Persistent sodium currents and repetitive firing in motoneurons of the sacrocaudal spinal cord of adult rats.
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Recovery of neuronal and network excitability after spinal cord injury and implications for spasticityIncreases in human motoneuron excitability after cervical spinal cord injury depend on the level of injuryRecovery of motoneuron and locomotor function after spinal cord injury depends on constitutive activity in 5-HT2C receptors.Heterogeneity of membrane properties in sympathetic preganglionic neurons of neonatal mice: evidence of four subpopulations in the intermediolateral nucleus.Disturbances of motor unit rate modulation are prevalent in muscles of spastic-paretic stroke survivorsBlockade of persistent sodium currents contributes to the riluzole-induced inhibition of spontaneous activity and oscillations in injured DRG neurons.Effect of fluoxetine on disease progression in a mouse model of ALS.Amphetamine increases persistent inward currents in human motoneurons estimated from paired motor-unit activityEpidural stimulation induced modulation of spinal locomotor networks in adult spinal rats.A review of the neural mechanisms of action and clinical efficiency of riluzole in treating amyotrophic lateral sclerosis: what have we learned in the last decade?Adrenergic receptors modulate motoneuron excitability, sensory synaptic transmission and muscle spasms after chronic spinal cord injury.Motoneuron excitability and muscle spasms are regulated by 5-HT2B and 5-HT2C receptor activity.Myofascial trigger points: spontaneous electrical activity and its consequences for pain induction and propagation.Dendritic spine dysgenesis contributes to hyperreflexia after spinal cord injury.Polysynaptic excitatory postsynaptic potentials that trigger spasms after spinal cord injury in rats are inhibited by 5-HT1B and 5-HT1F receptorsDecrease of mRNA Editing after Spinal Cord Injury is Caused by Down-regulation of ADAR2 that is Triggered by Inflammatory ResponsePersistent inward currents in spinal motoneurons and their influence on human motoneuron firing patterns.Effect of prolonged riluzole exposure on cultured motoneurons in a mouse model of ALS.Differential contributions of somatic and dendritic calcium-dependent potassium currents to the control of motoneuron excitability following spinal cord injury.The transformation of synaptic to system plasticity in motor output from the sacral cord of the adult mouse.Role of endogenous release of norepinephrine in muscle spasms after chronic spinal cord injuryEvidence from computer simulations for alterations in the membrane biophysical properties and dendritic processing of synaptic inputs in mutant superoxide dismutase-1 motoneurons.Spinal cord injury induces serotonin supersensitivity without increasing intrinsic excitability of mouse V2a interneurons.NMDA induces persistent inward and outward currents that cause rhythmic bursting in adult rodent motoneurons.Contribution of near-threshold currents to intrinsic oscillatory activity in rat medial entorhinal cortex layer II stellate cells.An in vitro protocol for recording from spinal motoneurons of adult ratsEstimation of the contribution of intrinsic currents to motoneuron firing based on paired motoneuron discharge records in the decerebrate catMotor neuron rescue in spinal muscular atrophy mice demonstrates that sensory-motor defects are a consequence, not a cause, of motor neuron dysfunctionMotoneuron model of self-sustained firing after spinal cord injury.Optical Imaging of the Motor Cortex Following Antidromic Activation of the Corticospinal Tract after Spinal Cord Injury.Alpha, beta and gamma motoneurons: functional diversity in the motor system's final pathway.Riluzole: what it does to spinal and brainstem neurons and how it does it.Motor unit.The activity of the serotonin receptor 2C is regulated by alternative splicing.Ionic mechanisms maintaining action potential conduction velocity at high firing frequencies in an unmyelinated axon.Diphenytoin, riluzole and lidocaine: three sodium channel blockers, with different mechanisms of action, decrease hippocampal epileptiform activity.Pacemaker and plateau potentials shape output of a developing locomotor networkA novel path to chronic proprioceptive disability with oxaliplatin: Distortion of sensory encoding.Inhibiting persistent inward sodium currents prevents myotonia.Intrinsic properties of mouse lumbar motoneurons revealed by intracellular recording in vivo.
P2860
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P2860
Persistent sodium currents and repetitive firing in motoneurons of the sacrocaudal spinal cord of adult rats.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Persistent sodium currents and ...... dal spinal cord of adult rats.
@en
Persistent sodium currents and ...... dal spinal cord of adult rats.
@nl
type
label
Persistent sodium currents and ...... dal spinal cord of adult rats.
@en
Persistent sodium currents and ...... dal spinal cord of adult rats.
@nl
prefLabel
Persistent sodium currents and ...... dal spinal cord of adult rats.
@en
Persistent sodium currents and ...... dal spinal cord of adult rats.
@nl
P2093
P2860
P356
P1476
Persistent sodium currents and ...... dal spinal cord of adult rats.
@en
P2093
P2860
P304
P356
10.1152/JN.00335.2005
P407
P577
2005-11-09T00:00:00Z