Effects of baclofen on spinal reflexes and persistent inward currents in motoneurons of chronic spinal rats with spasticity.
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Recovery of neuronal and network excitability after spinal cord injury and implications for spasticityOverexpression of BDNF increases excitability of the lumbar spinal network and leads to robust early locomotor recovery in completely spinalized ratsThoracic 9 Spinal Transection-Induced Model of Muscle Spasticity in the Rat: A Systematic Electrophysiological and Histopathological CharacterizationRecovery of motoneuron and locomotor function after spinal cord injury depends on constitutive activity in 5-HT2C receptors.The comparative study of clinical efficacy and safety of baclofen vs tolperisone in spasticity caused by spinal cord injuryEffects of baclofen on motor units paralysed by chronic cervical spinal cord injury.Management of spasticity after spinal cord injury: current techniques and future directions.Intrathecal baclofen therapy for neurological disorders: a sound knowledge base but many challenges remain.Progressive changes in synaptic inputs to motoneurons in adult sacral spinal cord of a mouse model of amyotrophic lateral sclerosisMotoneuron excitability and muscle spasms are regulated by 5-HT2B and 5-HT2C receptor activity.Polysynaptic excitatory postsynaptic potentials that trigger spasms after spinal cord injury in rats are inhibited by 5-HT1B and 5-HT1F receptorsA systematic review of the effects of pharmacological agents on walking function in people with spinal cord injury.The transformation of synaptic to system plasticity in motor output from the sacral cord of the adult mouse.Selective corticospinal tract injury in the rat induces primary afferent fiber sprouting in the spinal cord and hyperreflexiaNMDA induces persistent inward and outward currents that cause rhythmic bursting in adult rodent motoneurons.Constitutively active 5-HT2/α1 receptors facilitate muscle spasms after human spinal cord injuryReduction of spinal sensory transmission by facilitation of 5-HT1B/D receptors in noninjured and spinal cord-injured humans.Changes in sensory-evoked synaptic activation of motoneurons after spinal cord injury in man.Selective effects of baclofen on use-dependent modulation of GABAB inhibition after tetraplegia.Motor unit.Human spinal cord injury: motor unit properties and behaviour.Intrathecal baclofen therapy in children with severe spasticity: Outcome and complications.The use of intrathecal baclofen in pain management.Intrathecal baclofen therapy in children with acquired brain injuries after drowning: A case series.Intrathecal Baclofen therapy in Germany: Proceedings of the IAB-Interdisciplinary Working Group for Movement Disorders Consensus Meeting.Opposite, bidirectional shifts in excitation and inhibition in specific types of dorsal horn interneurons are associated with spasticity and pain post-SCI.Circuits generating corticomuscular coherence investigated using a biophysically based computational model. I. Descending systemsSpatiotemporal correlation of spinal network dynamics underlying spasms in chronic spinalized mice.Global gene expression analysis of rodent motor neurons following spinal cord injury associates molecular mechanisms with development of postinjury spasticity.Links between electrophysiological and molecular pathology of amyotrophic lateral sclerosis.Degeneration of serotonergic neurons in amyotrophic lateral sclerosis: a link to spasticity.Persistent sodium currents and repetitive firing in motoneurons of the sacrocaudal spinal cord of adult rats.Demystifying Spasticity: Reply to Dietz.5-HT2 receptor activation facilitates a persistent sodium current and repetitive firing in spinal motoneurons of rats with and without chronic spinal cord injury.Endogenous monoamine receptor activation is essential for enabling persistent sodium currents and repetitive firing in rat spinal motoneurons.Paradoxical worsening of spasticity and pain in the lower extremities after increasing the dose of intrathecal baclofen--case report.Effect of tendon vibration during wide-pulse neuromuscular electrical stimulation (NMES) on muscle force production in people with spinal cord injury (SCI).Degeneration of serotonin neurons triggers spasticity in amyotrophic lateral sclerosis.Motor Neurons.Genioglossus reflex responses to negative upper airway pressure are altered in people with tetraplegia and obstructive sleep apnoea.
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Effects of baclofen on spinal reflexes and persistent inward currents in motoneurons of chronic spinal rats with spasticity.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh-hant
name
Effects of baclofen on spinal ...... c spinal rats with spasticity.
@en
Effects of baclofen on spinal ...... c spinal rats with spasticity.
@nl
type
label
Effects of baclofen on spinal ...... c spinal rats with spasticity.
@en
Effects of baclofen on spinal ...... c spinal rats with spasticity.
@nl
prefLabel
Effects of baclofen on spinal ...... c spinal rats with spasticity.
@en
Effects of baclofen on spinal ...... c spinal rats with spasticity.
@nl
P2093
P2860
P356
P1476
Effects of baclofen on spinal ...... c spinal rats with spasticity.
@en
P2093
P2860
P304
P356
10.1152/JN.00164.2004
P407
P577
2004-11-01T00:00:00Z