Spondylarthritis in HLA-B27/human beta2-microglobulin-transgenic rats is not prevented by lack of CD8.
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Review: The interleukin-23/interleukin-17 axis in spondyloarthritis pathogenesis: Th17 and beyondHLA-B27 and human β2-microglobulin affect the gut microbiota of transgenic ratsFrom HLA-B27 to spondyloarthritis: a journey through the ER.Generation and characterization of a Tet-On (rtTA-M2) transgenic ratERAP1 reduces accumulation of aberrant and disulfide-linked forms of HLA-B27 on the cell surfaceInhibiting HLA-B27 homodimer-driven immune cell inflammation in spondylarthritis.An overview of genetics of paediatric rheumatic diseases.Classification of juvenile spondyloarthritis: Enthesitis-related arthritis and beyond.Update on ankylosing spondylitis: current concepts in pathogenesis.Reverse interferon signature is characteristic of antigen-presenting cells in human and rat spondyloarthritis.Pathogenicity of Misfolded and Dimeric HLA-B27 MoleculesThe danger model approach to the pathogenesis of the rheumatic diseases.Interactions of the innate and adaptive arms of the immune system in the pathogenesis of spondyloarthritisSulfasalazine Treatment Suppresses the Formation of HLA-B27 Heavy Chain Homodimer in Patients with Ankylosing Spondylitis.Silencing or inhibition of endoplasmic reticulum aminopeptidase 1 (ERAP1) suppresses free heavy chain expression and Th17 responses in ankylosing spondylitis.The genetic association of RUNX3 with ankylosing spondylitis can be explained by allele-specific effects on IRF4 recruitment that alter gene expression.Role of HLA-B27 in the pathogenesis of ankylosing spondylitis (Review).Are spondylarthritides related but distinct conditions or a single disease with a heterogeneous phenotype?The Human Leukocyte Antigen (HLA)-B27 Peptidome in Vivo, in Spondyloarthritis-susceptible HLA-B27 Transgenic Rats and the Effect of Erap1 Deletion.Revisiting MHC genes in spondyloarthritis.Review: animal models as a tool to dissect pivotal pathways driving spondyloarthritis.Innate Immune Activation Can Trigger Experimental Spondyloarthritis in HLA-B27/Huβ2m Transgenic Rats.Autoinflammation and HLA-B27: Beyond Antigen Presentation.Expression of aberrant HLA-B27 molecules is dependent on B27 dosage and peptide supply.The interplay between HLA-B27 and ERAP1/ERAP2 aminopeptidases: from anti-viral protection to spondyloarthritis.[Intestinal microbiota in individualized therapies].Disease-specific and inflammation-independent stromal alterations in spondylarthritis synovitis.IL-23 induces spondyloarthropathy by acting on ROR-γt+ CD3+CD4-CD8- entheseal resident T cells.The Insulin Receptor Plays a Critical Role in T Cell Function and Adaptive Immunity.Axial spondyloarthritis.Expression of MHC class I dimers and ERAP1 in an ankylosing spondylitis patient cohort.Whodunit? The Contribution of Interleukin (IL)-17/IL-22-Producing γδ T Cells, αβ T Cells, and Innate Lymphoid Cells to the Pathogenesis of Spondyloarthritis.Progress in our understanding of the pathogenesis of ankylosing spondylitis
P2860
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P2860
Spondylarthritis in HLA-B27/human beta2-microglobulin-transgenic rats is not prevented by lack of CD8.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh-hant
name
Spondylarthritis in HLA-B27/hu ...... not prevented by lack of CD8.
@en
Spondylarthritis in HLA-B27/hu ...... not prevented by lack of CD8.
@nl
type
label
Spondylarthritis in HLA-B27/hu ...... not prevented by lack of CD8.
@en
Spondylarthritis in HLA-B27/hu ...... not prevented by lack of CD8.
@nl
prefLabel
Spondylarthritis in HLA-B27/hu ...... not prevented by lack of CD8.
@en
Spondylarthritis in HLA-B27/hu ...... not prevented by lack of CD8.
@nl
P2093
P2860
P356
P1476
Spondylarthritis in HLA-B27/hu ...... not prevented by lack of CD8.
@en
P2093
Holger M Reichardt
Jens van den Brandt
Joel D Taurog
Martha L Dorris
Nimman Satumtira
Rohit Sharma
Tri M Tran
P2860
P304
P356
10.1002/ART.24599
P50
P577
2009-07-01T00:00:00Z