IL-13 and IL-4 cause eotaxin release in human airway smooth muscle cells: a role for ERK.
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Preclinical development of CAT-354, an IL-13 neutralizing antibody, for the treatment of severe uncontrolled asthmaThe effect of IL-13 and IL-13R130Q, a naturally occurring IL-13 polymorphism, on the gene expression of human airway smooth muscle cellsAnti-IL5 therapy for asthma and beyondTreatment of experimental asthma using a single small molecule with anti-inflammatory and BK channel-activating properties.Proinflammatory and Th2 cytokines regulate the high affinity IgE receptor (FcepsilonRI) and IgE-dependant activation of human airway smooth muscle cellsAcellular pertussis vaccine protects against exacerbation of allergic asthma due to Bordetella pertussis in a murine model.Interleukin-4 activates large-conductance, calcium-activated potassium (BKCa) channels in human airway smooth muscle cells.Interleukin (IL)-4 and IL-13 up-regulate monocyte chemoattractant protein-1 expression in human bronchial epithelial cells: involvement of p38 mitogen-activated protein kinase, extracellular signal-regulated kinase 1/2 and Janus kinase-2 but not c-JExpression of IL-4 receptor alpha on smooth muscle cells is not necessary for development of experimental allergic asthma.Ablation of tumor progression locus 2 promotes a type 2 Th cell response in Ovalbumin-immunized mice.Monocyte 15-lipoxygenase gene expression requires ERK1/2 MAPK activity.Interleukin-13: prospects for new treatments.Mechanistic systems biology of inflammatory gene expression in airway smooth muscle as tool for asthma drug development.Role of mitogen-activated protein kinases in peptidoglycan-induced expression of inducible nitric oxide synthase and nitric oxide in mouse peritoneal macrophages: extracellular signal-related kinase, a negative regulatorCCR3: a key to mast cell phenotypic and functional diversity?New insights into the role of the mast cell in asthma.Anti-inflammatory effects of thiazolidinediones in human airway smooth muscle cells.Mechanical stimuli and IL-13 interact at integrin adhesion complexes to regulate expression of smooth muscle myosin heavy chain in airway smooth muscle tissue.Markers of tyrosine kinase activity in eosinophilic esophagitis: a pilot study of the FIP1L1-PDGFRα fusion gene, pERK 1/2, and pSTAT5.Interleukin-13 in asthma pathogenesis.Mitogen-activated protein kinases and asthma.IL-4 inhibits calcium transients in bovine trachealis cells by a ryanodine receptor-dependent mechanismWhole-genome sequencing suggests a chemokine gene cluster that modifies age at onset in familial Alzheimer's disease.The 21st century renaissance of the basophil? Current insights into its role in allergic responses and innate immunity.IL-25 improves IgA levels during parenteral nutrition through the JAK-STAT pathwayHeparin and structurally related polymers attenuate eotaxin-1 (CCL11) release from human airway smooth muscle.Tenascin-C deficiency in apo E-/- mouse increases eotaxin levels: implications for atherosclerosis.Airway smooth muscle in the pathophysiology and treatment of asthma.Glucocorticoid regulation of CD38 expression in human airway smooth muscle cells: role of dual specificity phosphatase 1.Interleukin-4, interleukin-13, signal transducer and activator of transcription factor 6, and allergic asthma.IL-13 R110Q, a Naturally Occurring IL-13 Polymorphism, Confers Enhanced Functional Activity in Cultured Human Bronchial Smooth Muscle CellsTranscriptional regulation of cytokine function in airway smooth muscle cells.The enigmatic cytokine oncostatin m and roles in diseaseBlockade of ankyrin repeat-rich membrane spanning protein modulates extracellular signal-regulated kinase expression and inhibits allergic inflammation in ovalbumin-sensitized mice.Basophils as Key Regulators of Allergic Inflammation and Th2-type Immunity.IL-13 Gene Polymorphisms are Associated With Rhinosinusitis and Eosinophilic Inflammation in Aspirin Intolerant Asthma.A Mathematical Modeling Approach to Understanding the Effect of Anti-Interleukin Therapy on Eosinophils.ERK1/2 mitogen-activated protein kinase selectively mediates IL-13-induced lung inflammation and remodeling in vivo.Regulation of IL-17A responses in human airway smooth muscle cells by Oncostatin M.IFN-gamma, IL-4 and IL-13 modulate responsiveness of human airway smooth muscle cells to IL-13.
P2860
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P2860
IL-13 and IL-4 cause eotaxin release in human airway smooth muscle cells: a role for ERK.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh
2002年學術文章
@zh-hant
name
IL-13 and IL-4 cause eotaxin release in human airway smooth muscle cells: a role for ERK.
@en
IL-13 and IL-4 cause eotaxin release in human airway smooth muscle cells: a role for ERK.
@nl
type
label
IL-13 and IL-4 cause eotaxin release in human airway smooth muscle cells: a role for ERK.
@en
IL-13 and IL-4 cause eotaxin release in human airway smooth muscle cells: a role for ERK.
@nl
prefLabel
IL-13 and IL-4 cause eotaxin release in human airway smooth muscle cells: a role for ERK.
@en
IL-13 and IL-4 cause eotaxin release in human airway smooth muscle cells: a role for ERK.
@nl
P2093
P1476
IL-13 and IL-4 cause eotaxin release in human airway smooth muscle cells: a role for ERK.
@en
P2093
David D Chism
Paul E Moore
Reynold A Panettieri
Stephanie A Shore
Trudi L Church
P304
P356
10.1152/AJPLUNG.00245.2001
P577
2002-04-01T00:00:00Z