T-lymphocytes response to Friend virus-induced tumour cell lines in mice of strains congenic at H--2.
about
H-2 polymorphisms are more uniformly distributed than allozyme polymorphisms in natural populations of house mice.Periodic loss of reactivity of a myeloma tumor with cytotoxic thymus-derived lymphocytes.Specificity studies on cytotoxic thymus-derived lymphocytes reactive with influenza virus-infected cells: evidence for dual recognition of H-2 and viral hemagglutinin antigens.Joint recognition by cytotoxic T cells of inactivated Sendai virus and products of the major histocompatibility complexHuman histocompatibility determinants and virus antigens: effect of measles virus infection on HLA expression.Exclusive involvement of H-2Db or H-2Kd product in the interaction between T-killer lymphocytes and syngeneic H-2b or H-2d viral lymphomas.Lung tumor-associated derepressed alloantigen coded for by the K region of the H-2 major histocompatibility complex.Mechanisms of genetic resistance to Friend virus leukemia in mice. IV. Identification of a gene (Fv-3) regulating immunosuppression in vitro, and its distinction from Fv-2 and genes regulating marrow allograft reactivity.Increased synthesis and expression of H-2 antigens on thymocytes as a result of radiation leukemia virus infection: a possible mechanism for H-2 linked control of virus-induced neoplasia.Persistence and pathogenicity of defective Friend spleen focus-forming virus. Decreased transplantability of hemopoietic cells as a marker for preleukemic change.H-2-linked genetic control of murine T-cell-mediated lympholysis to autologous cells modified with low concentrations of trinitrobenzene sulfonateRegulation of T-cell-mediated lympholysis by the murine major histocompatibility complex. I. Preferential in vitro responses to trinitrophenyl-modified self K- and D-coded gene products in parental and F1 hybrid mouse strainsInvolvement of fusion activity of ultraviolet light-inactivated Sendai virus in formation of target antigens recognized by cytotoxic T cells.Viral specificity of H-2-restricted T killer cells directed against syngeneic tumors induced by Gross, Friend, or Rauscher leukemia virus.Studies of cloned Friend erythroleukemia tumor cells. Modulation of the tumor-specific cytologic T lymphocyte response by infectious Friend virus production in vitro.Influence of the major histocompatibility complex on the repertoire of allospecific cytolytic T lymphocytes.Specificity studies on cytolytic T lymphocytes directed against murine leukemia virus-induced tumors. Analysis of monoclonal cytolytic T lymphocytes.Generation of an HLA-restricted cytotoxic T cell line reactive against cultured tumor cells from a patient infected with human T cell leukemia/lymphoma virus.Cell-mediated lympholysis to H-2-matched target cells modified with a series of nitrophenyl compounds.Cross-reactive lysis of trinitrophenyl (TNP)-derivatized H-2 incompatible target cells by cytolytic T lymphocytes generated against syngeneic TNP spleen cellsMultiple H-2 linked immune response gene control of H-2 D-associated T-cell-mediated lympholysis to trinitrophenyl-modified autologous cells: Ir-like genes mapping to the left of I-A and within the I region.Evidence for H-2-linked control of retrovirus production in Friend virus-induced tumor cell linesAnti-influenza virus cytotoxic T lymphocytes recognize the three viral polymerases and a nonstructural protein: responsiveness to individual viral antigens is major histocompatibility complex controlled.Immunosuppression by Friend leukemia virus is H-2 restricted by alloreactive T lymphocytes.T cells, T cells recognition structures, and the major histocompatibility complex.Mechanisms of the H-2 effect on viral leukemogenesis.Genetic control of radiation leukemia virus-induced tumorigenesis. I. Role of the major murine histocompatibility complex, H-2.H-2 restriction and non-restriction of T-cell-mediated cytotoxicity against mouse mammary tumour targets.H-2 compatibility requirements for T suppressor cell functions induced by Friend leukaemia virus.The generation of killer cells to trinitrophenyl-modified allogeneic targets by lymphocyte populations negatively selected to strong alloantigens.Inhibition by sera and soluble antigens of T-cell-mediated cytotoxicity against leukaemia-associated antigens.In a radiation chimaera, host H-2 antigens determine immune responsiveness of donor cytotoxic cells.Cytotoxic T lymphocytes induced in mice by inactivated influenza virus vaccine.Nature and specificity of effector cells in cell-mediated cytolysis of mouse adenovirus-infected cells.A secretable serine proteinase with highly restricted specificity from cytolytic T lymphocytes inactivates retrovirus-associated reverse transcriptase.
P2860
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P2860
T-lymphocytes response to Friend virus-induced tumour cell lines in mice of strains congenic at H--2.
description
1976 nî lūn-bûn
@nan
1976年の論文
@ja
1976年学术文章
@wuu
1976年学术文章
@zh-cn
1976年学术文章
@zh-hans
1976年学术文章
@zh-my
1976年学术文章
@zh-sg
1976年學術文章
@yue
1976年學術文章
@zh
1976年學術文章
@zh-hant
name
T-lymphocytes response to Frie ...... e of strains congenic at H--2.
@en
T-lymphocytes response to Frie ...... e of strains congenic at H--2.
@nl
type
label
T-lymphocytes response to Frie ...... e of strains congenic at H--2.
@en
T-lymphocytes response to Frie ...... e of strains congenic at H--2.
@nl
prefLabel
T-lymphocytes response to Frie ...... e of strains congenic at H--2.
@en
T-lymphocytes response to Frie ...... e of strains congenic at H--2.
@nl
P2093
P356
P1433
P1476
T-lymphocytes response to Frie ...... e of strains congenic at H--2.
@en
P2093
P2888
P304
P356
10.1038/260250A0
P407
P577
1976-03-01T00:00:00Z
P6179
1008384120