Bcl-2 is a key factor for cardiac fibroblast resistance to programmed cell death.
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Role of mitochondria in aspirin-induced apoptosis in human gastric epithelial cellsInfarct-induced steroidogenic acute regulatory protein: a survival role in cardiac fibroblastsMature neurons dynamically restrict apoptosis via redundant premitochondrial brakes.Thrombin increases lung fibroblast survival while promoting alveolar epithelial cell apoptosis via the endoplasmic reticulum stress marker, CCAAT enhancer-binding homologous proteinTanshinoneIIA and cryptotanshinone protect against hypoxia-induced mitochondrial apoptosis in H9c2 cells.Overexpression of GRIM-19, a mitochondrial respiratory chain complex I protein, suppresses hepatocellular carcinoma growth.NF-κB inhibition compromises cardiac fibroblast viability under hypoxia.In vitro and in vivo effects of bone marrow stem cells on cardiac structure and function.Protective role of 5-azacytidine on myocardial infarction is associated with modulation of macrophage phenotype and inhibition of fibrosis.AKT2 Blocks Nucleus Translocation of Apoptosis-Inducing Factor (AIF) and Endonuclease G (EndoG) While Promoting Caspase Activation during Cardiac Ischemia.Mobilized bone marrow progenitor cells serve as donors of cytoprotective genes for cardiac repairCardioprotection by combination of three compounds from ShengMai preparations in mice with myocardial ischemia/reperfusion injury through AMPK activation-mediated mitochondrial fission.Mouse embryonic stem cells undergo charontosis, a novel programmed cell death pathway dependent upon cathepsins, p53, and EndoG, in response to etoposide treatment.microRNA-21 promotes cardiac fibrosis and development of heart failure with preserved left ventricular ejection fraction by up-regulating Bcl-2.Mouse Lung Fibroblast Resistance to Fas-Mediated Apoptosis Is Dependent on the Baculoviral Inhibitor of Apoptosis Protein 4 and the Cellular FLICE-Inhibitory Protein.Xinfuli improves cardiac function, histopathological changes and attenuate cardiomyocyte apoptosis in rats with doxorubicin-induced cardiotoxicity.Cellular senescence: from growth arrest to immunogenic conversion.Intracellular signaling of cardiac fibroblasts.Myocardial apoptosis in heart disease: does the emperor have clothes?MicroRNAs mediate the cardioprotective effect of angiotensin-converting enzyme inhibition in acute kidney injury.Differential regulation of the intrinsic pathway of apoptosis in brain and liver during ageing.Poly(ADP-ribose)polymerase activation mediates lung epithelial cell death in vitro but is not essential in hyperoxia-induced lung injury.Extract of Sheng-Mai-San Ameliorates Myocardial Ischemia-Induced Heart Failure by Modulating Ca2+-Calcineurin-Mediated Drp1 Signaling Pathways.Hypoxia induces cardiac fibroblast proliferation and phenotypic switch: a role for caveolae and caveolin-1/PTEN mediated pathway.Hyperglycaemia protects the heart after myocardial infarction: aspects of programmed cell survival and cell death.Paracrine effects of hypoxic fibroblast-derived factors on the MPT-ROS threshold and viability of adult rat cardiac myocytes.Combined hypoxia and sodium nitrite pretreatment for cardiomyocyte protection in vitro.Cyclovirobuxinum D alleviates cardiac hypertrophy in hyperthyroid rats by preventing apoptosis of cardiac cells and inhibiting the p38 mitogen-activated protein kinase signaling pathway.Nitidine chloride induces apoptosis, cell cycle arrest, and synergistic cytotoxicity with doxorubicin in breast cancer cells.Mechanisms in cardiac fibroblast growth: an obligate role for Skp2 and FOXO3a in ERK1/2 MAPK-dependent regulation of p27kip1.
P2860
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P2860
Bcl-2 is a key factor for cardiac fibroblast resistance to programmed cell death.
description
2004 nî lūn-bûn
@nan
2004年の論文
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2004年学术文章
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2004年学术文章
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2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
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2004年學術文章
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2004年學術文章
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name
Bcl-2 is a key factor for cardiac fibroblast resistance to programmed cell death.
@en
Bcl-2 is a key factor for cardiac fibroblast resistance to programmed cell death.
@nl
type
label
Bcl-2 is a key factor for cardiac fibroblast resistance to programmed cell death.
@en
Bcl-2 is a key factor for cardiac fibroblast resistance to programmed cell death.
@nl
prefLabel
Bcl-2 is a key factor for cardiac fibroblast resistance to programmed cell death.
@en
Bcl-2 is a key factor for cardiac fibroblast resistance to programmed cell death.
@nl
P2093
P2860
P356
P1476
Bcl-2 is a key factor for cardiac fibroblast resistance to programmed cell death
@en
P2093
Manel Ballester
Maritza Mayorga
Núria Bahi
P2860
P304
34882-34889
P356
10.1074/JBC.M404616200
P407
P577
2004-06-07T00:00:00Z