beta-Amyloid peptides impair PKC-dependent functions of metabotropic glutamate receptors in prefrontal cortical neurons.
about
Amyloid-beta protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memoryBorna disease virus blocks potentiation of presynaptic activity through inhibition of protein kinase C signaling.Unfolded Protein Response and Macroautophagy in Alzheimer's, Parkinson's and Prion DiseasesCell line specific modulation of extracellular aβ42 by Hsp40Disassembly of shank and homer synaptic clusters is driven by soluble beta-amyloid(1-40) through divergent NMDAR-dependent signalling pathwaysAMPAR removal underlies Abeta-induced synaptic depression and dendritic spine lossMicroglial receptor for advanced glycation end product-dependent signal pathway drives beta-amyloid-induced synaptic depression and long-term depression impairment in entorhinal cortex.Spatial relationship between synapse loss and beta-amyloid deposition in Tg2576 mice.Group II metabotropic glutamate receptor stimulation triggers production and release of Alzheimer's amyloid(beta)42 from isolated intact nerve terminalsRACK1 is involved in β-amyloid impairment of muscarinic regulation of GABAergic transmissionEffects of stress and stress hormones on amyloid-beta protein and plaque deposition.Current experimental therapy for Alzheimer's diseaseEffects of donepezil on amyloid-beta and synapse density in the Tg2576 mouse model of Alzheimer's disease.MAPK, beta-amyloid and synaptic dysfunction: the role of RAGE.Neurodegeneration, β-amyloid and mood disorders: state of the art and future perspectives.GABAergic neurotransmission and new strategies of neuromodulation to compensate synaptic dysfunction in early stages of Alzheimer's disease.Glutamate receptors function as scaffolds for the regulation of β-amyloid and cellular prion protein signaling complexes.Protein Kinase C Activation as a Potential Therapeutic Strategy in Alzheimer's Disease: Is there a Role for Embryonic Lethal Abnormal Vision-like Proteins?Aβ impairs nicotinic regulation of inhibitory synaptic transmission and interneuron excitability in prefrontal cortex.Hippocampal neuronal cells that accumulate α-synuclein fragments are more vulnerable to Aβ oligomer toxicity via mGluR5--implications for dementia with Lewy bodies.
P2860
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P2860
beta-Amyloid peptides impair PKC-dependent functions of metabotropic glutamate receptors in prefrontal cortical neurons.
description
2005 nî lūn-bûn
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2005年の論文
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2005年学术文章
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name
beta-Amyloid peptides impair P ...... n prefrontal cortical neurons.
@en
beta-Amyloid peptides impair P ...... n prefrontal cortical neurons.
@nl
type
label
beta-Amyloid peptides impair P ...... n prefrontal cortical neurons.
@en
beta-Amyloid peptides impair P ...... n prefrontal cortical neurons.
@nl
prefLabel
beta-Amyloid peptides impair P ...... n prefrontal cortical neurons.
@en
beta-Amyloid peptides impair P ...... n prefrontal cortical neurons.
@nl
P2860
P356
P1476
beta-Amyloid peptides impair P ...... n prefrontal cortical neurons.
@en
P2093
Joanna P Tyszkiewicz
P2860
P304
P356
10.1152/JN.00939.2004
P407
P577
2005-01-19T00:00:00Z