Acute renal failure in endotoxemia is dependent on caspase activation.
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Septic AKI in ICU patients. diagnosis, pathophysiology, and treatment type, dosing, and timing: a comprehensive review of recent and future developmentsAutophagy, Innate Immunity and Tissue Repair in Acute Kidney InjurySeptic acute kidney injury: the culprit is inflammatory apoptosis rather than ischemic necrosisTherapeutic translation in acute kidney injury: the epithelial/endothelial axisProtective effect of resin adsorption on septic plasma-induced tubular injury.Noble gas (argon and xenon)-saturated cold storage solutions reduce ischemia-reperfusion injury in a rat model of renal transplantation.Suppression of mitochondrial biogenesis through toll-like receptor 4-dependent mitogen-activated protein kinase kinase/extracellular signal-regulated kinase signaling in endotoxin-induced acute kidney injury.Peptide nanofibers preconditioned with stem cell secretome are renoprotective.Surgical sepsis and organ crosstalk: the role of the kidneyHemodynamic changes in the kidney in a pediatric rat model of sepsis-induced acute kidney injury.Role of mitochondrial oxidants in an in vitro model of sepsis-induced renal injury.Role of circulating fibroblast growth factor-2 in lipopolysaccharide-induced acute kidney injury in mice.Xenon Protects Against Septic Acute Kidney Injury via miR-21 Target Signaling Pathway.Renal hemodynamic, inflammatory, and apoptotic responses to lipopolysaccharide in HO-1-/- miceResveratrol improves renal microcirculation, protects the tubular epithelium, and prolongs survival in a mouse model of sepsis-induced acute kidney injury.Apoptosis and acute kidney injuryEndotoxemia alters tight junction gene and protein expression in the kidneyzVAD-fmk prevents cisplatin-induced cleavage of autophagy proteins but impairs autophagic flux and worsens renal function.Podocyte apoptosis is prevented by blocking the Toll-like receptor pathway.Sepsis-induced organ failure is mediated by different pathways in the kidney and liver: acute renal failure is dependent on MyD88 but not renal cell apoptosis.Septic acute kidney injury: molecular mechanisms and the importance of stratification and targeting therapy.Apoptosis and caspases regulate death and inflammation in sepsis.Kidney and Liver Injuries After Major Burns in Rats Are Prevented by Resolvin D2.Polymyxin-B hemoperfusion inactivates circulating proapoptotic factorsBCL2 genetic variants are associated with acute kidney injury in septic shock*The impact of aging on kidney repair.Antiapoptotic Effect of Paricalcitol in Gentamicin-induced Kidney InjuryInhibiting glycogen synthase kinase-3 reduces endotoxaemic acute renal failure by down-regulating inflammation and renal cell apoptosis.Pathophysiology of acute kidney injuryPharmacological targets in the renal peritubular microenvironment: implications for therapy for sepsis-induced acute kidney injury.Pathophysiology and management of septic acute kidney injury.TNF causes changes in glomerular endothelial permeability and morphology through a Rho and myosin light chain kinase-dependent mechanism.Synergistic effect of apoptosis and necroptosis inhibitors in cisplatin-induced nephrotoxicity.Alpha-lipoic acid attenuates lipopolysaccharide-induced kidney injury.Multiple caspases mediate acute renal cell apoptosis induced by bacterial cell wall components.Early septic insult in neonatal pigs increases serum and urinary soluble Fas ligand and decreases kidney function without inducing significant renal apoptosis.Protective Effects of Antioxidant Peptide SS-31 Against Multiple Organ Dysfunctions During Endotoxemia.A calpain inhibitor protects against fractalkine production in lipopolysaccharide-treated endothelial cellsScintigraphic comparison of renal ischemia-reperfusion injury models in rats: correlations with biochemical and histopathological findings.TNF induces caspase-dependent inflammation in renal endothelial cells through a Rho- and myosin light chain kinase-dependent mechanism.
P2860
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P2860
Acute renal failure in endotoxemia is dependent on caspase activation.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh-hant
name
Acute renal failure in endotoxemia is dependent on caspase activation.
@en
Acute renal failure in endotoxemia is dependent on caspase activation.
@nl
type
label
Acute renal failure in endotoxemia is dependent on caspase activation.
@en
Acute renal failure in endotoxemia is dependent on caspase activation.
@nl
prefLabel
Acute renal failure in endotoxemia is dependent on caspase activation.
@en
Acute renal failure in endotoxemia is dependent on caspase activation.
@nl
P2093
P1476
Acute renal failure in endotoxemia is dependent on caspase activation.
@en
P2093
Andrew W Minto
Patrick N Cunningham
Richard J Quigg
Rongqing Guo
P304
P356
10.1097/01.ASN.0000145530.73247.F5
P577
2004-12-01T00:00:00Z