Pulmonary overexpression of IL-10 augments lung fibrosis and Th2 responses induced by silica particles.
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The chemokine CCL18 causes maturation of cultured monocytes to macrophages in the M2 spectrumThe role of pro- and anti-inflammatory responses in silica-induced lung fibrosis.Silica, Silicosis, and AutoimmunityDust events, pulmonary diseases and immune systemImmune Components of Liver Damage Associated with Connective Tissue DiseasesFibrogenic and redox-related but not proinflammatory genes are upregulated in Lewis rat model of chronic silicosisFibrocytes Regulate Wilms Tumor 1-Positive Cell Accumulation in Severe Fibrotic Lung Disease.Uncoupling between inflammatory and fibrotic responses to silica: evidence from MyD88 knockout miceBlockade of Wnt/β-Catenin Pathway Aggravated Silica-Induced Lung Inflammation through Tregs Regulation on Th Immune ResponsesMacrophage silica nanoparticle response is phenotypically dependentGranuloma formation induced by low-dose chronic silica inhalation is associated with an anti-apoptotic response in Lewis ratsHsp90 regulation of fibroblast activation in pulmonary fibrosis.Accelerated and progressive and lethal liver fibrosis in mice that lack interleukin (IL)-10, IL-12p40, and IL-13Rα2.Resistance to bleomycin-induced lung fibrosis in MMP-8 deficient mice is mediated by interleukin-10CD4+CD25+Foxp3+ regulatory T cells depletion may attenuate the development of silica-induced lung fibrosis in mice.The green tea extract epigallocatechin-3-gallate inhibits irradiation-induced pulmonary fibrosis in adult rats.Innate immune processes are sufficient for driving silicosis in mice.Latent infection by γherpesvirus stimulates profibrotic mediator release from multiple cell types.Prevalence, hemodynamics, and cytokine profile of effusive-constrictive pericarditis in patients with tuberculous pericardial effusionMicrohemorrhage is an early event in the pulmonary fibrotic disease of PECAM-1 deficient FVB/n mice.Silica-directed mast cell activation is enhanced by scavenger receptorsIL-13Ralpha2 and IL-10 coordinately suppress airway inflammation, airway-hyperreactivity, and fibrosis in miceQuantification of echodensities in tuberculous pericardial effusion using fractal geometry: a proof of concept study.Sustained PI3K Activation exacerbates BLM-induced Lung Fibrosis via activation of pro-inflammatory and pro-fibrotic pathways.Macrophages and tissue injury: agents of defense or destruction?IL-17 stimulates differentiation of human anti-inflammatory macrophages and phagocytosis of apoptotic neutrophils in response to IL-10 and glucocorticoids.Host immune responses to chlamydial inclusion membrane proteins B and C in Chlamydia trachomatis infected women with or without fertility disordersReduction of IL-17A might suppress the Th1 response and promote the Th2 response by boosting the function of Treg cells during silica-induced inflammatory response in vitro.Expression of the immunomodulator IL-10 in type I pneumocytes of the rat: alterations of IL-10 expression in radiation-induced lung damageThe pathogenesis of systemic sclerosis revisited.Opposing roles of leukotrienes and prostaglandins in fibrotic lung disease.Dendritic cells: novel players in fibrosis and scleroderma.Toxicology and clinical potential of nanoparticles.Identifying mechanisms driving formation of granuloma-associated fibrosis during Mycobacterium tuberculosis infection.Exploring Animal Models That Resemble Idiopathic Pulmonary Fibrosis.The clinical and immunologic features of pulmonary fibrosis in sarcoidosis.Neutralization of interleukin-1 beta attenuates silica-induced lung inflammation and fibrosis in C57BL/6 mice.Platelet-derived growth factor-producing CD4+ Foxp3+ regulatory T lymphocytes promote lung fibrosis.Pulmonary lesions and serum levels of soluble Fas (sCD95) in former hard coal miners.Macrophage polarization and function: new prospects for fibrotic disease.
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Pulmonary overexpression of IL-10 augments lung fibrosis and Th2 responses induced by silica particles.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh-hant
name
Pulmonary overexpression of IL ...... s induced by silica particles.
@en
Pulmonary overexpression of IL ...... s induced by silica particles.
@nl
type
label
Pulmonary overexpression of IL ...... s induced by silica particles.
@en
Pulmonary overexpression of IL ...... s induced by silica particles.
@nl
prefLabel
Pulmonary overexpression of IL ...... s induced by silica particles.
@en
Pulmonary overexpression of IL ...... s induced by silica particles.
@nl
P2093
P2860
P1476
Pulmonary overexpression of IL ...... s induced by silica particles.
@en
P2093
Francois Huaux
Monique Delos
Virginie Barbarin
P2860
P304
P356
10.1152/AJPLUNG.00329.2004
P577
2004-12-17T00:00:00Z