Rho-kinase and myosin II activities are required for cell type and environment specific migration. - Wikidata - awgldk - TriplyDB

Rho-kinase and myosin II activities are required for cell type and environment specific migration.

Rho-kinase and myosin II activities are required for cell type and environment specific migration.

http://www.wikidata.org/entity/Q45241694

about

Rho-kinase/ROCK: A key regulator of the cytoskeleton and cell polarity Autotaxin and lysophosphatidic acid stimulate intestinal cell motility by redistribution of the actin modifying protein villin to the developing lamellipodia Generation of membrane structures during phagocytosis and chemotaxis of macrophages: role and regulation of the actin cytoskeleton A highly soluble, non-phototoxic, non-fluorescent blebbistatin derivative.Epithelial sheet folding induces lumen formation by Madin-Darby canine kidney cells in a collagen gel Endothelial cell traction and ECM density influence both capillary morphogenesis and maintenance in 3-D.Region-specific epithelial cell dynamics during branching morphogenesis Rapamycin regulates endothelial cell migration through regulation of the cyclin-dependent kinase inhibitor p27Kip1.A quantitative, facile, and high-throughput image-based cell migration method is a robust alternative to the scratch assay.INSIGHTS INTO THE ROLES OF NON-MUSCLE MYOSIN IIA IN HUMAN KERATINOCYTE MIGRATION Cells lacking β-actin are genetically reprogrammed and maintain conditional migratory capacity.ROCK1 as a novel prognostic marker in vulvar cancer Anti-inflammatory cytokine interleukin-19 inhibits smooth muscle cell migration and activation of cytoskeletal regulators of VSMC motility.RNAi mediated Tiam1 gene knockdown inhibits invasion of retinoblastoma Protease-dependent versus -independent cancer cell invasion programs: three-dimensional amoeboid movement revisited Mutation of ARHGAP9 in patients with coronary spastic angina.A novel regulator of angiogenesis in endothelial cells: 5-hydroxytriptamine 4 receptor Non-muscle myosin IIA differentially regulates intestinal epithelial cell restitution and matrix invasion Dysregulation of the Mammalian Target of Rapamycin and p27Kip1 Promotes Intimal Hyperplasia in Diabetes Mellitus Role of stress fibers and focal adhesions as a mediator for mechano-signal transduction in endothelial cells in situ.Rho kinases in cardiovascular physiology and pathophysiology: the effect of fasudil.Regulation of tyrosine phosphorylation in macrophage phagocytosis and chemotaxis.Rho-associated coiled-coil kinase (ROCK) signaling and disease.Extracellular matrix determinants and the regulation of cancer cell invasion stratagems.The cAMP-producing agonist beraprost inhibits human vascular smooth muscle cell migration via exchange protein directly activated by cAMP.Matrix geometry determines optimal cancer cell migration strategy and modulates response to interventions.Different effect of Rho kinase inhibition on calcium signaling in rat isolated large and small arteries.

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P2860

Rho-kinase and myosin II activities are required for cell type and environment specific migration.

http://www.wikidata.org/entity/Q45241694

description

2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年學術文章

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2005年學術文章

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2005年學術文章

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name

Rho-kinase and myosin II activ ...... nvironment specific migration.

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Rho-kinase and myosin II activ ...... nvironment specific migration.

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type

label

Rho-kinase and myosin II activ ...... nvironment specific migration.

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Rho-kinase and myosin II activ ...... nvironment specific migration.

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prefLabel

Rho-kinase and myosin II activ ...... nvironment specific migration.

@en

Rho-kinase and myosin II activ ...... nvironment specific migration.

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J.1365-2443.2005.00823.X

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Rho-kinase and myosin II activ ...... environment specific migration

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Akira Katsumi

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Kozo Kaibuchi

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Masanori Nakayama

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Mikito Takefuji

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Saeko Kawabata

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Takako Kaneko

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P2860

Restenosis after coronary angioplasty. Potential biologic determinants and role of intimal hyperplasia.

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107-117

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scholarly article

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