Silibinin: an old drug for hematological disorders.

about

Silibinin: an old drug for hematological disorders.

Item

http://www.wikidata.org/entity/Q45747315

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh-hant

name

Silibinin: an old drug for hematological disorders.

@en

Silibinin: an old drug for hematological disorders.

@nl

type

Item

label

Silibinin: an old drug for hematological disorders.

@en

Silibinin: an old drug for hematological disorders.

@nl

prefLabel

Silibinin: an old drug for hematological disorders.

@en

Silibinin: an old drug for hematological disorders.

@nl

P1476

Silibinin: an old drug for hematological disorders.

@en

P2093

Dong-Sheng Huang

http://www.w3.org/2001/XMLSchema#string

Guo-Bing Zhang

http://www.w3.org/2001/XMLSchema#string

Hai Zou

http://www.w3.org/2001/XMLSchema#string

Xing-Xing Zhu

http://www.w3.org/2001/XMLSchema#string

Yi Wu

http://www.w3.org/2001/XMLSchema#string

Yuan Ma

http://www.w3.org/2001/XMLSchema#string

P2860

A randomized, controlled, double-blind, pilot study of milk thistle for the treatment of hepatotoxicity in childhood acute lymphoblastic leukemia (ALL)

Cytoreductive antitumor activity of PF-2341066, a novel inhibitor of anaplastic lymphoma kinase and c-Met, in experimental models of anaplastic large-cell lymphoma.

'Acute myeloid leukemia: a comprehensive review and 2016 update'

Acute myeloid leukemia.

Vitamin D3-driven signals for myeloid cell differentiation--implications for differentiation therapy

Silibinin can induce differentiation as well as enhance vitamin D3-induced differentiation of human AML cells ex vivo and regulates the levels of differentiation-related transcription factors.

Distinct combinatorial effects of the plant polyphenols curcumin, carnosic acid, and silibinin on proliferation and apoptosis in acute myeloid leukemia cells.

Secondary iron overload.

Silibinin and STAT3: A natural way of targeting transcription factors for cancer therapy.

Inhibition of Cot1/Tlp2 oncogene in AML cells reduces ERK5 activation and up-regulates p27Kip1 concomitant with enhancement of differentiation and cell cycle arrest induced by silibinin and 1,25-dihydroxyvitamin D(3).

P304

89307-89314

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.18632/ONCOTARGET.19153

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2017-07-11T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29179521

http://www.w3.org/2001/XMLSchema#string

P932

5687691

http://www.w3.org/2001/XMLSchema#string