Cell cycle activation in striatal neurons from Huntington's disease patients and rats treated with 3-nitropropionic acid.
about
Prion-induced neurotoxicity: Possible role for cell cycle activity and DNA damage responseInsights into the regulation of neuronal viability by nucleophosmin/B23Targeted gene mutation of E2F1 evokes age-dependent synaptic disruption and behavioral deficits.E2F1 localizes predominantly to neuronal cytoplasm and fails to induce expression of its transcriptional targets in human immunodeficiency virus-induced neuronal damage.Comparison of modules of wild type and mutant Huntingtin and TP53 protein interaction networks: implications in biological processes and functions.Disruption of the nuclear membrane by perinuclear inclusions of mutant huntingtin causes cell-cycle re-entry and striatal cell death in mouse and cell models of Huntington's disease.Notch signaling in response to excitotoxicity induces neurodegeneration via erroneous cell cycle reentry.The dynamics of early-state transcriptional changes and aggregate formation in a Huntington's disease cell modelCell cycle reactivation in mature neurons: a link with brain plasticity, neuronal injury and neurodegenerative diseases?Role of cell cycle re-entry in neurons: a common apoptotic mechanism of neuronal cell death.Epigenetic treatment of neurological disease.Mechanism(s) of alteration of micro RNA expressions in Huntington's disease and their possible contributions to the observed cellular and molecular dysfunctions in the disease.Targeting Huntington's disease through histone deacetylases.Long Term Aggresome Accumulation Leads to DNA Damage, p53-dependent Cell Cycle Arrest, and Steric Interference in Mitosis.Chronic oxidative damage together with genome repair deficiency in the neurons is a double whammy for neurodegeneration: Is damage response signaling a potential therapeutic target?Identification and analysis of the promoter region of the human DHCR24 gene: involvement of DNA methylation and histone acetylation.Expression of cell cycle proteins in cortical neurons-Correlation with glutamate-induced neurotoxicity.Regulation of Neuronal Survival by Nucleophosmin 1 (NPM1) Is Dependent on Its Expression Level, Subcellular Localization, and Oligomerization Status.Phosphorylated retinoblastoma protein (p-Rb) is involved in neuronal apoptosis after traumatic brain injury in adult rats.The DNA Damage Response in Neurons: Die by Apoptosis or Survive in a Senescence-Like State?
P2860
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P2860
Cell cycle activation in striatal neurons from Huntington's disease patients and rats treated with 3-nitropropionic acid.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh-hant
name
Cell cycle activation in stria ...... ed with 3-nitropropionic acid.
@en
Cell cycle activation in stria ...... ed with 3-nitropropionic acid.
@nl
type
label
Cell cycle activation in stria ...... ed with 3-nitropropionic acid.
@en
Cell cycle activation in stria ...... ed with 3-nitropropionic acid.
@nl
prefLabel
Cell cycle activation in stria ...... ed with 3-nitropropionic acid.
@en
Cell cycle activation in stria ...... ed with 3-nitropropionic acid.
@nl
P50
P1476
Cell cycle activation in stria ...... ed with 3-nitropropionic acid.
@en
P2093
Isidre Ferrer
Natàlia Crespo-Biel
P304
P356
10.1016/J.IJDEVNEU.2008.07.016
P577
2008-08-12T00:00:00Z