Pulmonary overexpression of IL-10 augments lung fibrosis and Th2 responses induced by silica particles. - Wikidata - awgldk - TriplyDB

Pulmonary overexpression of IL-10 augments lung fibrosis and Th2 responses induced by silica particles.

Pulmonary overexpression of IL-10 augments lung fibrosis and Th2 responses induced by silica particles.

http://www.wikidata.org/entity/Q45193717

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The chemokine CCL18 causes maturation of cultured monocytes to macrophages in the M2 spectrum The role of pro- and anti-inflammatory responses in silica-induced lung fibrosis.Silica, Silicosis, and Autoimmunity Dust events, pulmonary diseases and immune system Immune Components of Liver Damage Associated with Connective Tissue Diseases Fibrogenic and redox-related but not proinflammatory genes are upregulated in Lewis rat model of chronic silicosis Fibrocytes Regulate Wilms Tumor 1-Positive Cell Accumulation in Severe Fibrotic Lung Disease.Uncoupling between inflammatory and fibrotic responses to silica: evidence from MyD88 knockout mice Blockade of Wnt/β-Catenin Pathway Aggravated Silica-Induced Lung Inflammation through Tregs Regulation on Th Immune Responses Macrophage silica nanoparticle response is phenotypically dependent Granuloma formation induced by low-dose chronic silica inhalation is associated with an anti-apoptotic response in Lewis rats Hsp90 regulation of fibroblast activation in pulmonary fibrosis.Accelerated and progressive and lethal liver fibrosis in mice that lack interleukin (IL)-10, IL-12p40, and IL-13Rα2.Resistance to bleomycin-induced lung fibrosis in MMP-8 deficient mice is mediated by interleukin-10 CD4+CD25+Foxp3+ regulatory T cells depletion may attenuate the development of silica-induced lung fibrosis in mice.The green tea extract epigallocatechin-3-gallate inhibits irradiation-induced pulmonary fibrosis in adult rats.Innate immune processes are sufficient for driving silicosis in mice.Latent infection by γherpesvirus stimulates profibrotic mediator release from multiple cell types.Prevalence, hemodynamics, and cytokine profile of effusive-constrictive pericarditis in patients with tuberculous pericardial effusion Microhemorrhage is an early event in the pulmonary fibrotic disease of PECAM-1 deficient FVB/n mice.Silica-directed mast cell activation is enhanced by scavenger receptors IL-13Ralpha2 and IL-10 coordinately suppress airway inflammation, airway-hyperreactivity, and fibrosis in mice Quantification of echodensities in tuberculous pericardial effusion using fractal geometry: a proof of concept study.Sustained PI3K Activation exacerbates BLM-induced Lung Fibrosis via activation of pro-inflammatory and pro-fibrotic pathways.Macrophages and tissue injury: agents of defense or destruction?IL-17 stimulates differentiation of human anti-inflammatory macrophages and phagocytosis of apoptotic neutrophils in response to IL-10 and glucocorticoids.Host immune responses to chlamydial inclusion membrane proteins B and C in Chlamydia trachomatis infected women with or without fertility disorders Reduction of IL-17A might suppress the Th1 response and promote the Th2 response by boosting the function of Treg cells during silica-induced inflammatory response in vitro.Expression of the immunomodulator IL-10 in type I pneumocytes of the rat: alterations of IL-10 expression in radiation-induced lung damage The pathogenesis of systemic sclerosis revisited.Opposing roles of leukotrienes and prostaglandins in fibrotic lung disease.Dendritic cells: novel players in fibrosis and scleroderma.Toxicology and clinical potential of nanoparticles.Identifying mechanisms driving formation of granuloma-associated fibrosis during Mycobacterium tuberculosis infection.Exploring Animal Models That Resemble Idiopathic Pulmonary Fibrosis.The clinical and immunologic features of pulmonary fibrosis in sarcoidosis.Neutralization of interleukin-1 beta attenuates silica-induced lung inflammation and fibrosis in C57BL/6 mice.Platelet-derived growth factor-producing CD4+ Foxp3+ regulatory T lymphocytes promote lung fibrosis.Pulmonary lesions and serum levels of soluble Fas (sCD95) in former hard coal miners.Macrophage polarization and function: new prospects for fibrotic disease.

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Pulmonary overexpression of IL-10 augments lung fibrosis and Th2 responses induced by silica particles.

http://www.wikidata.org/entity/Q45193717

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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name

Pulmonary overexpression of IL ...... s induced by silica particles.

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Pulmonary overexpression of IL ...... s induced by silica particles.

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type

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Pulmonary overexpression of IL ...... s induced by silica particles.

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Pulmonary overexpression of IL ...... s induced by silica particles.

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Pulmonary overexpression of IL ...... s induced by silica particles.

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Pulmonary overexpression of IL ...... s induced by silica particles.

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AJPLUNG.00329.2004

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American Journal of Physiology - Lung Cellular and Molecular Physiology

P1476

Pulmonary overexpression of IL ...... s induced by silica particles.

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P2093

Francois Huaux

http://www.w3.org/2001/XMLSchema#string

Monique Delos

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Virginie Barbarin

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Zhou Xing

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P2860

Use of tetrandrine to differentiate between mechanisms involved in silica-versus bleomycin-induced fibrosis

Transforming growth factor-beta (TGF-beta) in silicosis

Idiopathic pulmonary fibrosis: prevailing and evolving hypotheses about its pathogenesis and implications for therapy

Cyclooxygenase-2 deficiency results in a loss of the anti-proliferative response to transforming growth factor-beta in human fibrotic lung fibroblasts and promotes bleomycin-induced pulmonary fibrosis in mice

Eosinophils and T lymphocytes possess distinct roles in bleomycin-induced lung injury and fibrosis

Interleukin 10(IL-10) inhibits cytokine synthesis by human monocytes: an autoregulatory role of IL-10 produced by monocytes

Two types of mouse T helper cell. IV. Th2 clones secrete a factor that inhibits cytokine production by Th1 clones

Cultured lung fibroblasts isolated from patients with idiopathic pulmonary fibrosis have a diminished capacity to synthesize prostaglandin E2 and to express cyclooxygenase-2.

Molecular mechanisms of antifibrotic effect of interferon gamma in bleomycin-mouse model of lung fibrosis: downregulation of TGF-beta and procollagen I and III gene expression.

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Dominique Lison

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8122213

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pulmonary fibrosis