absence of glial fibrillary acidic protein and vimentin prevents hypertrophy of astrocytic processes and improves post-traumatic regeneration.

Absence of glial fibrillary acidic protein and vimentin prevents hypertrophy of astrocytic processes and improves post-traumatic regeneration.

http://www.wikidata.org/entity/Q44911153

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Astrogliosis in CNS pathologies: is there a role for microglia?Selective induction of astrocytic gliosis generates deficits in neuronal inhibition Molecular dissection of reactive astrogliosis and glial scar formation Neuroprotection for traumatic brain injury Structural remodeling of astrocytes in the injured CNS Glial cells in (patho)physiology Systemic simvastatin rescues retinal ganglion cells from optic nerve injury possibly through suppression of astroglial NF-κB activation Metabotropic glutamate receptor-mediated signaling in neuroglia Role of gap junction protein connexin43 in astrogliosis induced by brain injury Ndel1 promotes axon regeneration via intermediate filaments Photomechanical wave-driven delivery of siRNAs targeting intermediate filament proteins promotes functional recovery after spinal cord injury in rats Attenuating astrocyte activation accelerates plaque pathogenesis in APP/PS1 mice.Ependymal ciliary dysfunction and reactive astrocytosis in a reorganized subventricular zone after stroke Acute astrocyte activation in brain detected by MRI: new insights into T(1) hypointensity.Plasticity of astrocytic coverage and glutamate transporter expression in adult mouse cortex.A novel and efficient gene transfer strategy reduces glial reactivity and improves neuronal survival and axonal growth in vitro.Modulation of inflammatory responses after global ischemia by transplanted umbilical cord matrix stem cells Mechanically induced reactive gliosis causes ATP-mediated alterations in astrocyte stiffness.Oroxylin A promotes retinal ganglion cell survival in a rat optic nerve crush model Amelioration of cold injury-induced cortical brain edema formation by selective endothelin ETB receptor antagonists in mice Brain inflammation is a common feature of HIV-infected patients without HIV encephalitis or productive brain infection.The generation of definitive neural stem cells from PiggyBac transposon-induced pluripotent stem cells can be enhanced by induction of the NOTCH signaling pathway.Re-establishing the regenerative potential of central nervous system axons in postnatal mice.Expression profiles of nestin and synemin in reactive astrocytes and Müller cells following retinal injury: a comparison with glial fibrillar acidic protein and vimentin Multiple implants do not aggravate the tissue reaction in rat brain.Role of astrocytes in manganese mediated neurotoxicity.Reactive astrocytes promote axonal remodeling and neurological recovery after stroke Versatile and simple approach to determine astrocyte territories in mouse neocortex and hippocampus.Plasticity response in the contralesional hemisphere after subtle neurotrauma: gene expression profiling after partial deafferentation of the hippocampus.Beneficial effects of gfap/vimentin reactive astrocytes for axonal remodeling and motor behavioral recovery in mice after stroke.Axonal regeneration after sciatic nerve lesion is delayed but complete in GFAP- and vimentin-deficient mice Comparison of intravital thinned skull and cranial window approaches to study CNS immunobiology in the mouse cortex.The role of attenuated astrocyte activation in infantile neuronal ceroid lipofuscinosis.Lipocalin-2 Is a chemokine inducer in the central nervous system: role of chemokine ligand 10 (CXCL10) in lipocalin-2-induced cell migration.Isoflurane does not cause neuroapoptosis but reduces astroglial processes in young adult mice Astrogliosis involves activation of retinoic acid-inducible gene-like signaling in the innate immune response after spinal cord injury.Redefining the concept of reactive astrocytes as cells that remain within their unique domains upon reaction to injury.Neuroprotection from stroke in the absence of MHCI or PirB.Neurological diseases as primary gliopathies: a reassessment of neurocentrism Repeated transcranial low-level laser therapy for traumatic brain injury in mice: biphasic dose response and long-term treatment outcome.

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P2860

Absence of glial fibrillary acidic protein and vimentin prevents hypertrophy of astrocytic processes and improves post-traumatic regeneration.

http://www.wikidata.org/entity/Q44911153

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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2004年學術文章

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name

Absence of glial fibrillary ac ...... s post-traumatic regeneration.

@en

Absence of glial fibrillary ac ...... s post-traumatic regeneration.

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type

label

Absence of glial fibrillary ac ...... s post-traumatic regeneration.

@en

Absence of glial fibrillary ac ...... s post-traumatic regeneration.

@nl

prefLabel

Absence of glial fibrillary ac ...... s post-traumatic regeneration.

@en

Absence of glial fibrillary ac ...... s post-traumatic regeneration.

@nl

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JNEUROSCI.0820-04.2004

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Journal of Neuroscience

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Absence of glial fibrillary ac ...... es post-traumatic regeneration

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P2093

Camilla Eliasson

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Claes-Henric Berthold

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Eric Bushong

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Lizhen Li

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Mark Ellisman

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Milos Pekny

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Sofia Blom

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Todd E Morgan

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Ulrika Wilhelmsson

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5016-5021

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scholarly article

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10.1523/JNEUROSCI.0820-04.2004

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21

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2004-05-01T00:00:00Z

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