Intrinsic and extrinsic mechanisms contribute to maintain the JAK/STAT pathway aberrantly activated in T-type large granular lymphocyte leukemia.
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Vitamin D in hematological disorders and malignancies.Sphingosine kinase inhibitors decrease viability and induce cell death in natural killer-large granular lymphocyte leukemia.Suberoylanilide hydroxamic acid (SAHA) and cladribine synergistically induce apoptosis in NK-LGL leukaemia.The analysis of clonal diversity and therapy responses using STAT3 mutations as a molecular marker in large granular lymphocytic leukemia.Chronic natural killer lymphoproliferative disorders: characteristics of an international cohort of 70 patients.The pathogenesis and treatment of large granular lymphocyte leukemiaDetection of monoclonal T populations in patients with KIR-restricted chronic lymphoproliferative disorder of NK cells.STAT3 gene mutations and their association with pure red cell aplasia in large granular lymphocyte leukemiaNovel Mechanisms of Compromised Lymphatic Endothelial Cell Homeostasis in Obesity: The Role of Leptin in Lymphatic Endothelial Cell Tube Formation and ProliferationLack of common TCRA and TCRB clonotypes in CD8(+)/TCRαβ(+) T-cell large granular lymphocyte leukemia: a review on the role of antigenic selection in the immunopathogenesis of CD8(+) T-LGLMature T-cell leukemias: Molecular and Clinical Aspects.Are T-LGL Leukemia and NK-Chronic Lymphoproliferative Disorder really two distinct diseases?Submicroscopic genomic rearrangements change gene expression in T-cell large granular lymphocyte leukemia.Multiple myeloma causes clonal T-cell immunosenescence: identification of potential novel targets for promoting tumour immunity and implications for checkpoint blockade.Genomic landscape characterization of large granular lymphocyte leukemia with a systems genetics approachSTAT3 mutation impacts biological and clinical features of T-LGL leukemia.Cyclophosphamide as a first-line therapy in LGL leukemia.Mutations in the signal transducer and activator of transcription family of genes in cancer.Dysregulation of the IFN-γ-STAT1 signaling pathway in a cell line model of large granular lymphocyte leukemia.microRNA-23a, -27a and -24 synergistically regulate JAK1/Stat3 cascade and serve as novel therapeutic targets in human acute erythroid leukemia.
P2860
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P2860
Intrinsic and extrinsic mechanisms contribute to maintain the JAK/STAT pathway aberrantly activated in T-type large granular lymphocyte leukemia.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
@yue
2013年學術文章
@zh-hant
name
Intrinsic and extrinsic mechan ...... granular lymphocyte leukemia.
@en
Intrinsic and extrinsic mechan ...... granular lymphocyte leukemia.
@nl
type
label
Intrinsic and extrinsic mechan ...... granular lymphocyte leukemia.
@en
Intrinsic and extrinsic mechan ...... granular lymphocyte leukemia.
@nl
prefLabel
Intrinsic and extrinsic mechan ...... granular lymphocyte leukemia.
@en
Intrinsic and extrinsic mechan ...... granular lymphocyte leukemia.
@nl
P2093
P1433
P1476
Intrinsic and extrinsic mechan ...... granular lymphocyte leukemia.
@en
P2093
Albana Lico
Anna Cabrelle
Antonella Teramo
Cristina Gattazzo
Elisa Boscaro
Federica Frezzato
Francesca Passeri
Giulia Tasca
Livio Trentin
P304
3843-54, S1
P356
10.1182/BLOOD-2012-07-441378
P407
P577
2013-03-20T00:00:00Z