BDNF prevents NMDA-induced toxicity in models of Huntington's disease: the effects are genotype specific and adenosine A2A receptor is involved.
about
HD iPSC-derived neural progenitors accumulate in culture and are susceptible to BDNF withdrawal due to glutamate toxicity.Human Mesenchymal Stem Cells Genetically Engineered to Overexpress Brain-derived Neurotrophic Factor Improve Outcomes in Huntington's Disease Mouse Models.Synaptic signalling and its interface with neuropathologies: snapshots from the past, present and future.Elucidating the role of the A2A adenosine receptor in neurodegeneration using neurons derived from Huntington's disease iPSCs.How does adenosine control neuronal dysfunction and neurodegeneration?The Gut-Brain Axis, BDNF, NMDA and CNS Disorders.Pathological overproduction: the bad side of adenosine.The Role of Adenosine Tone and Adenosine Receptors in Huntington's Disease
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P2860
BDNF prevents NMDA-induced toxicity in models of Huntington's disease: the effects are genotype specific and adenosine A2A receptor is involved.
description
2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
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name
BDNF prevents NMDA-induced tox ...... sine A2A receptor is involved.
@en
BDNF prevents NMDA-induced tox ...... sine A2A receptor is involved.
@nl
type
label
BDNF prevents NMDA-induced tox ...... sine A2A receptor is involved.
@en
BDNF prevents NMDA-induced tox ...... sine A2A receptor is involved.
@nl
prefLabel
BDNF prevents NMDA-induced tox ...... sine A2A receptor is involved.
@en
BDNF prevents NMDA-induced tox ...... sine A2A receptor is involved.
@nl
P2093
P2860
P50
P356
P1476
BDNF prevents NMDA-induced tox ...... osine A2A receptor is involved
@en
P2093
Maria Rosaria Domenici
Patrizia Popoli
Rita Pepponi
P2860
P304
P356
10.1111/JNC.12177
P407
P577
2013-02-27T00:00:00Z