B cells suppress the inflammatory response in a mouse model of primary biliary cirrhosis. - Wikidata - awgldk - TriplyDB

B cells suppress the inflammatory response in a mouse model of primary biliary cirrhosis.

B cells suppress the inflammatory response in a mouse model of primary biliary cirrhosis.

http://www.wikidata.org/entity/Q45957513

about

B cell: T cell interactions occur within hepatic granulomas during experimental visceral leishmaniasis Identification of potential cytokine pathways for therapeutic intervention in murine primary biliary cirrhosis Alterations of serum protein N-glycosylation in two mouse models of chronic liver disease are hepatocyte and not B cell driven.B-cell depletion with anti-CD20 ameliorates autoimmune cholangitis but exacerbates colitis in transforming growth factor-beta receptor II dominant negative mice B cells delay neutrophil migration toward the site of stimulus: tardiness critical for effective bacillus Calmette-Guérin vaccination against tuberculosis infection in mice.Deletion of interleukin-6 in mice with the dominant negative form of transforming growth factor beta receptor II improves colitis but exacerbates autoimmune cholangitis B cells promote hepatic inflammation, biliary cyst formation, and salivary gland inflammation in the NOD.c3c4 model of autoimmune cholangitis.B cell depletion therapy exacerbates murine primary biliary cirrhosis Immune-mediated bile duct injury: The case of primary biliary cirrhosis.A novel mechanism of B cell-mediated immune suppression through CD73 expression and adenosine production Pathological features of new animal models for primary biliary cirrhosis.Plasma cells and the chronic nonsuppurative destructive cholangitis of primary biliary cirrhosis.Deletion of interleukin (IL)-12p35 induces liver fibrosis in dominant-negative TGFβ receptor type II mice.Role of autoimmunity in primary biliary cirrhosis.Overexpression of microRNA-21 is associated with elevated pro-inflammatory cytokines in dominant-negative TGF-β receptor type II mouse.Successful treatment of murine autoimmune cholangitis by parabiosis: Implications for hematopoietic therapy Nuclear antigens and auto/alloantibody responses: friend or foe in transplant immunology.Clonality, activated antigen-specific CD8(+) T cells, and development of autoimmune cholangitis in dnTGFβRII mice Dysregulation of peritoneal cavity B1a cells and murine primary biliary cholangitis.Experimental evidence on the immunopathogenesis of primary biliary cirrhosis.Immunological orchestration of liver fibrosis.Animal models in primary biliary cirrhosis and primary sclerosing cholangitis.Etiopathogenesis of primary biliary cirrhosis: an overview of recent developments.Trichloroethylene Exposure Reduces Liver Injury in a Mouse Model of Primary Biliary Cholangitis.Endogenous interleukin-22 protects against inflammatory bowel disease but not autoimmune cholangitis in dominant negative form of transforming growth factor beta receptor type II mice.Animal models of primary biliary cirrhosis.Hepatic B cells are readily activated by Toll-like receptor-4 ligation and secrete less interleukin-10 than lymphoid tissue B cells.Anti-CD40 ligand monoclonal antibody delays the progression of murine autoimmune cholangitis.Epitope-specific anti-nuclear antibodies are expressed in a mouse model of primary biliary cirrhosis and are cytokine-dependent.The role of natural killer (NK) and NK T cells in the loss of tolerance in murine primary biliary cirrhosis.FoxO1 is a regulator of MHC-II expression and anti-tumor effect of tumor-associated macrophages.Enteroantigen (eAg)-binding B lymphocytes in the mouse - phenotype, distribution, function and eAg-specific antibody secretion.Decreased production of interleukin-10 and transforming growth factor-β in Toll-like receptor-activated intestinal B cells in SAMP1/Yit mice Anti-drug Antibodies Against a Novel Humanized Anti-CD20 Antibody Impair Its Therapeutic Effect on Primary Biliary Cholangitis in Human CD20- and FcγR-Expressing Mice

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B cells suppress the inflammatory response in a mouse model of primary biliary cirrhosis.

http://www.wikidata.org/entity/Q45957513

description

2008 nî lūn-bûn

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2008年の論文

@ja

2008年学术文章

@wuu

2008年学术文章

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2008年学术文章

@zh-cn

2008年学术文章

@zh-hans

2008年学术文章

@zh-my

2008年学术文章

@zh-sg

2008年學術文章

@yue

2008年學術文章

@zh-hant

name

B cells suppress the inflammatory response in a mouse model of primary biliary cirrhosis.

@en

B cells suppress the inflammatory response in a mouse model of primary biliary cirrhosis.

@nl

type

label

B cells suppress the inflammatory response in a mouse model of primary biliary cirrhosis.

@en

B cells suppress the inflammatory response in a mouse model of primary biliary cirrhosis.

@nl

prefLabel

B cells suppress the inflammatory response in a mouse model of primary biliary cirrhosis.

@en

B cells suppress the inflammatory response in a mouse model of primary biliary cirrhosis.

@nl

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P356

J.GASTRO.2008.11.035

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Gastroenterology

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B cells suppress the inflammatory response in a mouse model of primary biliary cirrhosis.

@en

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Christopher Bowlus

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Guo-Xiang Yang

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Ian R Mackay

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Kanji Wakabayashi

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Katsunori Yoshida

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Koichi Tsuneyama

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Richard A Flavell

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Weici Zhang

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William M Ridgway

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Yoshiyuki Ueno

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1037-1047

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10.1053/J.GASTRO.2008.11.035

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M. Eric Gershwin

Aftab A. Ansari

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2008-11-19T00:00:00Z

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23715743

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