Activation of a HIF1alpha-PPARgamma axis underlies the integration of glycolytic and lipid anabolic pathways in pathologic cardiac hypertrophy. - Wikidata - awgldk - TriplyDB

Activation of a HIF1alpha-PPARgamma axis underlies the integration of glycolytic and lipid anabolic pathways in pathologic cardiac hypertrophy.

Activation of a HIF1alpha-PPARgamma axis underlies the integration of glycolytic and lipid anabolic pathways in pathologic cardiac hypertrophy.

http://www.wikidata.org/entity/Q45985589

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Metabolic insight into mechanisms of high-altitude adaptation in Tibetans Reactive oxygen species, nutrition, hypoxia and diseases: Problems solved?Hypoxia-inducible factor 1 and cardiovascular disease Targets for therapy in sarcomeric cardiomyopathies Lipid Droplets: A Key Cellular Organelle Associated with Cancer Cell Survival under Normoxia and Hypoxia The AP-1 transcription factor c-Jun prevents stress-imposed maladaptive remodeling of the heart.Molecular mechanisms of neutrophil dysfunction in glycogen storage disease type Ib.A switch in the source of ATP production and a loss in capacity to perform glycolysis are hallmarks of hepatocyte failure in advance liver disease.Impaired Ca(2+)-handling in HIF-1alpha(+/-) mice as a consequence of pressure overload.Time-dependent PPARγ Modulation of HIF-1α Signaling in Hypoxic Pulmonary Artery Smooth Muscle Cells.Mitochondrial-nuclear genome interactions in non-alcoholic fatty liver disease in mice Hypoxia-Inducible Factor 1α Is a Critical Downstream Mediator for Hypoxia-Induced Mitogenic Factor (FIZZ1/RELMα)-Induced Pulmonary Hypertension.Identification and validation of the pathways and functions regulated by the orphan nuclear receptor, ROR alpha1, in skeletal muscle.Induction of PGC-1α expression can be detected in blood samples of patients with ST-segment elevation acute myocardial infarction AMPK-Dependent Metabolic Regulation by PPAR Agonists.Hypoxic signature of microRNAs in glioblastoma: insights from small RNA deep sequencing.Loss of hypoxia-inducible factor prolyl hydroxylase activity in cardiomyocytes phenocopies ischemic cardiomyopathy.Conditional PPARγ knockout from cardiomyocytes of adult mice impairs myocardial fatty acid utilization and cardiac function.Thyroid Hormone-Regulated Cardiac microRNAs are Predicted to Suppress Pathological Hypertrophic Signaling.Circadian rhythms, Wnt/beta-catenin pathway and PPAR alpha/gamma profiles in diseases with primary or secondary cardiac dysfunction.Cardiac muscle regeneration: lessons from development.Redox signaling in cardiac myocytes.Cardiomyocyte-specific prolyl-4-hydroxylase domain 2 knock out protects from acute myocardial ischemic injury Mitochondrial adaptations to physiological vs. pathological cardiac hypertrophy.Enhanced cardiac Akt/protein kinase B signaling contributes to pathological cardiac hypertrophy in part by impairing mitochondrial function via transcriptional repression of mitochondrion-targeted nuclear genes.Peroxisome proliferator-activated receptor γ inhibits pulmonary hypertension targeting store-operated calcium entry The sarcoplasmic reticulum luminal thiol oxidase ERO1 regulates cardiomyocyte excitation-coupled calcium release and response to hemodynamic load.Cardiac-specific ablation of ARNT leads to lipotoxicity and cardiomyopathy.A systems biology approach to understand the pathophysiological mechanisms of cardiac pathological hypertrophy associated with rosiglitazone.Hypoxia induces a lipogenic cancer cell phenotype via HIF1α-dependent and -independent pathways.Mice with cardiac overexpression of peroxisome proliferator-activated receptor γ have impaired repolarization and spontaneous fatal ventricular arrhythmias Admission hypoxia-inducible factor 1α levels and in-hospital mortality in patients with acute decompensated heart failure.Protective effect of olmesartan against cardiac ischemia/reperfusion injury in spontaneously hypertensive rats Endothelial expression of hypoxia-inducible factor 1 protects the murine heart and aorta from pressure overload by suppression of TGF-β signaling.Renal cancer: oxygen meets metabolism Metabolic stress in the myocardium: adaptations of gene expression.Transient hypoxia reprograms differentiating adipocytes for enhanced insulin sensitivity and triglyceride accumulation Hypoxia causes triglyceride accumulation by HIF-1-mediated stimulation of lipin 1 expression Nitro-fatty acids in cardiovascular regulation and diseases: characteristics and molecular mechanisms.Cardiomyocyte autophagy: metabolic profit and loss.

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P2860

Activation of a HIF1alpha-PPARgamma axis underlies the integration of glycolytic and lipid anabolic pathways in pathologic cardiac hypertrophy.

http://www.wikidata.org/entity/Q45985589

description

2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年學術文章

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2009年學術文章

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name

Activation of a HIF1alpha-PPAR ...... athologic cardiac hypertrophy.

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Activation of a HIF1alpha-PPAR ...... athologic cardiac hypertrophy.

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type

label

Activation of a HIF1alpha-PPAR ...... athologic cardiac hypertrophy.

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Activation of a HIF1alpha-PPAR ...... athologic cardiac hypertrophy.

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prefLabel

Activation of a HIF1alpha-PPAR ...... athologic cardiac hypertrophy.

@en

Activation of a HIF1alpha-PPAR ...... athologic cardiac hypertrophy.

@nl

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J.CMET.2009.05.005

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Cell Metabolism

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Activation of a HIF1alpha-PPAR ...... athologic cardiac hypertrophy.

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Allison Felley

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Anna Bogdanova

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Christophe Montessuit

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Ellen Aasum

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Evelyne Perriard

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Jean-Claude Perriard

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Malgorzata Tokarska-Schlattner

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Marianne Suter

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Renata Windak

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Tatiana Krebs

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512-524

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10.1016/J.CMET.2009.05.005

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6

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2009-06-01T00:00:00Z

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