Action potential duration determines sarcoplasmic reticulum Ca2+ reloading in mammalian ventricular myocytes.
about
Control of Ca2+ release by action potential configuration in normal and failing murine cardiomyocytesA mathematical treatment of integrated Ca dynamics within the ventricular myocyte.Ionic mechanisms of cellular electrical and mechanical abnormalities in Brugada syndrome.Complex excitation dynamics underlie polymorphic ventricular tachycardia in a transgenic rabbit model of long QT syndrome type 1Restitution of Ca(2+) release and vulnerability to arrhythmias.The mechanisms of calcium cycling and action potential dynamics in cardiac alternansModels of HERG gatingTransient outward potassium current, 'Ito', phenotypes in the mammalian left ventricle: underlying molecular, cellular and biophysical mechanisms.Role of the transient outward potassium current in the genesis of early afterdepolarizations in cardiac cells.Simulation of Ca-calmodulin-dependent protein kinase II on rabbit ventricular myocyte ion currents and action potentials.Transient outward potassium current and Ca2+ homeostasis in the heart: beyond the action potential.Longitudinal study of cardiac remodelling in rabbits following infarction.Modulation of ventricular transient outward K⁺ current by acidosis and its effects on excitation-contraction coupling.Melanocyte-like cells in the heart and pulmonary veins contribute to atrial arrhythmia triggers.Ca(2+)-activated chloride channel activity during Ca(2+) alternans in ventricular myocytes.Alternans in atria: Mechanisms and clinical relevance.Species-Dependent Mechanisms of Cardiac Arrhythmia: A Cellular Focus.Persistent increases in Ca(2+) influx through Cav1.2 shortens action potential and causes Ca(2+) overload-induced afterdepolarizations and arrhythmias.Impaired oxidative metabolism and calcium mishandling underlie cardiac dysfunction in a rat model of post-acute isoproterenol-induced cardiomyopathy.Comparison of contraction and calcium handling between right and left ventricular myocytes from adult mouse heart: a role for repolarization waveform.Intermittent hypoxia protects cardiomyocytes against ischemia-reperfusion injury-induced alterations in Ca2+ homeostasis and contraction via the sarcoplasmic reticulum and Na+/Ca2+ exchange mechanisms.Altered Repolarization Reserve in Failing Rabbit Ventricular Myocytes: Calcium and β-Adrenergic Effects on Delayed- and Inward-Rectifier Potassium Currents.Membrane potential determines calcium alternans through modulation of SR Ca2+ load and L-type Ca2+ current.Calcium-activated chloride current determines action potential morphology during calcium alternans in atrial myocytes.
P2860
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P2860
Action potential duration determines sarcoplasmic reticulum Ca2+ reloading in mammalian ventricular myocytes.
description
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name
Action potential duration dete ...... ammalian ventricular myocytes.
@en
Action potential duration dete ...... ammalian ventricular myocytes.
@nl
type
label
Action potential duration dete ...... ammalian ventricular myocytes.
@en
Action potential duration dete ...... ammalian ventricular myocytes.
@nl
prefLabel
Action potential duration dete ...... ammalian ventricular myocytes.
@en
Action potential duration dete ...... ammalian ventricular myocytes.
@nl
P2860
P50
P1476
Action potential duration dete ...... ammalian ventricular myocytes.
@en
P2093
José L Puglisi
P2860
P304
P356
10.1113/JPHYSIOL.2004.067959
P407
P577
2004-07-08T00:00:00Z