Differences in inflammation, MMP activation and collagen damage account for gender difference in murine cardiac rupture following myocardial infarction. - Wikidata - awgldk - TriplyDB

Differences in inflammation, MMP activation and collagen damage account for gender difference in murine cardiac rupture following myocardial infarction.

Differences in inflammation, MMP activation and collagen damage account for gender difference in murine cardiac rupture following myocardial infarction.

http://www.wikidata.org/entity/Q44913591

about

Medical therapies with adult stem/progenitor cells (MSCs): a backward journey from dramatic results in vivo to the cellular and molecular explanations Donepezil, anti-Alzheimer's disease drug, prevents cardiac rupture during acute phase of myocardial infarction in mice Intravenous hMSCs improve myocardial infarction in mice because cells embolized in lung are activated to secrete the anti-inflammatory protein TSG-6 Mechanistic Pathways of Sex Differences in Cardiovascular Disease.Influence of sex hormones and phytoestrogens on heart disease in men and women Combining experimental and mathematical modeling to reveal mechanisms of macrophage-dependent left ventricular remodeling Early activation of matrix metalloproteinases underlies the exacerbated systolic and diastolic dysfunction in mice lacking TIMP3 following myocardial infarction.An association between gene expression and better survival in female mice following myocardial infarction.Decreased fibrocyte number is associated with atherosclerotic plaque instability in man.Predictive factors of cardiac rupture in patients with ST-elevation myocardial infarction Selective homocysteine lowering gene transfer improves infarct healing, attenuates remodelling, and enhances diastolic function after myocardial infarction in mice Differential roles of cardiac and leukocyte derived macrophage migration inhibitory factor in inflammatory responses and cardiac remodelling post myocardial infarction.Mesenchymal stem/stromal cells (MSCs): role as guardians of inflammation.Mitochondrial cyclophilin-D as a potential therapeutic target for post-myocardial infarction heart failure Gender disparity in the role of TLR2 in post-ischemic myocardial inflammation and injury Sex differences in porcine left ventricular myocardial remodeling due to right ventricular pacing.Evolving paradigms for repair of tissues by adult stem/progenitor cells (MSCs).The Importance of Biological Sex and Estrogen in Rodent Models of Cardiovascular Health and Disease Matrix metalloproteinase (MMP)-9: a proximal biomarker for cardiac remodeling and a distal biomarker for inflammation.Risk of cardiac rupture after acute myocardial infarction is related to a risk of hemorrhage Pro-inflammatory action of MIF in acute myocardial infarction via activation of peripheral blood mononuclear cells.Structural composition of myocardial infarction scar in middle-aged male and female rats: does sex matter?Macrophage migration inhibitory factor for the early prediction of infarct size.Effects of gender, ejection fraction and weight on cardiac force development in patients undergoing cardiac surgery--an experimental examination.Using MRI to predict future adverse cardiac remodelling in a male mouse model of myocardial infarction.Emerging concepts in cardiac matrix biology.Commonly asked questions: thrombolytic therapy in the management of acute stroke.Targeting matrix metalloproteinase activity and expression for the treatment of viral myocarditis.Mathematical modeling of cardiac growth and remodeling.Myocardial expression of PDECGF is associated with extracellular matrix remodeling in experimental myocardial infarction in rats.Sex differences in myocardial infarction and rupture.Mouse strain determines the outcome of wound healing after myocardial infarction.Inhibition of the Renin-Angiotensin System Post Myocardial Infarction Prevents Inflammation-Associated Acute Cardiac Rupture.Intravenous administration of bone marrow mesenchymal stromal cells is safe for the lung in a chronic myocardial infarction model.Factors related to heart rupture in acute coronary syndromes in the Global Registry of Acute Coronary Events.Twinkle overexpression prevents cardiac rupture after myocardial infarction by alleviating impaired mitochondrial biogenesis.

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Differences in inflammation, MMP activation and collagen damage account for gender difference in murine cardiac rupture following myocardial infarction.

http://www.wikidata.org/entity/Q44913591

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2007 nî lūn-bûn

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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2007年學術文章

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2007年學術文章

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Differences in inflammation, M ...... llowing myocardial infarction.

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Differences in inflammation, M ...... llowing myocardial infarction.

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Differences in inflammation, M ...... llowing myocardial infarction.

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Differences in inflammation, M ...... llowing myocardial infarction.

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J.YJMCC.2007.06.011

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Journal of Molecular and Cellular Cardiology

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Anthony M Dart

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Helen Kiriazis

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Lu Fang

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Xiao-Jun Du

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Xiao-Lei Moore

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Xiao-Ming Gao

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Yean Leng Lim

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Yidan Su

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Ziqiu Ming

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scholarly article

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