Glomerular epithelial cell injury associated with mutant alpha-actinin-4. - Wikidata - awgldk - TriplyDB

Glomerular epithelial cell injury associated with mutant alpha-actinin-4.

Glomerular epithelial cell injury associated with mutant alpha-actinin-4.

http://www.wikidata.org/entity/Q45916571

about

Podocyte injury and albuminuria in mice with podocyte-specific overexpression of the Ste20-like kinase, SLK.Genetic causes of proteinuria and nephrotic syndrome: impact on podocyte pathobiology.Alpha-actinin-4 and CLP36 protein deficiencies contribute to podocyte defects in multiple human glomerulopathies.Angiotensin II induces endoplasmic reticulum stress in podocyte, which would be further augmented by PI3-kinase inhibition Essential Role of X-Box Binding Protein-1 during Endoplasmic Reticulum Stress in Podocytes.Does endoplasmic reticulum stress mediate endothelin-1-induced renal inflammation?Proteostasis in endoplasmic reticulum--new mechanisms in kidney disease.Stress Signal Network between Hypoxia and ER Stress in Chronic Kidney Disease.Targeted inhibition of endoplasmic reticulum stress: New hope for renal fibrosis (Review)Mesencephalic astrocyte-derived neurotrophic factor (MANF), a new player in endoplasmic reticulum diseases: structure, biology, and therapeutic roles.Mesencephalic Astrocyte-Derived Neurotrophic Factor as a Urine Biomarker for Endoplasmic Reticulum Stress-Related Kidney Diseases.The inhibitory effect of quercetin on asymmetric dimethylarginine-induced apoptosis is mediated by the endoplasmic reticulum stress pathway in glomerular endothelial cells.Nonfunction of the ECT2 gene may cause renal tubulointerstitial injury leading to focal segmental glomerulosclerosis.Podocyte Injury Associated with Mutant α-Actinin-4 Structure-activity relationship of piperine and its synthetic amide analogs for therapeutic potential to prevent experimentally induced ER stress in vitro.The Chemical Chaperone, PBA, Reduces ER Stress and Autophagy and Increases Collagen IV α5 Expression in Cultured Fibroblasts From Men With X-Linked Alport Syndrome and Missense Mutations.Endoplasmic reticulum stress in the pathogenesis of fibrotic disease.Endoplasmic reticulum stress, the unfolded protein response and autophagy in kidney diseases.Doxorubicin-induced glomerulosclerosis with proteinuria in GFP-GABARAP transgenic mice.Glomerulosclerosis Induced by Deficiency of Membrane-Associated Guanylate Kinase Inverted 2 in Kidney Podocytes.The proteasome inhibitor bortezomib prevents lupus nephritis in the NZB/W F1 mouse model by preservation of glomerular and tubulointerstitial architecture.

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P2860

Glomerular epithelial cell injury associated with mutant alpha-actinin-4.

http://www.wikidata.org/entity/Q45916571

description

2009 nî lūn-bûn

@nan

2009年の論文

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2009年学术文章

@wuu

2009年学术文章

@zh

2009年学术文章

@zh-cn

2009年学术文章

@zh-hans

2009年学术文章

@zh-my

2009年学术文章

@zh-sg

2009年學術文章

@yue

2009年學術文章

@zh-hant

name

Glomerular epithelial cell injury associated with mutant alpha-actinin-4.

@en

Glomerular epithelial cell injury associated with mutant alpha-actinin-4.

@nl

type

label

Glomerular epithelial cell injury associated with mutant alpha-actinin-4.

@en

Glomerular epithelial cell injury associated with mutant alpha-actinin-4.

@nl

prefLabel

Glomerular epithelial cell injury associated with mutant alpha-actinin-4.

@en

Glomerular epithelial cell injury associated with mutant alpha-actinin-4.

@nl

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P356

AJPRENAL.00055.2009

P1433

American Journal of Physiology - Renal Physiology

P1476

Glomerular epithelial cell injury associated with mutant alpha-actinin-4.

@en

P2093

Andrey V Cybulsky

http://www.w3.org/2001/XMLSchema#string

Chris R J Kennedy

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Joan Papillon

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Julie Guillemette

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Krikor Bijian

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Tomoko Takano

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P2860

Caspase-12 mediates endoplasmic-reticulum-specific apoptosis and cytotoxicity by amyloid-beta

ASK1 is essential for endoplasmic reticulum stress-induced neuronal cell death triggered by expanded polyglutamine repeats

Actinin-4, a novel actin-bundling protein associated with cell motility and cancer invasion

Endoplasmic reticulum stress: cell life and death decisions.

Alpha-actinin-4-mediated FSGS: an inherited kidney disease caused by an aggregated and rapidly degraded cytoskeletal protein

FSGS-associated alpha-actinin-4 (K256E) impairs cytoskeletal dynamics in podocytes

Actin cytoskeleton regulates extracellular matrix-dependent survival signals in glomerular epithelial cells

Mice deficient in alpha-actinin-4 have severe glomerular disease

Impairment of the ubiquitin-proteasome system by protein aggregation

Adaptation to ER stress is mediated by differential stabilities of pro-survival and pro-apoptotic mRNAs and proteins

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F987-95

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scholarly article

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10.1152/AJPRENAL.00055.2009

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4

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297

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19640905

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