Selective inactivation or reconstitution of adenosine A2A receptors in bone marrow cells reveals their significant contribution to the development of ischemic brain injury. - Wikidata - awgldk - TriplyDB

Selective inactivation or reconstitution of adenosine A2A receptors in bone marrow cells reveals their significant contribution to the development of ischemic brain injury.

Selective inactivation or reconstitution of adenosine A2A receptors in bone marrow cells reveals their significant contribution to the development of ischemic brain injury.

http://www.wikidata.org/entity/Q45078353

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Adenosine receptors as therapeutic targets A2A receptors in inflammation and injury: lessons learned from transgenic animals Potential therapeutic interest of adenosine A2A receptors in psychiatric disorders.Adenosine A2A receptors modulate acute injury and neuroinflammation in brain ischemia Adenosine receptors as drug targets--what are the challenges?Adenosine A(2A) receptors in early ischemic vascular injury after subarachnoid hemorrhage. Laboratory investigation.Inactivation of the adenosine A2A receptor protects apolipoprotein E-deficient mice from atherosclerosis The A2B adenosine receptor protects against inflammation and excessive vascular adhesion.Adenosine through the A2A adenosine receptor increases IL-1β in the brain contributing to anxiety Perivascular AQP4 dysregulation in the hippocampal CA1 area after traumatic brain injury is alleviated by adenosine A2A receptor inactivation.Absence of the adenosine A2A receptor confers pulmonary arterial hypertension and increased pulmonary vascular remodeling in mice.Adenosine A2A receptor activation and macrophage-mediated experimental glomerulonephritis.Neurobiology of microglial action in CNS injuries: receptor-mediated signaling mechanisms and functional roles Genetic deletion of the adenosine A2A receptor confers postnatal development of relative myopia in mice.Adenosine A2A receptor deficiency up-regulates cystatin F expression in white matter lesions induced by chronic cerebral hypoperfusion.2',3'-cAMP, 3'-AMP, 2'-AMP and adenosine inhibit TNF-α and CXCL10 production from activated primary murine microglia via A2A receptors.Modulators of nucleoside metabolism in the therapy of brain diseases.Caffeine protects against disruptions of the blood-brain barrier in animal models of Alzheimer's and Parkinson's diseases.NF-kappaB regulation of endothelial cell function during LPS-induced toxemia and cancer.Adenosine kinase determines the degree of brain injury after ischemic stroke in mice.Past, present and future of A(2A) adenosine receptor antagonists in the therapy of Parkinson's disease.Liver damage and systemic inflammatory responses are exacerbated by the genetic deletion of CD39 in total hepatic ischemia AM-36 modulates the neutrophil inflammatory response and reduces breakdown of the blood brain barrier after endothelin-1 induced focal brain ischaemia.The role of ATP and adenosine in the brain under normoxic and ischemic conditions Introduction to adenosine receptors as therapeutic targets.Adenosine receptor signaling in the brain immune system.Activation of microglial cells triggers a release of brain-derived neurotrophic factor (BDNF) inducing their proliferation in an adenosine A2A receptor-dependent manner: A2A receptor blockade prevents BDNF release and proliferation of microglia.Targeting the hypoxia-adenosinergic signaling pathway to improve the adoptive immunotherapy of cancer.Dual roles of the adenosine A2a receptor in autoimmune neuroinflammation Neuroprotection by caffeine in the MPTP model of parkinson's disease and its dependence on adenosine A2A receptors.Plasma glutamate-modulated interaction of A2AR and mGluR5 on BMDCs aggravates traumatic brain injury-induced acute lung injury Increases in cerebrospinal fluid caffeine concentration are associated with favorable outcome after severe traumatic brain injury in humans Adenosine kinase is a new therapeutic target to prevent ischemic neuronal death.Adenosine A2A receptor deficiency alleviates blast-induced cognitive dysfunction.Local glutamate level dictates adenosine A2A receptor regulation of neuroinflammation and traumatic brain injury Adenosine 2A receptor: a crucial neuromodulator with bidirectional effect in neuroinflammation and brain injury.Adenosine receptor subtypes and the heart failure phenotype: translating lessons from mice to man.Adenosine receptors and epilepsy: current evidence and future potential.How does adenosine control neuronal dysfunction and neurodegeneration?Pathological overproduction: the bad side of adenosine.

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Selective inactivation or reconstitution of adenosine A2A receptors in bone marrow cells reveals their significant contribution to the development of ischemic brain injury.

http://www.wikidata.org/entity/Q45078353

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2004 nî lūn-bûn

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Selective inactivation or reco ...... ment of ischemic brain injury.

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Selective inactivation or reco ...... ment of ischemic brain injury.

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Selective inactivation or reco ...... ment of ischemic brain injury.

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Selective inactivation or reco ...... ment of ischemic brain injury.

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Jiang-Fan Chen

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Juliana Mariani

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Liqun Yu

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Michael Moskowitz

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Yumei Wang

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Zhihong Huang

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P2860

Neuroprotection by caffeine and adenosine A2A receptor blockade of beta-amyloid neurotoxicity

Peak plasma interleukin-6 and other peripheral markers of inflammation in the first week of ischaemic stroke correlate with brain infarct volume, stroke severity and long-term outcome

Role of G-protein-coupled adenosine receptors in downregulation of inflammation and protection from tissue damage

Molecular approach to adenosine receptors: receptor-mediated mechanisms of tissue protection.

Adenosine A2A receptor activation decreases reperfusion injury associated with high-flow reperfusion.

Protection from ischemic liver injury by activation of A2A adenosine receptors during reperfusion: inhibition of chemokine induction.

Immunohistochemical localization of adenosine A2A receptors in the rat central nervous system.

Aggravated brain damage after hypoxic ischemia in immature adenosine A2A knockout mice.

Neuroprotective role of adenosine in cerebral ischaemia.

Hyperalgesia, anxiety, and decreased hypoxic neuroprotection in mice lacking the adenosine A1 receptor.

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10

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